Chisato Kunitomi

701 total citations
23 papers, 538 citations indexed

About

Chisato Kunitomi is a scholar working on Reproductive Medicine, Public Health, Environmental and Occupational Health and Cell Biology. According to data from OpenAlex, Chisato Kunitomi has authored 23 papers receiving a total of 538 indexed citations (citations by other indexed papers that have themselves been cited), including 17 papers in Reproductive Medicine, 14 papers in Public Health, Environmental and Occupational Health and 7 papers in Cell Biology. Recurrent topics in Chisato Kunitomi's work include Ovarian function and disorders (16 papers), Reproductive Biology and Fertility (14 papers) and Endoplasmic Reticulum Stress and Disease (6 papers). Chisato Kunitomi is often cited by papers focused on Ovarian function and disorders (16 papers), Reproductive Biology and Fertility (14 papers) and Endoplasmic Reticulum Stress and Disease (6 papers). Chisato Kunitomi collaborates with scholars based in Japan, United States and Malaysia. Chisato Kunitomi's co-authors include Yutaka Osuga, Miyuki Harada, Nozomi Takahashi, Jerilee Mariam Khong Azhary, Tomoyuki Fujii, Osamu Wada‐Hiraike, Yasushi Hirota, Kaori Koga, Emi Nose and Tetsuya Hirata and has published in prestigious journals such as Development, Scientific Reports and The FASEB Journal.

In The Last Decade

Chisato Kunitomi

22 papers receiving 534 citations

Peers

Chisato Kunitomi
Chisato Kunitomi
Citations per year, relative to Chisato Kunitomi Chisato Kunitomi (= 1×) peers Nozomi Takahashi

Countries citing papers authored by Chisato Kunitomi

Since Specialization
Citations

This map shows the geographic impact of Chisato Kunitomi's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Chisato Kunitomi with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Chisato Kunitomi more than expected).

Fields of papers citing papers by Chisato Kunitomi

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Chisato Kunitomi. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Chisato Kunitomi. The network helps show where Chisato Kunitomi may publish in the future.

