Christopher P. Arnold

808 total citations
10 papers, 533 citations indexed

About

Christopher P. Arnold is a scholar working on Molecular Biology, Immunology and Cancer Research. According to data from OpenAlex, Christopher P. Arnold has authored 10 papers receiving a total of 533 indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Molecular Biology, 2 papers in Immunology and 2 papers in Cancer Research. Recurrent topics in Christopher P. Arnold's work include Planarian Biology and Electrostimulation (3 papers), Immune Cell Function and Interaction (2 papers) and Healthcare Policy and Management (1 paper). Christopher P. Arnold is often cited by papers focused on Planarian Biology and Electrostimulation (3 papers), Immune Cell Function and Interaction (2 papers) and Healthcare Policy and Management (1 paper). Christopher P. Arnold collaborates with scholars based in United States, China and Singapore. Christopher P. Arnold's co-authors include Chang‐Zheng Chen, Steven Schaffert, Masayuki Iwasaki, Alejandro Sánchez Alvarado, Michael L. Cleary, Piu Wong, Francesca Ficara, Tim C. P. Somervaille, Christine Carico and Xiaohong Cai and has published in prestigious journals such as Nature, Nature Communications and The EMBO Journal.

In The Last Decade

Christopher P. Arnold

10 papers receiving 526 citations

Peers

Christopher P. Arnold

MB

CR

IMMUN

GPC

IS

Norbert Eichner Germany

Hitoshi Aihara Japan

Julie Huxley‐Jones United Kingdom

Vairavan Lakshmanan India

М. П. Самойлович Russia

Foster C. Gonsalves United States

Anastasiya Boltengagen Germany

Chunxue Guo China

Conghui Yang China

Alexis Perez‐Gonzalez Australia

Norbert Eichner Germany

Christopher P. Arnold

424 ×1.2

MB

237 ×1.6

CR

50 ×0.5

IMMUN

64 ×0.9

GPC

10 ×0.2

IS

Citations per year, relative to Christopher P. Arnold Christopher P. Arnold (= 1×) peers Norbert Eichner

Countries citing papers authored by Christopher P. Arnold

Since Specialization

Citations

This map shows the geographic impact of Christopher P. Arnold's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Christopher P. Arnold with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Christopher P. Arnold more than expected).

Fields of papers citing papers by Christopher P. Arnold

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Christopher P. Arnold. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Christopher P. Arnold. The network helps show where Christopher P. Arnold may publish in the future.

Co-authorship network of co-authors of Christopher P. Arnold

This figure shows the co-authorship network connecting the top 25 collaborators of Christopher P. Arnold. A scholar is included among the top collaborators of Christopher P. Arnold based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Christopher P. Arnold. Christopher P. Arnold is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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10 of 10 papers shown

1.

Arnold, Christopher P., Frederick G. Mann, Stephanie H. Nowotarski, et al.. (2021). Hox genes regulate asexual reproductive behavior and tissue segmentation in adult animals. Nature Communications. 12(1). 6706–6706. 19 indexed citations

2.

Arnold, Christopher P., Blair W. Benham-Pyle, Jeffrey J. Lange, Christopher J. Wood, & Alejandro Sánchez Alvarado. (2019). Wnt and TGFβ coordinate growth and patterning to regulate size-dependent behaviour. Nature. 572(7771). 655–659. 36 indexed citations

3.

Arnold, Christopher P., et al.. (2016). Pathogenic shifts in endogenous microbiota impede tissue regeneration via distinct activation of TAK1/MKK/p38. eLife. 5. 71 indexed citations

4.

Schaffert, Steven, Christina Loh, Song Wang, et al.. (2015). mir-181a-1/b-1 Modulates Tolerance through Opposing Activities in Selection and Peripheral T Cell Function. The Journal of Immunology. 195(4). 1470–1479. 40 indexed citations

5.

Zheng, Qiupeng, Xiaohong Cai, Meng How Tan, et al.. (2014). Precise Gene Deletion and Replacement Using the CRISPR/Cas9 System in Human Cells. BioTechniques. 57(3). 115–124. 112 indexed citations

6.

Arnold, Christopher P., et al.. (2010). BDA special care case mix model. BDJ. 208(7). 291–296. 26 indexed citations

7.

Wong, Piu, Masayuki Iwasaki, Tim C. P. Somervaille, et al.. (2010). The miR-17-92 microRNA Polycistron Regulates MLL Leukemia Stem Cell Potential by Modulating p21 Expression. Cancer Research. 70(9). 3833–3842. 137 indexed citations

8.

Brown, Spencer A., Jordan P. Farkas, Christopher P. Arnold, et al.. (2007). Heat shock proteins 47 and 70 expression in rodent skin model as a function of contact cooling temperature: Are we overcooling our target?. Lasers in Surgery and Medicine. 39(6). 504–512. 12 indexed citations

9.

Arnold, Christopher P., et al.. (2007). A κB sequence code for pathway‐specific innate immune responses. The EMBO Journal. 26(16). 3826–3835. 66 indexed citations

10.

Olson, A., et al.. (1992). A gelled propellant sustainer stage. 14 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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