Cheol O. Joe

6.6k total citations
74 papers, 2.2k citations indexed

About

Cheol O. Joe is a scholar working on Molecular Biology, Oncology and Cancer Research. According to data from OpenAlex, Cheol O. Joe has authored 74 papers receiving a total of 2.2k indexed citations (citations by other indexed papers that have themselves been cited), including 58 papers in Molecular Biology, 15 papers in Oncology and 13 papers in Cancer Research. Recurrent topics in Cheol O. Joe's work include Cell death mechanisms and regulation (9 papers), DNA Repair Mechanisms (8 papers) and Ubiquitin and proteasome pathways (7 papers). Cheol O. Joe is often cited by papers focused on Cell death mechanisms and regulation (9 papers), DNA Repair Mechanisms (8 papers) and Ubiquitin and proteasome pathways (7 papers). Cheol O. Joe collaborates with scholars based in South Korea, United States and Japan. Cheol O. Joe's co-authors include Jin Woo Kim, Doo Yeon Kim, Eui‐Ju Choi, Soo Kyung Koo, Yoonseok Kam, Ke Won Kang, Ji Hoon Jeong, Tae Gwan Park, Jeong Keun Ahn and Jungyeon Won and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Nature Communications.

In The Last Decade

Cheol O. Joe

74 papers receiving 2.1k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Cheol O. Joe South Korea 27 1.6k 300 260 214 213 74 2.2k
Boo Ahn Shin South Korea 21 1.2k 0.8× 244 0.8× 218 0.8× 231 1.1× 297 1.4× 41 2.1k
Shin‐Il Kim South Korea 31 1.9k 1.2× 192 0.6× 154 0.6× 162 0.8× 275 1.3× 71 2.5k
Yung Joon Yoo South Korea 23 1.5k 1.0× 310 1.0× 214 0.8× 128 0.6× 194 0.9× 54 2.2k
Michael L. Sprengart Singapore 6 1.8k 1.2× 460 1.5× 205 0.8× 275 1.3× 301 1.4× 6 2.5k
Byoung Chul Park South Korea 30 1.6k 1.0× 287 1.0× 215 0.8× 158 0.7× 267 1.3× 107 2.3k
Salvatore Feo Italy 29 1.6k 1.0× 417 1.4× 576 2.2× 184 0.9× 283 1.3× 76 2.6k
Asha Acharya United States 24 2.6k 1.6× 267 0.9× 473 1.8× 259 1.2× 176 0.8× 33 3.4k
Wenfang Wang China 25 1.2k 0.8× 220 0.7× 182 0.7× 235 1.1× 224 1.1× 98 2.3k
Michael Lee South Korea 24 1.2k 0.8× 297 1.0× 305 1.2× 107 0.5× 197 0.9× 101 2.0k
Chang Bai United States 15 1.5k 1.0× 409 1.4× 156 0.6× 195 0.9× 160 0.8× 27 2.3k

Countries citing papers authored by Cheol O. Joe

Since Specialization
Citations

This map shows the geographic impact of Cheol O. Joe's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Cheol O. Joe with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Cheol O. Joe more than expected).

Fields of papers citing papers by Cheol O. Joe

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Cheol O. Joe. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Cheol O. Joe. The network helps show where Cheol O. Joe may publish in the future.

