Christopher J. Passero

535 total citations
8 papers, 425 citations indexed

About

Christopher J. Passero is a scholar working on Molecular Biology, Sensory Systems and Nephrology. According to data from OpenAlex, Christopher J. Passero has authored 8 papers receiving a total of 425 indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Molecular Biology, 3 papers in Sensory Systems and 1 paper in Nephrology. Recurrent topics in Christopher J. Passero's work include Ion Transport and Channel Regulation (7 papers), Ion channel regulation and function (6 papers) and Ion Channels and Receptors (3 papers). Christopher J. Passero is often cited by papers focused on Ion Transport and Channel Regulation (7 papers), Ion channel regulation and function (6 papers) and Ion Channels and Receptors (3 papers). Christopher J. Passero collaborates with scholars based in United States and United Kingdom. Christopher J. Passero's co-authors include Thomas R. Kleyman, Rebecca P. Hughey, Marcelo D. Carattino, Gunhild M. Mueller, Stevan P. Tofovic, Helbert Rondon‐Berrios, Mike M. Myerburg, Joseph M. Pilewski, Carlos A. Steren and Ahmad B. Maarouf and has published in prestigious journals such as Journal of Biological Chemistry, American Journal of Physiology-Renal Physiology and Current Opinion in Nephrology & Hypertension.

In The Last Decade

Christopher J. Passero

8 papers receiving 422 citations

Peers

Christopher J. Passero

MB

PRM

SS

NEPHR

ND

Kozo Iwashita Japan

Rolando Carrisoza‐Gaytan United States

Eriko Kikuchi Japan

Nannette Marr Netherlands

Erik-Jan Kamsteeg Netherlands

Tamehito Onoe Japan

Holli Shumaker United States

Deborah A. Keeton United States

Sharon Mulroy United Kingdom

L. Rome United States

Kozo Iwashita Japan

Christopher J. Passero

276 ×0.8

MB

143 ×1.1

PRM

22 ×0.3

SS

45 ×0.7

NEPHR

65 ×1.2

ND

Citations per year, relative to Christopher J. Passero Christopher J. Passero (= 1×) peers Kozo Iwashita

Countries citing papers authored by Christopher J. Passero

Since Specialization

Citations

This map shows the geographic impact of Christopher J. Passero's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Christopher J. Passero with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Christopher J. Passero more than expected).

Fields of papers citing papers by Christopher J. Passero

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Christopher J. Passero. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Christopher J. Passero. The network helps show where Christopher J. Passero may publish in the future.

Co-authorship network of co-authors of Christopher J. Passero

This figure shows the co-authorship network connecting the top 25 collaborators of Christopher J. Passero. A scholar is included among the top collaborators of Christopher J. Passero based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Christopher J. Passero. Christopher J. Passero is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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8 of 8 papers shown

1.

Jindal, Nidhi, Dhiraj Yadav, Christopher J. Passero, et al.. (2011). Membranous nephropathy: a rare renal manifestation of IgG4-related systemic disease. Clinical Nephrology. 77(4). 321–328. 14 indexed citations

2.

Chen, Jingxin, et al.. (2011). External Cu2+ Inhibits Human Epithelial Na+ Channels by Binding at a Subunit Interface of Extracellular Domains. Journal of Biological Chemistry. 286(31). 27436–27446. 22 indexed citations

3.

Passero, Christopher J., Gunhild M. Mueller, Michael M. Myerburg, et al.. (2011). TMPRSS4-dependent activation of the epithelial sodium channel requires cleavage of the γ-subunit distal to the furin cleavage site. American Journal of Physiology-Renal Physiology. 302(1). F1–F8. 44 indexed citations

4.

Passero, Christopher J., Marcelo D. Carattino, Ossama B. Kashlan, et al.. (2010). Defining an inhibitory domain in the gamma subunit of the epithelial sodium channel. American Journal of Physiology-Renal Physiology. 299(4). F854–F861. 42 indexed citations

5.

Passero, Christopher J., Rebecca P. Hughey, & Thomas R. Kleyman. (2009). New role for plasmin in sodium homeostasis. Current Opinion in Nephrology & Hypertension. 19(1). 13–19. 44 indexed citations

6.

Passero, Christopher J., et al.. (2009). Conformational Changes Associated with Proton-dependent Gating of ASIC1a. Journal of Biological Chemistry. 284(52). 36473–36481. 34 indexed citations

7.

Passero, Christopher J., Gunhild M. Mueller, Helbert Rondon‐Berrios, et al.. (2008). Plasmin Activates Epithelial Na+ Channels by Cleaving the γ Subunit. Journal of Biological Chemistry. 283(52). 36586–36591. 151 indexed citations

8.

Carattino, Marcelo D., Christopher J. Passero, Carlos A. Steren, et al.. (2007). Defining an inhibitory domain in the α-subunit of the epithelial sodium channel. American Journal of Physiology-Renal Physiology. 294(1). F47–F52. 74 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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