Christopher M. Loughrey

2.0k total citations
43 papers, 1.5k citations indexed

About

Christopher M. Loughrey is a scholar working on Molecular Biology, Cardiology and Cardiovascular Medicine and Cellular and Molecular Neuroscience. According to data from OpenAlex, Christopher M. Loughrey has authored 43 papers receiving a total of 1.5k indexed citations (citations by other indexed papers that have themselves been cited), including 27 papers in Molecular Biology, 26 papers in Cardiology and Cardiovascular Medicine and 8 papers in Cellular and Molecular Neuroscience. Recurrent topics in Christopher M. Loughrey's work include Cardiac electrophysiology and arrhythmias (20 papers), Ion channel regulation and function (16 papers) and Signaling Pathways in Disease (6 papers). Christopher M. Loughrey is often cited by papers focused on Cardiac electrophysiology and arrhythmias (20 papers), Ion channel regulation and function (16 papers) and Signaling Pathways in Disease (6 papers). Christopher M. Loughrey collaborates with scholars based in United Kingdom, Germany and Japan. Christopher M. Loughrey's co-authors include Godfrey L. Smith, Ian Young, Paul McNamee, Janet H. Lightbody, E.R. Trimble, Stuart A. Nicklin, Susan Currie, Dorothy McMaster, Tim Seidler and Graeme Milligan and has published in prestigious journals such as Circulation, Journal of the American College of Cardiology and Circulation Research.

In The Last Decade

Christopher M. Loughrey

42 papers receiving 1.4k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Christopher M. Loughrey United Kingdom 20 711 654 246 185 170 43 1.5k
Meilei Ma Japan 27 603 0.8× 753 1.2× 82 0.3× 60 0.3× 49 0.3× 58 1.6k
R. Takayanagi Japan 23 722 1.0× 346 0.5× 58 0.2× 68 0.4× 149 0.9× 49 1.6k
Maritza J. Romero United States 23 512 0.7× 364 0.6× 114 0.5× 97 0.5× 27 0.2× 44 1.8k
Alice Habermeier Germany 19 499 0.7× 458 0.7× 43 0.2× 54 0.3× 77 0.5× 32 1.8k
Xavier Maréchal France 25 909 1.3× 581 0.9× 61 0.2× 55 0.3× 44 0.3× 55 2.0k
Gwen S. Scott United States 21 534 0.8× 99 0.2× 141 0.6× 73 0.4× 111 0.7× 30 1.7k
Patrick Vinay Canada 19 563 0.8× 220 0.3× 217 0.9× 83 0.4× 62 0.4× 61 1.2k
Tomoyuki Kita Japan 16 324 0.5× 129 0.2× 39 0.2× 175 0.9× 95 0.6× 33 1.2k
G. Palladini Italy 20 395 0.6× 185 0.3× 54 0.2× 62 0.3× 63 0.4× 54 1.1k
Liangzheng Chang China 9 486 0.7× 319 0.5× 40 0.2× 67 0.4× 57 0.3× 15 1.1k

Countries citing papers authored by Christopher M. Loughrey

Since Specialization
Citations

This map shows the geographic impact of Christopher M. Loughrey's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Christopher M. Loughrey with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Christopher M. Loughrey more than expected).

Fields of papers citing papers by Christopher M. Loughrey

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Christopher M. Loughrey. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Christopher M. Loughrey. The network helps show where Christopher M. Loughrey may publish in the future.

