Changlian Lu

748 total citations
44 papers, 556 citations indexed

About

Changlian Lu is a scholar working on Molecular Biology, Cancer Research and Pulmonary and Respiratory Medicine. According to data from OpenAlex, Changlian Lu has authored 44 papers receiving a total of 556 indexed citations (citations by other indexed papers that have themselves been cited), including 22 papers in Molecular Biology, 16 papers in Cancer Research and 10 papers in Pulmonary and Respiratory Medicine. Recurrent topics in Changlian Lu's work include Cancer-related molecular mechanisms research (6 papers), RNA modifications and cancer (6 papers) and Eicosanoids and Hypertension Pharmacology (5 papers). Changlian Lu is often cited by papers focused on Cancer-related molecular mechanisms research (6 papers), RNA modifications and cancer (6 papers) and Eicosanoids and Hypertension Pharmacology (5 papers). Changlian Lu collaborates with scholars based in China, Belgium and United States. Changlian Lu's co-authors include Xuefeng Gu, Lili Deng, Guoqing Wan, Yuexian Hu, R. Grant Rowe, Stephen J. Weiss, Xiaoyan Li, Lingyun Zhou, Xiaojiao Sun and Daling Zhu and has published in prestigious journals such as SHILAP Revista de lepidopterología, Blood and British Journal of Cancer.

In The Last Decade

Changlian Lu

41 papers receiving 549 citations

Peers

Changlian Lu
Monika D. Polewski United States
Fenghua Zhou China
Minjie Luo United States
Richard de Goeij-de Haas Netherlands
Walid J. Azar Australia
Degui Wang China
Sarah Short United States
Eliot Fletcher-Sananikone United States
Delphine Bacq France
Khadijeh Bijangi‐Vishehsaraei United States
Monika D. Polewski United States
Changlian Lu
Citations per year, relative to Changlian Lu Changlian Lu (= 1×) peers Monika D. Polewski

Countries citing papers authored by Changlian Lu

Since Specialization
Citations

This map shows the geographic impact of Changlian Lu's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Changlian Lu with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Changlian Lu more than expected).

Fields of papers citing papers by Changlian Lu

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Changlian Lu. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Changlian Lu. The network helps show where Changlian Lu may publish in the future.

Co-authorship network of co-authors of Changlian Lu

This figure shows the co-authorship network connecting the top 25 collaborators of Changlian Lu. A scholar is included among the top collaborators of Changlian Lu based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Changlian Lu. Changlian Lu is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Zhu, Xinping, et al.. (2025). Berberine overcomes paclitaxel resistance in triple-negative breast cancer by inhibiting HSP90-mediated BRCA1 phosphorylation. Experimental Cell Research. 454(1). 114815–114815.
2.
Li, Jingyi, Ziyu Chen, Mingming Jin, et al.. (2024). Histone H4K12 lactylation promotes malignancy progression in triple-negative breast cancer through SLFN5 downregulation. Cellular Signalling. 124. 111468–111468. 23 indexed citations
3.
He, Lan, et al.. (2024). Melittin treatment suppressed malignant NSCLC progression through enhancing CTSB-mediated hyperautophagy. Biomedicine & Pharmacotherapy. 180. 117573–117573. 5 indexed citations
4.
Liu, Jiao, Xiaodan Li, Meng Qian, et al.. (2024). Melittin suppresses aerobic glycolysis by regulating HSF1/PDK3 to increase chemosensitivity of NSCLC. European Journal of Pharmacology. 986. 177084–177084. 5 indexed citations
5.
Ma, Wenhao, et al.. (2023). Identification of mitochondrial-related genes as potential biomarkers for the subtyping and prediction of Alzheimer’s disease. Frontiers in Molecular Neuroscience. 16. 1205541–1205541. 4 indexed citations
6.
Gu, Xuefeng, Yue Shao, Wenhao Ma, et al.. (2023). Gene expression changes reveal the impact of the space environment on the skin of International Space Station astronauts. Clinical and Experimental Dermatology. 48(10). 1128–1137. 1 indexed citations
7.
Gu, Xuefeng, Jingyi Li, Kai‐Jie Chen, et al.. (2022). The Long Noncoding RNA MEG3 Retains Epithelial-Mesenchymal Transition by Sponging miR-146b-5p to Regulate SLFN5 Expression in Breast Cancer Cells. Journal of Immunology Research. 2022. 1–17. 6 indexed citations
8.
Zheng, Yanjun, et al.. (2022). Analysis of Time Series Gene Expression and DNA Methylation Reveals the Molecular Features of Myocardial Infarction Progression. Frontiers in Cardiovascular Medicine. 9. 912454–912454. 7 indexed citations
9.
Chen, Kai‐Jie, Shuang Liu, Changlian Lu, & Xuefeng Gu. (2022). A prognostic and therapeutic hallmark developed by the integrated profile of basement membrane and immune infiltrative landscape in lung adenocarcinoma. Frontiers in Immunology. 13. 1058493–1058493. 7 indexed citations
10.
Chen, Kai‐Jie, Beibei Liang, Wenhao Ma, et al.. (2022). Immunological and prognostic analysis of PSENEN in low-grade gliomas: An immune infiltration-related prognostic biomarker. Frontiers in Molecular Neuroscience. 15. 933855–933855. 7 indexed citations
11.
12.
Wang, Feng, Ning Zhao, Ge Gao, et al.. (2020). Prognostic value of TP53 co-mutation status combined with EGFR mutation in patients with lung adenocarcinoma. Journal of Cancer Research and Clinical Oncology. 146(11). 2851–2859. 27 indexed citations
13.
Wan, Guoqing, Xuefeng Gu, Yihao Liu, et al.. (2020). Human Schlafen 5 regulates reversible epithelial and mesenchymal transitions in breast cancer by suppression of ZEB1 transcription. British Journal of Cancer. 123(4). 633–643. 19 indexed citations
14.
Gu, Xuefeng, Guoqing Wan, Yue Yang, et al.. (2020). SLFN5 influences proliferation and apoptosis by upregulating PTEN transcription via ZEB1 and inhibits the purine metabolic pathway in breast cancer.. PubMed. 10(9). 2832–2850. 19 indexed citations
15.
Gu, Xuefeng, Guoqing Wan, Nianhong Chen, et al.. (2019). DGKζ Plays Crucial Roles in the Proliferation and Tumorigenicity of Human glioblastoma. International Journal of Biological Sciences. 15(9). 1872–1881. 13 indexed citations
16.
17.
Zhao, Xiaodan, et al.. (2016). Schedule-dependent cytotoxicity of sunitinib and TRAIL in human non-small cell lung cancer cells with or without EGFR and KRAS mutations. Cellular Oncology. 39(4). 343–352. 11 indexed citations
18.
Zhuo, Qin, et al.. (2015). PDGF-D promotes dermal fibroblast invasion in 3-dimensional extracellular matrix via Snail-mediated MT1-MMP upregulation. Tumor Biology. 37(1). 591–599. 4 indexed citations
19.
Deng, Lili, Changlian Lu, Yang Guo, et al.. (2014). Mutations of EGFR or KRAS and expression of chemotherapy-related genes based on small biopsy samples in stage IIIB and IV inoperable non-small cell lung cancer. Journal of Cancer Research and Clinical Oncology. 140(12). 2097–2105. 7 indexed citations
20.
Liu, Ye, et al.. (2009). Expression of 15-lipoxygenases in pulmonary arteries after hypoxia. Pathology. 41(5). 476–483. 6 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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