Christopher Gully

595 total citations
8 papers, 407 citations indexed

About

Christopher Gully is a scholar working on Molecular Biology, Oncology and Pathology and Forensic Medicine. According to data from OpenAlex, Christopher Gully has authored 8 papers receiving a total of 407 indexed citations (citations by other indexed papers that have themselves been cited), including 5 papers in Molecular Biology, 4 papers in Oncology and 3 papers in Pathology and Forensic Medicine. Recurrent topics in Christopher Gully's work include Cancer Mechanisms and Therapy (3 papers), Peptidase Inhibition and Analysis (3 papers) and Microtubule and mitosis dynamics (3 papers). Christopher Gully is often cited by papers focused on Cancer Mechanisms and Therapy (3 papers), Peptidase Inhibition and Analysis (3 papers) and Microtubule and mitosis dynamics (3 papers). Christopher Gully collaborates with scholars based in United States, Japan and Australia. Christopher Gully's co-authors include Sai‐Ching J. Yeung, Mong‐Hong Lee, Guermarie Velázquez-Torres, Stephanie Darmanin, Masanobu Kobayashi, Ran Zhao, Ruiying Zhao, Fanmao Zhang, Jian Chen and Yin‐Hsun Feng and has published in prestigious journals such as Cancer Research, Oncogene and American Journal Of Pathology.

In The Last Decade

Christopher Gully

7 papers receiving 404 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Christopher Gully United States	7	281	221	126	87	66	8	407
Akito Nakamura Japan	8	326 1.2×	119 0.5×	63 0.5×	58 0.7×	56 0.8×	13	424
Kazuhiro Ohara Japan	4	346 1.2×	138 0.6×	85 0.7×	40 0.5×	30 0.5×	6	407
Jienan Kong China	9	293 1.0×	113 0.5×	48 0.4×	53 0.6×	62 0.9×	13	408
Vishnu Kumarasamy United States	14	271 1.0×	360 1.6×	33 0.3×	68 0.8×	103 1.6×	22	621
Saveur-Michel Maira Switzerland	6	323 1.1×	79 0.4×	59 0.5×	31 0.4×	51 0.8×	8	424
Tatiana Y. Prudnikova Russia	13	267 1.0×	97 0.4×	32 0.3×	149 1.7×	78 1.2×	21	404
Marie Villedieu France	10	416 1.5×	151 0.7×	61 0.5×	31 0.4×	195 3.0×	10	531
Danrui Cui China	14	563 2.0×	220 1.0×	34 0.3×	56 0.6×	92 1.4×	27	655
Steven Do United States	7	293 1.0×	154 0.7×	104 0.8×	23 0.3×	37 0.6×	10	499
Victoria Lock United Kingdom	9	207 0.7×	187 0.8×	43 0.3×	126 1.4×	30 0.5×	11	353

Countries citing papers authored by Christopher Gully

Since Specialization

Citations

This map shows the geographic impact of Christopher Gully's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Christopher Gully with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Christopher Gully more than expected).

Fields of papers citing papers by Christopher Gully

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Christopher Gully. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Christopher Gully. The network helps show where Christopher Gully may publish in the future.

Co-authorship network of co-authors of Christopher Gully

This figure shows the co-authorship network connecting the top 25 collaborators of Christopher Gully. A scholar is included among the top collaborators of Christopher Gully based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Christopher Gully. Christopher Gully is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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8 of 8 papers shown

1.

Choi, Hyun Ho, Christopher Gully, Guermarie Velázquez-Torres, et al.. (2011). COP9 signalosome subunit 6 stabilizes COP1, which functions as an E3 ubiquitin ligase for 14-3-3σ. Oncogene. 30(48). 4791–4801. 44 indexed citations

2.

Gully, Christopher, Fanmao Zhang, Jian Chen, et al.. (2010). Antineoplastic effects of an Aurora B kinase inhibitor in breast cancer. Molecular Cancer. 9(1). 42–42. 87 indexed citations

3.

Su, Chun‐Hui, Ruiying Zhao, Guermarie Velázquez-Torres, et al.. (2010). Nuclear export regulation of COP1 by 14-3-3σ in response to DNA damage. Molecular Cancer. 9(1). 243–243. 38 indexed citations

4.

Feng, Yin‐Hsun, Guermarie Velázquez-Torres, Christopher Gully, et al.. (2010). The impact of type 2 diabetes and antidiabetic drugs on cancer cell growth. Journal of Cellular and Molecular Medicine. 15(4). 825–836. 67 indexed citations

5.

Chen, Jian, Masanobu Kobayashi, Stephanie Darmanin, et al.. (2010). Abstract 457: Hypoxia-mediated upregulation of Pim-1 contributes to tumor survival. Cancer Research. 70(8_Supplement). 457–457.

6.

Kobayashi, Masanobu, Stephanie Darmanin, Christopher Gully, et al.. (2009). Pim-1 plays a pivotal role in hypoxia-induced chemoresistance. Oncogene. 28(28). 2581–2592. 80 indexed citations

7.

Chen, Jian, Masanobu Kobayashi, Stephanie Darmanin, et al.. (2009). Hypoxia-Mediated Up-Regulation of Pim-1 Contributes to Solid Tumor Formation. American Journal Of Pathology. 175(1). 400–411. 76 indexed citations

8.

Yeung, Sai‐Ching J., Christopher Gully, & Mong‐Hong Lee. (2008). Aurora-B Kinase Inhibitors for Cancer Chemotherapy. Mini-Reviews in Medicinal Chemistry. 8(14). 1514–1525. 15 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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