Christopher J. Hetherington

5.8k total citations · 1 hit paper
32 papers, 4.8k citations indexed

About

Christopher J. Hetherington is a scholar working on Molecular Biology, Immunology and Hematology. According to data from OpenAlex, Christopher J. Hetherington has authored 32 papers receiving a total of 4.8k indexed citations (citations by other indexed papers that have themselves been cited), including 20 papers in Molecular Biology, 18 papers in Immunology and 15 papers in Hematology. Recurrent topics in Christopher J. Hetherington's work include Acute Myeloid Leukemia Research (15 papers), Epigenetics and DNA Methylation (7 papers) and Retinoids in leukemia and cellular processes (7 papers). Christopher J. Hetherington is often cited by papers focused on Acute Myeloid Leukemia Research (15 papers), Epigenetics and DNA Methylation (7 papers) and Retinoids in leukemia and cellular processes (7 papers). Christopher J. Hetherington collaborates with scholars based in United States, Japan and Singapore. Christopher J. Hetherington's co-authors include Daniel G. Tenen, Dong‐Er Zhang, D E Zhang, Pu Zhang, Gretchen J. Darlington, Liming Zhou, Scott W. Hiebert, Kristina Rhoades, Huimin Chen and Koichi Akashi and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Nature Genetics.

In The Last Decade

Christopher J. Hetherington

32 papers receiving 4.7k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Absence of granulocyte colony-stimulating factor signaling and neutrophil development in CCAAT enhancer binding protein α-deficient mice

1997 761 citations Dong‐Er Zhang, Pu Zhang et al. Proceedings of the National Academy of Sciences profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Christopher J. Hetherington United States	29	2.8k	2.1k	1.8k	817	508	32	4.8k
Frank Rosenbauer Germany	31	2.8k 1.0×	1.4k 0.6×	2.1k 1.2×	788 1.0×	649 1.3×	54	5.0k
M Fagioli Italy	27	3.4k 1.2×	2.4k 1.1×	939 0.5×	695 0.9×	184 0.4×	46	4.5k
Norio Asou Japan	34	2.4k 0.8×	2.9k 1.4×	1.0k 0.6×	783 1.0×	366 0.7×	180	4.8k
Cheryl D. Helgason Canada	38	3.1k 1.1×	836 0.4×	1.8k 1.0×	882 1.1×	742 1.5×	71	5.2k
Lorraine Robb Australia	37	2.3k 0.8×	900 0.4×	1.8k 1.0×	1.3k 1.6×	401 0.8×	73	4.9k
J D Griffin United States	32	1.3k 0.4×	945 0.4×	1.5k 0.9×	897 1.1×	347 0.7×	47	3.7k
François Sigaux France	30	1.8k 0.6×	989 0.5×	1.1k 0.6×	954 1.2×	382 0.8×	76	3.8k
Peter D. Aplan United States	48	4.4k 1.6×	2.3k 1.1×	1.1k 0.6×	899 1.1×	549 1.1×	155	6.4k
Mathijs Baens Belgium	29	1.5k 0.5×	742 0.4×	1.2k 0.7×	959 1.2×	998 2.0×	51	3.7k
JD Griffin United States	25	1.3k 0.4×	2.1k 1.0×	1.9k 1.1×	1.2k 1.5×	195 0.4×	61	4.7k

Countries citing papers authored by Christopher J. Hetherington

Since Specialization

Citations

This map shows the geographic impact of Christopher J. Hetherington's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Christopher J. Hetherington with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Christopher J. Hetherington more than expected).

Fields of papers citing papers by Christopher J. Hetherington

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Christopher J. Hetherington. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Christopher J. Hetherington. The network helps show where Christopher J. Hetherington may publish in the future.

Co-authorship network of co-authors of Christopher J. Hetherington

This figure shows the co-authorship network connecting the top 25 collaborators of Christopher J. Hetherington. A scholar is included among the top collaborators of Christopher J. Hetherington based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Christopher J. Hetherington. Christopher J. Hetherington is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Welner, Robert S., Deepak Bararia, Giovanni Amabile, et al.. (2013). C/EBPα is required for development of dendritic cell progenitors. Blood. 121(20). 4073–4081. 30 indexed citations

Levantini, Elena, Sanghoon Lee, Hanna S. Radomska, et al.. (2011). RUNX1 regulates the CD34 gene in haematopoietic stem cells by mediating interactions with a distal regulatory element. The EMBO Journal. 30(19). 4059–4070. 21 indexed citations

Kobayashi, Susumu, Takeshi Shimamura, Stefano Monti, et al.. (2006). Transcriptional Profiling Identifies Cyclin D1 as a Critical Downstream Effector of Mutant Epidermal Growth Factor Receptor Signaling. Cancer Research. 66(23). 11389–11398. 98 indexed citations