Co-authorship network of co-authors of Cheol O. Joe

This figure shows the co-authorship network connecting the top 25 collaborators of Cheol O. Joe. A scholar is included among the top collaborators of Cheol O. Joe based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Cheol O. Joe. Cheol O. Joe is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Kwak, Cheol, Kyung Hwa Kang, Seyun Kim, et al.. (2015). Amino acid-dependent NPRL2 interaction with Raptor determines mTOR Complex 1 activation. Cellular Signalling. 28(2). 32–41. 12 indexed citations
2.
Kwak, Cheol, Jae Ho Lee, Ji Hye Choi, et al.. (2011). Alkaline stress‐induced autophagy is mediated by mTORC1 inactivation. Journal of Cellular Biochemistry. 112(9). 2566–2573. 13 indexed citations
3.
Lee, Jae Ho, Cheol Kwak, Jin Woo Kim, et al.. (2009). Notch Signal Activates Hypoxia Pathway through HES1-Dependent SRC/Signal Transducers and Activators of Transcription 3 Pathway. Molecular Cancer Research. 7(10). 1663–1671. 37 indexed citations
4.
Mo, Jung‐Soon, Mi‐Yeon Kim, In‐Sook Kim, et al.. (2007). Integrin-Linked Kinase Controls Notch1 Signaling by Down-Regulation of Protein Stability through Fbw7 Ubiquitin Ligase. Molecular and Cellular Biology. 27(15). 5565–5574. 54 indexed citations
5.
Joe, Cheol O., et al.. (2007). BTB/POZ domain of speckle‐type POZ protein (SPOP) confers proapoptotic function in HeLa cells. BioFactors. 31(3-4). 165–169. 15 indexed citations
6.
Won, Jungyeon, et al.. (2006). Dose-dependent UV Stabilization of p53 in Cultured Human Cells Undergoing Apoptosis Is Mediated by Poly(ADP-ribosyl)ation. Molecules and Cells. 21(2). 218–223. 11 indexed citations
7.
Kwon, Jeong Eun, Young Mi Oh, Jae Hong Seol, et al.. (2006). BTB Domain-containing Speckle-type POZ Protein (SPOP) Serves as an Adaptor of Daxx for Ubiquitination by Cul3-based Ubiquitin Ligase. Journal of Biological Chemistry. 281(18). 12664–12672. 167 indexed citations
8.
Kim, Doyeun, Jungyeon Won, Dong Wook Shin, et al.. (2004). Regulation of Dyrk1A kinase activity by 14-3-3. Biochemical and Biophysical Research Communications. 323(2). 499–504. 28 indexed citations
9.
Kim, Karam, Jungyeon Won, Jeung‐Hoon Lee, et al.. (2004). Daxx-mediated transcriptional repression of MMP1 gene is reversed by SPOP. Biochemical and Biophysical Research Communications. 320(3). 760–765. 16 indexed citations
11.
Cho, Su Jin, et al.. (2001). Tob-Mediated Cross-Talk between MARCKS Phosphorylation and ErbB-2 Activation. Biochemical and Biophysical Research Communications. 283(2). 273–277. 25 indexed citations
12.
Kim, Jin Woo, Cheol O. Joe, & Eui‐Ju Choi. (2001). Role of Receptor-interacting Protein in Tumor Necrosis Factor-α-dependent MEKK1 Activation. Journal of Biological Chemistry. 276(29). 27064–27070. 30 indexed citations
13.
Kim, Jin Woo, Tong-Shin Chang, Ji Eun Lee, et al.. (2001). Negative Regulation of the Sapk/Jnk Signaling Pathway by Presenilin 1. The Journal of Cell Biology. 153(3). 457–464. 25 indexed citations
14.
Kam, Yoonseok, Doo Yeon Kim, Soo Kyung Koo, & Cheol O. Joe. (1998). Transfer of second messengers through gap junction connexin 43 channels reconstituted in liposomes. Biochimica et Biophysica Acta (BBA) - Biomembranes. 1372(2). 384–388. 48 indexed citations
15.
Shin, Incheol, et al.. (1996). Inhibition of the phosphorylation of a myristoylated alanine-rich C kinase substrate by methyl methanesulfonate in cultured NIH 3T3 cells. Mutation research. Fundamental and molecular mechanisms of mutagenesis. 351(2). 163–171. 3 indexed citations
16.
Rhee, Sang Ki, et al.. (1993). The Leech as a Laboratory Animal for the biomedical Research. KRIBB Repository. 36(4). 588–595. 2 indexed citations
17.
Shin, Incheol, et al.. (1993). INHIBITION OF 80 kDa PROTEIN PHOSPHORYLATION BY SHORT‐WAVELENGTH UV LIGHT IN NIH 3T3 CELLS. Photochemistry and Photobiology. 58(4). 536–540. 1 indexed citations
18.
Sylvia, Victor L., et al.. (1989). Interaction of phosphatidylinositol-4-monophosphate with a low activity form of dna polymerase alpha: a potential mechanism for enzyme activation. International Journal of Biochemistry. 21(4). 347–353. 10 indexed citations
19.
Joe, Cheol O., et al.. (1986). Phosphatidylinositol-dependent activation of DNA polymerase alpha. Biochemical and Biophysical Research Communications. 135(3). 880–885. 22 indexed citations
20.
Busbee, David L., Cheol O. Joe, James O’Higgins Norman, & Patrick W. Rankin. (1984). Inhibition of DNA synthesis by an electrophilic metabolite of benzo[a]pyrene.. Proceedings of the National Academy of Sciences. 81(17). 5300–5304. 16 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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