Chizu Tanikawa

5.8k total citations · 1 hit paper
57 papers, 3.1k citations indexed

About

Chizu Tanikawa is a scholar working on Molecular Biology, Oncology and Surgery. According to data from OpenAlex, Chizu Tanikawa has authored 57 papers receiving a total of 3.1k indexed citations (citations by other indexed papers that have themselves been cited), including 24 papers in Molecular Biology, 21 papers in Oncology and 10 papers in Surgery. Recurrent topics in Chizu Tanikawa's work include Cancer-related Molecular Pathways (16 papers), Helicobacter pylori-related gastroenterology studies (9 papers) and RNA modifications and cancer (7 papers). Chizu Tanikawa is often cited by papers focused on Cancer-related Molecular Pathways (16 papers), Helicobacter pylori-related gastroenterology studies (9 papers) and RNA modifications and cancer (7 papers). Chizu Tanikawa collaborates with scholars based in Japan, United States and Canada. Chizu Tanikawa's co-authors include Koichi Matsuda, Yusuke Nakamura, Hirofumi Arakawa, Katsutoshi Oda, Katsuyuki Tamai, Yoichi Taya, Hiroyuki Nishimori, Takashi Tokino, Toshiki Mori and Tomoaki Tanaka and has published in prestigious journals such as Cell, Journal of Clinical Investigation and Nature Communications.

In The Last Decade

Chizu Tanikawa

56 papers receiving 3.1k citations

Hit Papers

p53AIP1, a Potential Mediator of p53-Dependent Apoptosis,... 2000 2026 2008 2017 2000 250 500 750

Peers

Chizu Tanikawa
Michel M. Ouellette United States
Chizu Tanikawa
Citations per year, relative to Chizu Tanikawa Chizu Tanikawa (= 1×) peers Michel M. Ouellette

Countries citing papers authored by Chizu Tanikawa

Since Specialization
Citations

This map shows the geographic impact of Chizu Tanikawa's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Chizu Tanikawa with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Chizu Tanikawa more than expected).

Fields of papers citing papers by Chizu Tanikawa

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Chizu Tanikawa. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Chizu Tanikawa. The network helps show where Chizu Tanikawa may publish in the future.