Hirai, Hideyo, Pu Zhang, Tajhal Dayaram, et al.. (2006). C/EBPβ is required for 'emergency' granulopoiesis. Nature Immunology. 7(7). 732–739. 324 indexed citations

Ritchie, Kenneth J., Michael P. Malakhov, Christopher J. Hetherington, et al.. (2002). Dysregulation of protein modification by ISG15 results in brain cell injury. Genes & Development. 16(17). 2207–2212. 136 indexed citations

Malakhova, Oxana A., Michael P. Malakhov, Christopher J. Hetherington, & Dong‐Er Zhang. (2002). Lipopolysaccharide Activates the Expression of ISG15-specific Protease UBP43 via Interferon Regulatory Factor 3. Journal of Biological Chemistry. 277(17). 14703–14711. 105 indexed citations

Yuan, Youzhong, Liming Zhou, Toshihiro Miyamoto, et al.. (2001). AML1-ETO expression is directly involved in the development of acute myeloid leukemia in the presence of additional mutations. Proceedings of the National Academy of Sciences. 98(18). 10398–10403. 324 indexed citations

Zhang, Dong‐Er, Sebastien A. Burel, Liming Zhou, Christopher J. Hetherington, & Youzhong Yuan. (2001). AML1 and AML1 Fusion Protein AML1-ETO in Myeloid Gene Regulation and Leukemogenesis. Blood Cells Molecules and Diseases. 27(2). 368–376. 3 indexed citations

Liu, Liqin, et al.. (2000). Cloning and Characterization of a Novel Human Ubiquitin-Specific Protease, a Homologue of Murine UBP43 (Usp18). Genomics. 65(1). 44–52. 59 indexed citations

10.

Hetherington, Christopher J., et al.. (1999). CCAAT/Enhancer-binding Protein Activates the CD14 Promoter and Mediates Transforming Growth Factor β Signaling in Monocyte Development. Journal of Biological Chemistry. 274(33). 23242–23248. 70 indexed citations

11.

Petrovick, Martha, Scott W. Hiebert, Alan D. Friedman, et al.. (1998). Multiple Functional Domains of AML1: PU.1 and C/EBPα Synergize with Different Regions of AML1. Molecular and Cellular Biology. 18(7). 3915–3925. 192 indexed citations

12.

Zhang, Dong‐Er, et al.. (1997). Absence of granulocyte colony-stimulating factor signaling and neutrophil development in CCAAT enhancer binding protein α-deficient mice. Proceedings of the National Academy of Sciences. 94(2). 569–574. 761 indexed citations breakdown →

13.

Yergeau, Donald, Christopher J. Hetherington, Qing Wang, et al.. (1997). Embryonic lethality and impairment of haematopoiesis in mice heterozygous for an AML1-ETO fusion gene. Nature Genetics. 15(3). 303–306. 296 indexed citations

14.

Zhang, Dong‐Er, Christopher J. Hetherington, Shari Meyers, et al.. (1996). CCAAT Enhancer-Binding Protein (C/EBP) and AML1 (CBFα2) Synergistically Activate the Macrophage Colony-Stimulating Factor Receptor Promoter. Molecular and Cellular Biology. 16(3). 1231–1240. 358 indexed citations

15.

Chen, Huimin, Pu Zhang, Hanna S. Radomska, et al.. (1996). Octamer Binding Factors and Their Coactivator Can Activate the Murine PU.1 (spi-1) Promoter. Journal of Biological Chemistry. 271(26). 15743–15752. 57 indexed citations

16.

Zhang, D.E., Stefan Hohaus, Maria Teresa Voso, et al.. (1996). Function of PU.1 (Spi-1), C/EBP, and AML1 in Early Myelopoiesis: Regulation of Multiple Myeloid CSF Receptor Promoters. Current topics in microbiology and immunology. 211. 137–147. 117 indexed citations

17.

Zhang, Dong‐Er, Christopher J. Hetherington, Huimin Chen, & Daniel G. Tenen. (1994). The macrophage transcription factor PU.1 directs tissue-specific expression of the macrophage colony-stimulating factor receptor. Molecular and Cellular Biology. 14(1). 373–381. 121 indexed citations

18.

Zhang, D E, et al.. (1994). Regulation of CD14 expression during monocytic differentiation induced with 1 alpha ,25-dihydroxyvitamin D3.. The Journal of Immunology. 153(7). 3276–3284. 92 indexed citations

19.

Zhang, D E, et al.. (1994). Identification of a region which directs the monocytic activity of the colony-stimulating factor 1 (macrophage colony-stimulating factor) receptor promoter and binds PEBP2/CBF (AML1).. Molecular and Cellular Biology. 14(12). 8085–8095. 180 indexed citations

20.

Zhang, D E, et al.. (1994). The macrophage transcription factor PU.1 directs tissue-specific expression of the macrophage colony-stimulating factor receptor.. Molecular and Cellular Biology. 14(1). 373–381. 351 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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