Cheng Ai

1.4k total citations
25 papers, 362 citations indexed

About

Cheng Ai is a scholar working on Molecular Biology, Oncology and Surgery. According to data from OpenAlex, Cheng Ai has authored 25 papers receiving a total of 362 indexed citations (citations by other indexed papers that have themselves been cited), including 11 papers in Molecular Biology, 7 papers in Oncology and 4 papers in Surgery. Recurrent topics in Cheng Ai's work include Mitochondrial Function and Pathology (6 papers), ATP Synthase and ATPases Research (4 papers) and Multiple Myeloma Research and Treatments (3 papers). Cheng Ai is often cited by papers focused on Mitochondrial Function and Pathology (6 papers), ATP Synthase and ATPases Research (4 papers) and Multiple Myeloma Research and Treatments (3 papers). Cheng Ai collaborates with scholars based in China, United States and New Zealand. Cheng Ai's co-authors include Min‐Xin Guan, Hailin Liu, Xiaogang Dai, Feng Hu, Alun Li, Nan Hu, Fuliang Cao, Tongming Yin, Jue Ruan and Shigang Wu and has published in prestigious journals such as Nucleic Acids Research, Journal of Clinical Investigation and Medicine.

In The Last Decade

Cheng Ai

23 papers receiving 360 citations

Peers

Cheng Ai
Lei Peng China
Chong Xu United States
Giorgia Testoni Argentina
Bai Qin China
Cheng Ai
Citations per year, relative to Cheng Ai Cheng Ai (= 1×) peers Xiaolan Liu

Countries citing papers authored by Cheng Ai

Since Specialization
Citations

This map shows the geographic impact of Cheng Ai's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Cheng Ai with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Cheng Ai more than expected).

Fields of papers citing papers by Cheng Ai

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Cheng Ai. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Cheng Ai. The network helps show where Cheng Ai may publish in the future.

Co-authorship network of co-authors of Cheng Ai

This figure shows the co-authorship network connecting the top 25 collaborators of Cheng Ai. A scholar is included among the top collaborators of Cheng Ai based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Cheng Ai. Cheng Ai is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
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Ai, Cheng, Chunyan Wang, Yanchun Ji, et al.. (2025). Vitamin A treatment restores vision failures arising from Leber’s hereditary optic neuropathy–linked mtDNA mutation. JCI Insight. 10(8). 1 indexed citations
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Wang, Chenghui, Min Liang, Hanqing Liu, et al.. (2024). Mutation of CRYAB encoding a conserved mitochondrial chaperone and antiapoptotic protein causes hereditary optic atrophy. JCI Insight. 10(1). 4 indexed citations
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Gao, Limin, et al.. (2023). Solitary extramedullary plasmacytoma of the left main bronchus: A case report and literature review. Thoracic Cancer. 14(4). 419–422. 1 indexed citations
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Wang, Jing, Yanchun Ji, Cheng Ai, et al.. (2023). Optimized allotopic expression of mitochondrial ND6 transgene restored complex I and apoptosis deficiencies caused by LHON-linked ND6 14484T > C mutation. Journal of Biomedical Science. 30(1). 63–63. 11 indexed citations
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Tan, Xiang, Tao Xu, Shen Wang, et al.. (2023). Primary pulmonary adenoid cystic carcinoma: A clinicopathological study of 64 patients. Thoracic Cancer. 15(5). 386–393. 3 indexed citations
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Liu, Hailin, Xiaobo Wang, Guibin Wang, et al.. (2021). The nearly complete genome of Ginkgo biloba illuminates gymnosperm evolution. Nature Plants. 7(6). 748–756. 144 indexed citations
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Yu, Jialing, Xiaoyang Liang, Yanchun Ji, et al.. (2020). PRICKLE3 linked to ATPase biogenesis manifested Leber’s hereditary optic neuropathy. Journal of Clinical Investigation. 130(9). 4935–4946. 48 indexed citations
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Ai, Cheng, Guangzhi Ma, Yunfu Deng, et al.. (2020). Nm23‐H1 inhibits lung cancer bone‐specific metastasis by upregulating miR‐660‐5p targeted SMARCA5. Thoracic Cancer. 11(3). 640–650. 26 indexed citations
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Ma, Guangzhi, Yunfu Deng, Wenjie Chen, et al.. (2019). The Prognostic Role of MET Protein Expression Among Surgically Resected Non-small Cell Lung Cancer Patients: A Meta-Analysis. Frontiers in Oncology. 9. 9 indexed citations
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Dai, Yu, Chenghui Wang, Jiaming Han, et al.. (2017). Mutation analysis of Leber's hereditary optic neuropathy using a multi‑gene panel. Biomedical Reports. 8(1). 51–58. 30 indexed citations
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Zhang, Linlin, Lin Li, Huijun Wei, et al.. (2015). Transcriptional factor FOXO3 negatively regulates the expression of nm23‐H1 in non‐small cell lung cancer. Thoracic Cancer. 7(1). 9–16. 17 indexed citations
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Ai, Cheng. (2008). Comparative Proteomics Analysis of LCM Purified Nasopharyngeal Carcinoma and Normal Nasopharyngeal Epithelial Tissue. PROGRESS IN BIOCHEMISTRY AND BIOPHYSICS. 3 indexed citations
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Ai, Cheng. (2002). Low calorie parenteral nutrition in the elderly abdominal surgical patients. Zhonghua putong waike zazhi. 1 indexed citations
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Ai, Cheng. (2001). Intraperitoneal chemotherapy for ovarian cancer. 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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