Céline Vanderplanck

555 total citations
7 papers, 439 citations indexed

About

Céline Vanderplanck is a scholar working on Molecular Biology, Genetics and Surgery. According to data from OpenAlex, Céline Vanderplanck has authored 7 papers receiving a total of 439 indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Molecular Biology, 5 papers in Genetics and 1 paper in Surgery. Recurrent topics in Céline Vanderplanck's work include Muscle Physiology and Disorders (7 papers), Neurogenetic and Muscular Disorders Research (5 papers) and RNA Research and Splicing (4 papers). Céline Vanderplanck is often cited by papers focused on Muscle Physiology and Disorders (7 papers), Neurogenetic and Muscular Disorders Research (5 papers) and RNA Research and Splicing (4 papers). Céline Vanderplanck collaborates with scholars based in Belgium, France and United States. Céline Vanderplanck's co-authors include Alexandra Belayew, Eugénie Ansseau, Alexandra Tassin, Dalila Laoudj‐Chenivesse, Frédérique Coppée, Steve D. Wilton, Marietta Barro, Jacques Mercier, Yiwen Chen and Scott Q. Harper and has published in prestigious journals such as PLoS ONE, Journal of Cellular and Molecular Medicine and Stem Cells and Development.

In The Last Decade

Céline Vanderplanck

7 papers receiving 433 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Céline Vanderplanck Belgium	7	416	139	112	47	39	7	439
Richard J F L Lemmers Netherlands	7	539 1.3×	120 0.9×	140 1.3×	81 1.7×	89 2.3×	7	577
Ngoc Lu‐Nguyen United Kingdom	11	347 0.8×	86 0.6×	64 0.6×	43 0.9×	59 1.5×	28	392
Rita Perlingeiro United States	6	373 0.9×	66 0.5×	54 0.5×	38 0.8×	51 1.3×	8	383
Christophe Pichavant United States	9	319 0.8×	62 0.4×	65 0.6×	39 0.8×	76 1.9×	13	353
Maryna Panamarova United Kingdom	6	337 0.8×	50 0.4×	54 0.5×	33 0.7×	28 0.7×	10	355
Milena Cau Italy	10	313 0.8×	122 0.9×	256 2.3×	45 1.0×	52 1.3×	19	533
Ludovic Arandel France	9	351 0.8×	53 0.4×	69 0.6×	70 1.5×	96 2.5×	11	393
Reinaldo Issao Takata Brazil	11	239 0.6×	44 0.3×	62 0.6×	63 1.3×	62 1.6×	26	300
Ana Buj‐Bello France	9	325 0.8×	84 0.6×	68 0.6×	86 1.8×	56 1.4×	19	369
D. Powell United Kingdom	3	293 0.7×	43 0.3×	48 0.4×	36 0.8×	86 2.2×	3	305

Countries citing papers authored by Céline Vanderplanck

Since Specialization

Citations

This map shows the geographic impact of Céline Vanderplanck's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Céline Vanderplanck with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Céline Vanderplanck more than expected).

Fields of papers citing papers by Céline Vanderplanck

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Céline Vanderplanck. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Céline Vanderplanck. The network helps show where Céline Vanderplanck may publish in the future.

Co-authorship network of co-authors of Céline Vanderplanck

This figure shows the co-authorship network connecting the top 25 collaborators of Céline Vanderplanck. A scholar is included among the top collaborators of Céline Vanderplanck based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Céline Vanderplanck. Céline Vanderplanck is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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7 of 7 papers shown

1.

Vanderplanck, Céline, Alexandra Tassin, Eugénie Ansseau, et al.. (2018). Overexpression of the double homeodomain protein DUX4c interferes with myofibrillogenesis and induces clustering of myonuclei. Skeletal Muscle. 8(1). 2–2. 17 indexed citations

2.

Ansseau, Eugénie, et al.. (2017). Antisense Oligonucleotides Used to Target the DUX4 mRNA as Therapeutic Approaches in FaciosScapuloHumeral Muscular Dystrophy (FSHD). Genes. 8(3). 93–93. 52 indexed citations

3.

Ansseau, Eugénie, Karlien Pieters, Céline Vanderplanck, et al.. (2015). The Role of D4Z4-Encoded Proteins in the Osteogenic Differentiation of Mesenchymal Stromal Cells Isolated from Bone Marrow. Stem Cells and Development. 24(22). 2674–2686. 12 indexed citations

4.

Tassin, Alexandra, Baptiste Leroy, Dalila Laoudj‐Chenivesse, et al.. (2012). FSHD Myotubes with Different Phenotypes Exhibit Distinct Proteomes. PLoS ONE. 7(12). e51865–e51865. 28 indexed citations

5.

Tassin, Alexandra, Dalila Laoudj‐Chenivesse, Céline Vanderplanck, et al.. (2012). DUX4 expression in FSHD muscle cells: how could such a rare protein cause a myopathy?. Journal of Cellular and Molecular Medicine. 17(1). 76–89. 117 indexed citations

6.

Vanderplanck, Céline, Eugénie Ansseau, Alexandra Tassin, et al.. (2011). The FSHD Atrophic Myotube Phenotype Is Caused by DUX4 Expression. PLoS ONE. 6(10). e26820–e26820. 159 indexed citations

7.

Ansseau, Eugénie, Dalila Laoudj‐Chenivesse, Alexandra Tassin, et al.. (2009). DUX4c Is Up-Regulated in FSHD. It Induces the MYF5 Protein and Human Myoblast Proliferation. PLoS ONE. 4(10). e7482–e7482. 54 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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