Co-authorship network of co-authors of Christopher M. Loughrey

This figure shows the co-authorship network connecting the top 25 collaborators of Christopher M. Loughrey. A scholar is included among the top collaborators of Christopher M. Loughrey based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Christopher M. Loughrey. Christopher M. Loughrey is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Boland, Erin, William Fuller, Joe Swift, et al.. (2025). Biomechanical and compositional basement membrane defects due to a Col4a1 mutation affect cardiac morphology and function. Matrix Biology. 141. 82–100.
2.
Koay, Yen Chin, Yang Cao, Xiaosuo Wang, et al.. (2025). The Heart Has Intrinsic Ketogenic Capacity that Mediates NAD + Therapy in HFpEF. Circulation Research. 136(10). 1113–1130. 2 indexed citations
3.
Martin, Tamara P., et al.. (2021). Preclinical models of myocardial infarction: from mechanism to translation. British Journal of Pharmacology. 179(5). 770–791. 38 indexed citations
4.
Riddell, Alexandra, Martin McBride, Thomas Braun, et al.. (2020). RUNX1: an emerging therapeutic target for cardiovascular disease. Cardiovascular Research. 116(8). 1410–1423. 58 indexed citations
5.
Nicklin, Stuart A., et al.. (2020). Signalling pathways linking cysteine cathepsins to adverse cardiac remodelling. Cellular Signalling. 76. 109770–109770. 9 indexed citations
6.
Hortigón-Vinagre, María P., Víctor Zamora, Mónica Flores‐Muñoz, et al.. (2016). Gene Therapy With Angiotensin-(1-9) Preserves Left Ventricular Systolic Function After Myocardial Infarction. Journal of the American College of Cardiology. 68(24). 2652–2666. 36 indexed citations
7.
Guy, Jacky, Jim Selfridge, K.E. Tanner, et al.. (2016). Exclusive expression of MeCP2 in the nervous system distinguishes between brain and peripheral Rett syndrome-like phenotypes. Human Molecular Genetics. 25(20). ddw269–ddw269. 51 indexed citations
8.
He, Weihong, Kirsty Foote, Peter R. T. Bowman, et al.. (2015). 7 Runx1 deficiency protects against adverse cardiac remodelling following myocardial infarction. Abstracts. A3.1–A3. 2 indexed citations
9.
He, Weihong, et al.. (2014). The Cathepsin-L Inhibitor CAA0225 Improves Cardiac Function During Ischaemia-Reperfusion. Biophysical Journal. 106(2). 729a–729a. 2 indexed citations
10.
Kelly, Anthony E., et al.. (2011). The effect of K201 on isolated working rabbit heart mechanical function during pharmacologically induced Ca2+ overload. British Journal of Pharmacology. 165(4b). 1068–1083. 6 indexed citations
11.
Lockhart, Christopher, et al.. (2010). Effect of pioglitazone on endothelial function in impaired glucose tolerance. Diabetes Obesity and Metabolism. 12(8). 709–715. 16 indexed citations
12.
Toischer, Karl, Stephan E. Lehnart, Gero Tenderich, et al.. (2009). K201 improves aspects of the contractile performance of human failing myocardium via reduction in Ca2+ leak from the sarcoplasmic reticulum. Basic Research in Cardiology. 105(2). 279–287. 42 indexed citations
13.
Kelly, Allen, et al.. (2009). A Quantitative Assessment Of Selective Pharmacological Inhibition Of Serca In Isolated Rabbit Working Hearts. Biophysical Journal. 96(3). 515a–515a. 1 indexed citations
14.
Loughrey, Christopher M., Naoyuki Otani, Tim Seidler, et al.. (2007). K201 modulates excitation–contraction coupling and spontaneous Ca2+ release in normal adult rabbit ventricular cardiomyocytes. Cardiovascular Research. 76(2). 236–246. 44 indexed citations
15.
Seidler, Tim, Christopher M. Loughrey, Darya Zibrova, et al.. (2007). Overexpression of FK-506–Binding Protein 12.0 Modulates Excitation–Contraction Coupling in Adult Rabbit Ventricular Cardiomyocytes. Circulation Research. 101(10). 1020–1029. 30 indexed citations
16.
Loughrey, Christopher M., et al.. (2006). S100A1 decreases calcium spark frequency and alters their spatial characteristics in ventricular cardiomyocytes. Circulation. 114(18). 92–92. 1 indexed citations
17.
Miller, Stephen E., Susan Currie, Christopher M. Loughrey, et al.. (2005). Effects of calsequestrin over-expression on excitation–contraction coupling in isolated rabbit cardiomyocytes. Cardiovascular Research. 67(4). 667–677. 18 indexed citations
18.
Loughrey, Christopher M., et al.. (2002). The Relationship between Intracellular [Ca2+] and Ca2+ Wave Characteristics in Permeabilised Cardiomyocytes from the Rabbit. The Journal of Physiology. 543(3). 859–870. 30 indexed citations
19.
Loughrey, Christopher M., et al.. (2001). Relationship between extracellular [Ca 2+ ] and intracellular Ca 2+ wave characteristics in β-escin-permeabilised single cardiac myocytes (rabbit). ENLIGHTEN (Jurnal Bimbingan dan Konseling Islam). 1 indexed citations
20.
Riley, M., Christopher M. Loughrey, Paul Wilkinson, Chris Patterson, & George Varghese. (1997). Tuberculosis in health service employees in Northern Ireland. Respiratory Medicine. 91(9). 546–550. 6 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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