Christopher Zelinka

956 total citations
16 papers, 700 citations indexed

About

Christopher Zelinka is a scholar working on Molecular Biology, Ophthalmology and Neurology. According to data from OpenAlex, Christopher Zelinka has authored 16 papers receiving a total of 700 indexed citations (citations by other indexed papers that have themselves been cited), including 13 papers in Molecular Biology, 9 papers in Ophthalmology and 6 papers in Neurology. Recurrent topics in Christopher Zelinka's work include Retinal Development and Disorders (12 papers), Retinal Diseases and Treatments (6 papers) and Neuroinflammation and Neurodegeneration Mechanisms (6 papers). Christopher Zelinka is often cited by papers focused on Retinal Development and Disorders (12 papers), Retinal Diseases and Treatments (6 papers) and Neuroinflammation and Neurodegeneration Mechanisms (6 papers). Christopher Zelinka collaborates with scholars based in United States. Christopher Zelinka's co-authors include Andy J. Fischer, Melissa A. Scott, Donika Gallina, Kanika Ghai, Levi Todd, Eric R. Ritchey, Leo Volkov, Nima Milani‐Nejad, Jun Liu and Patrick Sherwood and has published in prestigious journals such as Journal of Neuroscience, PLoS ONE and Development.

In The Last Decade

Christopher Zelinka

16 papers receiving 689 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Christopher Zelinka United States 15 500 227 167 146 135 16 700
Brian P. Buckingham United States 6 918 1.8× 510 2.2× 148 0.9× 150 1.0× 142 1.1× 7 1.1k
Levi Todd United States 19 865 1.7× 187 0.8× 252 1.5× 243 1.7× 83 0.6× 27 1.0k
Mark K. Walsh United States 11 172 0.3× 227 1.0× 79 0.5× 113 0.8× 167 1.2× 28 654
Zofia Dreher Australia 12 650 1.3× 319 1.4× 224 1.3× 78 0.5× 150 1.1× 14 888
Manuela Lahne United States 10 440 0.9× 79 0.3× 109 0.7× 110 0.8× 41 0.3× 18 586
Lidia Bonfanti Italy 7 622 1.2× 126 0.6× 97 0.6× 148 1.0× 61 0.5× 9 915
Karen Eastlake United Kingdom 13 387 0.8× 203 0.9× 63 0.4× 45 0.3× 131 1.0× 22 548
Lilian Kisiswa United Kingdom 13 221 0.4× 129 0.6× 64 0.4× 53 0.4× 56 0.4× 21 480
Isabella Palazzo United States 11 328 0.7× 97 0.4× 129 0.8× 98 0.7× 28 0.2× 13 432
Rebecca Jo United States 6 317 0.6× 105 0.5× 89 0.5× 180 1.2× 28 0.2× 8 491

Countries citing papers authored by Christopher Zelinka

Since Specialization
Citations

This map shows the geographic impact of Christopher Zelinka's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Christopher Zelinka with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Christopher Zelinka more than expected).

Fields of papers citing papers by Christopher Zelinka

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Christopher Zelinka. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Christopher Zelinka. The network helps show where Christopher Zelinka may publish in the future.

Co-authorship network of co-authors of Christopher Zelinka

This figure shows the co-authorship network connecting the top 25 collaborators of Christopher Zelinka. A scholar is included among the top collaborators of Christopher Zelinka based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Christopher Zelinka. Christopher Zelinka is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

16 of 16 papers shown
1.
Pierce, Kenneth E., Paul Curran, Christopher Zelinka, et al.. (2019). Sildenafil Administration in Dogs Heterozygous for a Functional Null Mutation in Pde6a: Suppressed Rod-Mediated ERG Responses and Apparent Retinal Outer Nuclear Layer Thinning. Advances in experimental medicine and biology. 1185. 371–376. 2 indexed citations
2.
Zelinka, Christopher, et al.. (2018). Targeted disruption of the endogenous zebrafish rhodopsin locus as models of rapid rod photoreceptor degeneration.. PubMed. 24. 587–602. 15 indexed citations
3.
Zelinka, Christopher, et al.. (2016). mTor-signaling is required for the formation of proliferating Müller glia-derived progenitor cells in the chick retina. Development. 143(11). 1859–73. 51 indexed citations
4.
Gallina, Donika, Christopher Zelinka, Colleen M. Cebulla, & Andy J. Fischer. (2015). Activation of glucocorticoid receptors in Müller glia is protective to retinal neurons and suppresses microglial reactivity. Experimental Neurology. 273. 114–125. 34 indexed citations
5.
Todd, Levi, et al.. (2015). Comparative analysis of glucagonergic cells, glia, and the circumferential marginal zone in the reptilian retina. The Journal of Comparative Neurology. 524(1). 74–89. 26 indexed citations
6.
Fischer, Andy J., Christopher Zelinka, & Nima Milani‐Nejad. (2014). Reactive retinal microglia, neuronal survival, and the formation of retinal folds and detachments. Glia. 63(2). 313–327. 50 indexed citations
7.
Gallina, Donika, Christopher Zelinka, & Andy J. Fischer. (2014). Glucocorticoid receptors in the retina, Müller glia and the formation of Müller glia-derived progenitors. Development. 141(17). 3340–3351. 63 indexed citations
8.
Fischer, Andy J., Christopher Zelinka, Donika Gallina, Melissa A. Scott, & Levi Todd. (2014). Reactive microglia and macrophage facilitate the formation of Müller glia‐derived retinal progenitors. Glia. 62(10). 1608–1628. 79 indexed citations
9.
Ritchey, Eric R., et al.. (2012). The combination of IGF1 and FGF2 and the induction of excessive ocular growth and extreme myopia. Experimental Eye Research. 99. 1–16. 52 indexed citations
10.
Zelinka, Christopher, Melissa A. Scott, Leo Volkov, & Andy J. Fischer. (2012). The Reactivity, Distribution and Abundance of Non-Astrocytic Inner Retinal Glial (NIRG) Cells Are Regulated by Microglia, Acute Damage, and IGF1. PLoS ONE. 7(9). e44477–e44477. 39 indexed citations
11.
Ritchey, Eric R., et al.. (2012). Vision-guided ocular growth in a mutant chicken model with diminished visual acuity. Experimental Eye Research. 102. 59–69. 18 indexed citations
12.
Ritchey, Eric R., et al.. (2011). The chicken cornea as a model of wound healing and neuronal re-innervation.. PubMed. 17. 2440–54. 39 indexed citations
13.
Fischer, Andy J., Christopher Zelinka, & Melissa A. Scott. (2010). Heterogeneity of Glia in the Retina and Optic Nerve of Birds and Mammals. PLoS ONE. 5(6). e10774–e10774. 50 indexed citations
14.
Ghai, Kanika, Christopher Zelinka, & Andy J. Fischer. (2010). Notch Signaling Influences Neuroprotective and Proliferative Properties of Mature Müller Glia. Journal of Neuroscience. 30(8). 3101–3112. 84 indexed citations
15.
Ghai, Kanika, Christopher Zelinka, & Andy J. Fischer. (2009). Serotonin released from amacrine neurons is scavenged and degraded in bipolar neurons in the retina. Journal of Neurochemistry. 111(1). 1–14. 45 indexed citations
16.
Fischer, Andy J., Melissa A. Scott, Christopher Zelinka, & Patrick Sherwood. (2009). A novel type of glial cell in the retina is stimulated by insulin‐like growth factor 1 and may exacerbate damage to neurons and Müller glia. Glia. 58(6). 633–649. 53 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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