Christopher H. Emfinger

700 total citations
18 papers, 465 citations indexed

About

Christopher H. Emfinger is a scholar working on Surgery, Genetics and Molecular Biology. According to data from OpenAlex, Christopher H. Emfinger has authored 18 papers receiving a total of 465 indexed citations (citations by other indexed papers that have themselves been cited), including 13 papers in Surgery, 9 papers in Genetics and 8 papers in Molecular Biology. Recurrent topics in Christopher H. Emfinger's work include Pancreatic function and diabetes (10 papers), Diabetes and associated disorders (6 papers) and Metabolism, Diabetes, and Cancer (5 papers). Christopher H. Emfinger is often cited by papers focused on Pancreatic function and diabetes (10 papers), Diabetes and associated disorders (6 papers) and Metabolism, Diabetes, and Cancer (5 papers). Christopher H. Emfinger collaborates with scholars based in United States, Australia and Austria. Christopher H. Emfinger's co-authors include Marı́a S. Remedi, John M. Stafford, Melissa Martinez, Brian T. Palmisano, Lin Zhu, Radhika Aramandla, Ioannis Papagiannis, Chunhua Dai, Courtney S. Thompson and Alena Shostak and has published in prestigious journals such as Journal of Clinical Investigation, Diabetes and Journal of Lipid Research.

In The Last Decade

Christopher H. Emfinger

17 papers receiving 460 citations

Peers

Christopher H. Emfinger

SURGE

MB

EDM

GENET

PHYSI

Oliver Kluth Germany

Damien Demozay United States

Anett Seelig Germany

Maria Lytrivi Belgium

Kazuki Orime Japan

Sofie Hædersdal Denmark

Krzysztof Wanic Poland

Yoshinori Shimajiri Japan

Sachihiko Ozawa Japan

Neehar Gupta Canada

Oliver Kluth Germany

Christopher H. Emfinger

209 ×0.9

SURGE

213 ×1.2

MB

130 ×0.8

EDM

112 ×0.7

GENET

112 ×1.3

PHYSI

Citations per year, relative to Christopher H. Emfinger Christopher H. Emfinger (= 1×) peers Oliver Kluth

Countries citing papers authored by Christopher H. Emfinger

Since Specialization

Citations

This map shows the geographic impact of Christopher H. Emfinger's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Christopher H. Emfinger with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Christopher H. Emfinger more than expected).

Fields of papers citing papers by Christopher H. Emfinger

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Christopher H. Emfinger. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Christopher H. Emfinger. The network helps show where Christopher H. Emfinger may publish in the future.

Co-authorship network of co-authors of Christopher H. Emfinger

This figure shows the co-authorship network connecting the top 25 collaborators of Christopher H. Emfinger. A scholar is included among the top collaborators of Christopher H. Emfinger based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Christopher H. Emfinger. Christopher H. Emfinger is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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18 of 18 papers shown

1.

Binns, Derk D., G C King, Andrew T. Templin, et al.. (2025). VDAC1 is a target for pharmacologically induced insulin hypersecretion in β cells. Cell Reports. 44(6). 115834–115834.

2.

Emfinger, Christopher H., Kathryn L. Schueler, Sarah M. King, et al.. (2023). Identification of genetic drivers of plasma lipoprotein size in the Diversity Outbred mouse population. Journal of Lipid Research. 64(12). 100471–100471. 2 indexed citations

3.

Klerk, Eleonora de, Christopher H. Emfinger, Mark P. Keller, et al.. (2023). Loss of ZNF148 enhances insulin secretion in human pancreatic β cells. JCI Insight. 8(11). 3 indexed citations

4.

Emfinger, Christopher H., Brian S. Yandell, Kathryn L. Schueler, et al.. (2023). Novel regulators of islet function identified from genetic variation in mouse islet Ca2+ oscillations. eLife. 12. 4 indexed citations

5.

Emfinger, Christopher H., Brian S. Yandell, Kathryn L. Schueler, et al.. (2023). Novel regulators of islet function identified from genetic variation in mouse islet Ca2+ oscillations. eLife. 12. 4 indexed citations

6.

Keller, Mark P., Chris O’Connor, Markus Bitzer, et al.. (2023). Genetic Analysis of Obesity-Induced Diabetic Nephropathy in BTBR Mice. Diabetes. 73(2). 312–317. 4 indexed citations

7.

Zhang, Haixia, Christopher H. Emfinger, Conor McClenaghan, et al.. (2021). Complex consequences of Cantu syndrome SUR2 variant R1154Q in genetically modified mice. JCI Insight. 6(5). 10 indexed citations

8.

Keller, Mark P., Mary E. Rabaglia, Kathryn L. Schueler, et al.. (2019). Gene loci associated with insulin secretion in islets from nondiabetic mice. Journal of Clinical Investigation. 129(10). 4419–4432. 46 indexed citations

9.

Emfinger, Christopher H., Réka Lőrincz, Yixi Wang, et al.. (2019). Beta‐cell excitability and excitability‐driven diabetes in adult Zebrafish islets. Physiological Reports. 7(11). e14101–e14101. 13 indexed citations

10.

Lőrincz, Réka, Christopher H. Emfinger, Michael Giolai, et al.. (2018). In vivomonitoring of intracellular Ca²⁺dynamics in the pancreatic β-cells of zebrafish embryos. Islets. 10(6). 221–238. 11 indexed citations

11.

Zhu, Lin, Christopher H. Emfinger, Bryan A. Parks, et al.. (2018). CETP Inhibition Improves HDL Function but Leads to Fatty Liver and Insulin Resistance in CETP-Expressing Transgenic Mice on a High-Fat Diet. Diabetes. 67(12). 2494–2506. 30 indexed citations

12.

Emfinger, Christopher H., et al.. (2018). Contribution of systemic inflammation to permanence of K_ATP-induced neonatal diabetes in mice. American Journal of Physiology-Endocrinology and Metabolism. 315(6). E1121–E1132. 1 indexed citations

13.

Emfinger, Christopher H., Zihan Yan, Yixi Wang, et al.. (2017). Expression and function of ATP-dependent potassium channels in zebrafish islet β-cells. Royal Society Open Science. 4(2). 160808–160808. 12 indexed citations

14.

Dai, Chunhua, Alena Shostak, Greg Poffenberger, et al.. (2016). Stress-impaired transcription factor expression and insulin secretion in transplanted human islets. Journal of Clinical Investigation. 126(5). 1857–1870. 81 indexed citations

15.

Remedi, Marı́a S. & Christopher H. Emfinger. (2016). Pancreatic β‐cell identity in diabetes. Diabetes Obesity and Metabolism. 18(S1). 110–116. 97 indexed citations

16.

Zhu, Lin, Melissa Martinez, Christopher H. Emfinger, Brian T. Palmisano, & John M. Stafford. (2014). Estrogen signaling prevents diet-induced hepatic insulin resistance in male mice with obesity. American Journal of Physiology-Endocrinology and Metabolism. 306(10). E1188–E1197. 94 indexed citations

17.

Palmisano, Brian T., et al.. (2013). Cholesteryl ester transfer protein protects against insulin resistance in obese female mice. Molecular Metabolism. 2(4). 457–467. 23 indexed citations

18.

Martinez, Melissa, Christopher H. Emfinger, Salisha Hill, et al.. (2012). Obesity and altered glucose metabolism impact HDL composition in CETP transgenic mice: a role for ovarian hormones. Journal of Lipid Research. 53(3). 379–389. 30 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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