Changhui Diao

477 total citations
9 papers, 387 citations indexed

About

Changhui Diao is a scholar working on Molecular Biology, Oncology and Epidemiology. According to data from OpenAlex, Changhui Diao has authored 9 papers receiving a total of 387 indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Molecular Biology, 2 papers in Oncology and 2 papers in Epidemiology. Recurrent topics in Changhui Diao's work include Heme Oxygenase-1 and Carbon Monoxide (3 papers), Cancer-related gene regulation (3 papers) and Autophagy in Disease and Therapy (2 papers). Changhui Diao is often cited by papers focused on Heme Oxygenase-1 and Carbon Monoxide (3 papers), Cancer-related gene regulation (3 papers) and Autophagy in Disease and Therapy (2 papers). Changhui Diao collaborates with scholars based in China. Changhui Diao's co-authors include Hao Liu, Xiuheng Liu, Xiaodong Weng, Yang Du, Lei Wang, Hui Chen, Zhiyuan Chen, Tao Qiu, Yuanyuan Yang and Lei Wang and has published in prestigious journals such as The FASEB Journal, Oxidative Medicine and Cellular Longevity and Redox Biology.

In The Last Decade

Changhui Diao

7 papers receiving 384 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Changhui Diao China 5 256 86 57 39 36 9 387
Hsiao‐Fen Li Taiwan 11 213 0.8× 71 0.8× 74 1.3× 59 1.5× 39 1.1× 12 383
Zhimei Lv China 12 234 0.9× 149 1.7× 73 1.3× 55 1.4× 46 1.3× 15 460
Chakradhar Velagapudi United States 8 254 1.0× 77 0.9× 47 0.8× 50 1.3× 51 1.4× 11 463
Bridgit B. Bowskill Canada 10 237 0.9× 166 1.9× 44 0.8× 59 1.5× 68 1.9× 11 457
Daoyuan Lv China 9 229 0.9× 64 0.7× 47 0.8× 30 0.8× 23 0.6× 11 381
Yu Lin China 11 191 0.7× 83 1.0× 31 0.5× 43 1.1× 51 1.4× 23 445
Xiaoqiang Ding China 11 229 0.9× 97 1.1× 132 2.3× 41 1.1× 43 1.2× 21 412
Makoto Kuro-o United States 7 245 1.0× 80 0.9× 25 0.4× 46 1.2× 67 1.9× 9 595
Lin Mu China 10 211 0.8× 137 1.6× 36 0.6× 61 1.6× 68 1.9× 22 420

Countries citing papers authored by Changhui Diao

Since Specialization
Citations

This map shows the geographic impact of Changhui Diao's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Changhui Diao with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Changhui Diao more than expected).

Fields of papers citing papers by Changhui Diao

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Changhui Diao. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Changhui Diao. The network helps show where Changhui Diao may publish in the future.

Co-authorship network of co-authors of Changhui Diao

This figure shows the co-authorship network connecting the top 25 collaborators of Changhui Diao. A scholar is included among the top collaborators of Changhui Diao based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Changhui Diao. Changhui Diao is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

9 of 9 papers shown
1.
Zhang, Xiaoli, Changhui Diao, Jinglin Zhang, et al.. (2025). [68Ga]Ga-NOTA-C-IPB-AIT: A novel radiotracer for in vivo detection of TREM-1 expression. Bioorganic Chemistry. 162. 108611–108611.
2.
Zhou, Bo, Y. Wang, Changhui Diao, et al.. (2025). A novel 68Ga-labeled cyclic peptide: A potential radiotracer for PET imaging of CD36-positive cancers. Bioorganic Chemistry. 161. 108515–108515.
3.
Yuan, Chao, Lin Liu, Yang Du, et al.. (2023). PCMT1 regulates the migration, invasion, and apoptosis of prostate cancer through modulating the PI3K/AKT/GSK-3β pathway. Aging. 15(20). 11654–11671. 3 indexed citations
4.
Liu, Hao, Lei Wang, Xiaodong Weng, et al.. (2019). Inhibition of Brd4 alleviates renal ischemia/reperfusion injury-induced apoptosis and endoplasmic reticulum stress by blocking FoxO4-mediated oxidative stress. Redox Biology. 24. 101195–101195. 186 indexed citations
5.
Diao, Changhui, et al.. (2019). <p>Aged kidneys are refractory to autophagy activation in a rat model of renal ischemia-reperfusion injury</p>. Clinical Interventions in Aging. Volume 14. 525–534. 19 indexed citations
6.
Liu, Hao, Zhi-yuan Chen, Xiaodong Weng, et al.. (2019). Enhancer of zeste homolog 2 modulates oxidative stress‐mediated pyroptosis in vitro and in a mouse kidney ischemia‐reperfusion injury model. The FASEB Journal. 34(1). 835–852. 47 indexed citations
7.
Diao, Changhui, et al.. (2019). Inhibition of PRMT5 Attenuates Oxidative Stress-Induced Pyroptosis via Activation of the Nrf2/HO-1 Signal Pathway in a Mouse Model of Renal Ischemia-Reperfusion Injury. Oxidative Medicine and Cellular Longevity. 2019. 1–18. 107 indexed citations
8.
Gao, Xiaoning, et al.. (2018). [Expression of BRD4 in squamous cell carcinoma and its effects on cell proliferation and invasion ability].. PubMed. 47(5). 344–348. 2 indexed citations
9.
Shen, Hao, Xiaodong Weng, Xiuheng Liu, et al.. (2018). miR-181a-5p is downregulated and inhibits proliferation and the cell cycle in prostate cancer.. PubMed. 11(8). 3969–3976. 23 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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