Co-authorship network of co-authors of Chizu Tanikawa

This figure shows the co-authorship network connecting the top 25 collaborators of Chizu Tanikawa. A scholar is included among the top collaborators of Chizu Tanikawa based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Chizu Tanikawa. Chizu Tanikawa is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Namba, Shinichi, Masato Akiyama, Haruka Hamanoue, et al.. (2024). Inconsistent embryo selection across polygenic score methods. Nature Human Behaviour. 8(12). 2264–2267. 1 indexed citations
2.
Koguchi, Tomoyuki, Hao Liu, Masanori Kakuta, et al.. (2023). Regulation of the innate immune response and gut microbiome by p53. Cancer Science. 115(1). 184–196. 4 indexed citations
3.
He, Yunye, Masaru Koido, Yoichi Sutoh, et al.. (2023). East Asian-specific and cross-ancestry genome-wide meta-analyses provide mechanistic insights into peptic ulcer disease. Nature Genetics. 55(12). 2129–2138. 9 indexed citations
4.
Shi, Mingyang, Chizu Tanikawa, Hans Markus Münter, et al.. (2023). Genotype imputation accuracy and the quality metrics of the minor ancestry in multi-ancestry reference panels. Briefings in Bioinformatics. 25(1). 2 indexed citations
5.
Rojsitthisak, Pornchai, et al.. (2021). Collagen XVII inhibits breast cancer cell proliferation and growth through deactivation of the AKT/mTOR signaling pathway. PLoS ONE. 16(7). e0255179–e0255179. 25 indexed citations
6.
Onabajo, Olusegun O., Fang Wang, Mei–Hsuan Lee, et al.. (2021). Intracellular Accumulation of IFN-λ4 Induces ER Stress and Results in Anti-Cirrhotic but Pro-HCV Effects. Frontiers in Immunology. 12. 692263–692263. 8 indexed citations
7.
Okumura, Kazuhiro, Eriko Isogai, Kimi Araki, et al.. (2019). A Polymorphic Variant in p19Arf Confers Resistance to Chemically Induced Skin Tumors by Activating the p53 Pathway. Journal of Investigative Dermatology. 139(7). 1459–1469. 4 indexed citations
8.
Howles, Sarah, Akira Wiberg, Michelle Goldsworthy, et al.. (2019). Genetic variants of calcium and vitamin D metabolism in kidney stone disease. Nature Communications. 10(1). 5175–5175. 85 indexed citations
9.
Tsuda, Yusuke, Chizu Tanikawa, Takafumi Miyamoto, et al.. (2017). Identification of a p53 target, CD137L, that mediates growth suppression and immune response of osteosarcoma cells. Scientific Reports. 7(1). 10739–10739. 4 indexed citations
10.
Lo, Paulisally Hau Yi, Chizu Tanikawa, Toyomasa Katagiri, Yusuke Nakamura, & Koichi Matsuda. (2014). Identification of novel epigenetically inactivated gene PAMR1 in breast carcinoma. Oncology Reports. 33(1). 267–273. 23 indexed citations
11.
Tanikawa, Chizu, Yukinori Okada, Atsushi Takahashi, et al.. (2013). Correction: Genome Wide Association Study of Age at Menarche in the Japanese Population. PLoS ONE. 8(10). 12 indexed citations
12.
Lo, Paulisally Hau Yi, Yuji Urabe, Vinod Kumar, et al.. (2013). Identification of a Functional Variant in the MICA Promoter Which Regulates MICA Expression and Increases HCV-Related Hepatocellular Carcinoma Risk. PLoS ONE. 8(4). e61279–e61279. 48 indexed citations
13.
Tanikawa, Chizu, Yukinori Okada, Atsushi Takahashi, et al.. (2013). Genome Wide Association Study of Age at Menarche in the Japanese Population. PLoS ONE. 8(5). e63821–e63821. 33 indexed citations
14.
Tanikawa, Chizu, Yuji Urabe, Keitaro Matsuo, et al.. (2012). A genome-wide association study identifies two susceptibility loci for duodenal ulcer in the Japanese population. Nature Genetics. 44(4). 430–434. 87 indexed citations
15.
Kumar, Vinod, Keitaro Matsuo, Atsushi Takahashi, et al.. (2011). Common variants on 14q32 and 13q12 are associated with DLBCL susceptibility. Journal of Human Genetics. 56(6). 436–439. 16 indexed citations
16.
Tanikawa, Chizu, Koji Ueda, Hidewaki Nakagawa, et al.. (2009). Regulation of Protein Citrullination through p53/PADI4 Network in DNA Damage Response. Cancer Research. 69(22). 8761–8769. 107 indexed citations
17.
Tanikawa, Chizu, Yoichi Furukawa, Naoko Yoshida, et al.. (2009). XEDAR as a putative colorectal tumor suppressor that mediates p53-regulated anoikis pathway. Oncogene. 28(34). 3081–3092. 55 indexed citations
18.
Morioka, Kazuhito, Chizu Tanikawa, Kensuke Ochi, et al.. (2009). Orphan receptor tyrosine kinase ROR2 as a potential therapeutic target for osteosarcoma. Cancer Science. 100(7). 1227–1233. 75 indexed citations
19.
Kidokoro, Tetsuo, Chizu Tanikawa, Yoichi Furukawa, et al.. (2007). CDC20, a potential cancer therapeutic target, is negatively regulated by p53. Oncogene. 27(11). 1562–1571. 173 indexed citations
20.
Oda, Katsutoshi, Hirofumi Arakawa, Tomoaki Tanaka, et al.. (2000). p53AIP1, a Potential Mediator of p53-Dependent Apoptosis, and Its Regulation by Ser-46-Phosphorylated p53. Cell. 102(6). 849–862. 977 indexed citations breakdown →

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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