Co-authorship network of co-authors of Chisato Kunitomi

This figure shows the co-authorship network connecting the top 25 collaborators of Chisato Kunitomi. A scholar is included among the top collaborators of Chisato Kunitomi based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Chisato Kunitomi. Chisato Kunitomi is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Koike, Hiroshi, Katsuhiko Noda, Toshihiro Fujiwara, et al.. (2025). Assessment and prediction models for the quantitative and qualitative reserve of the ovary using machine learning. Journal of Ovarian Research. 18(1). 153–153.
3.
Harada, Miyuki, Asami Matsumoto, Kentaro Oka, et al.. (2024). Effects of the prenatal and postnatal nurturing environment on the phenotype and gut microbiota of mice with polycystic ovary syndrome induced by prenatal androgen exposure: a cross-fostering study. Frontiers in Cell and Developmental Biology. 12. 1365624–1365624. 1 indexed citations
4.
Urata, Yoko, Miyuki Harada, Chisato Kunitomi, et al.. (2024). Cellular senescence of granulosa cells in the pathogenesis of polycystic ovary syndrome. Molecular Human Reproduction. 30(5). 6 indexed citations
5.
Conti, Marco & Chisato Kunitomi. (2023). A genome-wide perspective of the maternal mRNA translation program during oocyte development. Seminars in Cell and Developmental Biology. 154(Pt B). 88–98. 17 indexed citations
6.
Takahashi, Nozomi, Miyuki Harada, Chisato Kunitomi, et al.. (2023). The Role of Cellular Senescence in Cyclophosphamide-Induced Primary Ovarian Insufficiency. International Journal of Molecular Sciences. 24(24). 17193–17193. 18 indexed citations
7.
Koike, Hiroshi, Miyuki Harada, Chisato Kunitomi, et al.. (2023). Roles of endoplasmic reticulum stress in the pathophysiology of polycystic ovary syndrome. Frontiers in Endocrinology. 14. 1124405–1124405. 30 indexed citations
8.
Kunitomi, Chisato, Miyuki Harada, Yasushi Takai, et al.. (2022). The possible effects of the Japan Society of Clinical Oncology Clinical Practice Guidelines 2017 on the practice of fertility preservation in female cancer patients in Japan. Reproductive Medicine and Biology. 21(1). 7 indexed citations
9.
Koike, Hisashi, Mutsuo Harada, Chisato Kunitomi, et al.. (2022). P-610 Endoplasmic reticulum stress-induced Notch signaling stimulates cumulus-oocyte complex expansion in PCOS. Human Reproduction. 37(Supplement_1). 1 indexed citations
10.
Kunitomi, Chisato, Miyuki Harada, Jerilee Mariam Khong Azhary, et al.. (2021). Induction of aryl hydrocarbon receptor in granulosa cells by endoplasmic reticulum stress contributes to pathology of polycystic ovary syndrome. Molecular Human Reproduction. 27(3). 21 indexed citations
11.
Harada, Miyuki, Jerilee Mariam Khong Azhary, Chisato Kunitomi, et al.. (2021). Temporal relationship between alterations in the gut microbiome and the development of polycystic ovary syndrome‐like phenotypes in prenatally androgenized female mice. The FASEB Journal. 35(11). 10 indexed citations
12.
Kunitomi, Chisato, et al.. (2021). Upregulation of Aryl Hydrocarbon Receptor in Granulosa Cells by Endoplasmic Reticulum Stress Contributes to the PCOS Pathophysiology. Journal of the Endocrine Society. 5(Supplement_1). A773–A773. 2 indexed citations
13.
Kobayashi, Mutsumi, Osamu Yoshino, Akitoshi Nakashima, et al.. (2020). Inhibition of autophagy in theca cells induces CYP17A1 and PAI-1 expression via ROS/p38 and JNK signalling during the development of polycystic ovary syndrome. Molecular and Cellular Endocrinology. 508. 110792–110792. 39 indexed citations
14.
Azhary, Jerilee Mariam Khong, Miyuki Harada, Nozomi Takahashi, et al.. (2020). MON-033 Androgen Increases the Accumulation of Advanced Glycation End Products in Granulosa Cells by Activating ER Stress in PCOS. Journal of the Endocrine Society. 4(Supplement_1). 1 indexed citations
15.
Azhary, Jerilee Mariam Khong, Miyuki Harada, Chisato Kunitomi, et al.. (2020). Androgens Increase Accumulation of Advanced Glycation End Products in Granulosa Cells by Activating ER Stress in PCOS. Endocrinology. 161(2). 46 indexed citations
16.
Harada, Miyuki, Chisato Kunitomi, Gentaro Izumi, et al.. (2019). A Japanese nationwide survey on the cryopreservation of embryos, oocytes and ovarian tissue for cancer patients. Journal of obstetrics and gynaecology research. 45(10). 2021–2028. 16 indexed citations
17.
Kunitomi, Chisato, Miyuki Harada, Nozomi Takahashi, et al.. (2019). Activation of endoplasmic reticulum stress mediates oxidative stress–induced apoptosis of granulosa cells in ovaries affected by endometrioma. Molecular Human Reproduction. 26(1). 40–52. 39 indexed citations
18.
Wada‐Hiraike, Osamu, Chisato Kunitomi, Jerilee Mariam Khong Azhary, et al.. (2019). Role of oxidative stress in follicular fluid on embryos of patients undergoing assisted reproductive technology treatment. Journal of obstetrics and gynaecology research. 45(9). 1884–1891. 14 indexed citations
19.
Takahashi, Nozomi, Miyuki Harada, Jerilee Mariam Khong Azhary, et al.. (2019). Accumulation of advanced glycation end products in follicles is associated with poor oocyte developmental competence. Molecular Human Reproduction. 25(11). 684–694. 37 indexed citations
20.
Takahashi, Nozomi, Miyuki Harada, Yasushi Hirota, et al.. (2017). Activation of Endoplasmic Reticulum Stress in Granulosa Cells from Patients with Polycystic Ovary Syndrome Contributes to Ovarian Fibrosis. Scientific Reports. 7(1). 10824–10824. 98 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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