Celien Lismont

1.4k total citations
27 papers, 995 citations indexed

About

Celien Lismont is a scholar working on Molecular Biology, Physiology and Cancer Research. According to data from OpenAlex, Celien Lismont has authored 27 papers receiving a total of 995 indexed citations (citations by other indexed papers that have themselves been cited), including 25 papers in Molecular Biology, 8 papers in Physiology and 7 papers in Cancer Research. Recurrent topics in Celien Lismont's work include Peroxisome Proliferator-Activated Receptors (25 papers), Adenosine and Purinergic Signaling (7 papers) and Cancer, Hypoxia, and Metabolism (7 papers). Celien Lismont is often cited by papers focused on Peroxisome Proliferator-Activated Receptors (25 papers), Adenosine and Purinergic Signaling (7 papers) and Cancer, Hypoxia, and Metabolism (7 papers). Celien Lismont collaborates with scholars based in Belgium, Egypt and Netherlands. Celien Lismont's co-authors include Marc Fransen, Paul A. Walton, Paul P. Van Veldhoven, Iulia Revenco, Marcus Nordgren, Chantal Brees, Oksana Apanasets, Jorge E. Azevedo, Tânia Francisco and Hongli Li and has published in prestigious journals such as The Journal of Cell Biology, PLoS ONE and Free Radical Biology and Medicine.

In The Last Decade

Celien Lismont

26 papers receiving 993 citations

Peers

Celien Lismont
Celien Lismont
Citations per year, relative to Celien Lismont Celien Lismont (= 1×) peers Oksana Apanasets

Countries citing papers authored by Celien Lismont

Since Specialization
Citations

This map shows the geographic impact of Celien Lismont's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Celien Lismont with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Celien Lismont more than expected).

Fields of papers citing papers by Celien Lismont

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Celien Lismont. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Celien Lismont. The network helps show where Celien Lismont may publish in the future.

Co-authorship network of co-authors of Celien Lismont

This figure shows the co-authorship network connecting the top 25 collaborators of Celien Lismont. A scholar is included among the top collaborators of Celien Lismont based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Celien Lismont. Celien Lismont is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Lismont, Celien, Jorge E. Azevedo, Wim Vandenberghe, et al.. (2025). PEX14 acts as a molecular link between optineurin and the autophagic machinery to induce pexophagy. The Journal of Cell Biology. 224(11).
2.
Siegerist, Florian, Celien Lismont, Sara Cairoli, et al.. (2025). Targeting oxidative stress-induced lipid peroxidation enhances podocyte function in cystinosis. Journal of Translational Medicine. 23(1). 206–206. 2 indexed citations
3.
Lismont, Celien, et al.. (2024). Characterization of the Peroxisomal Proteome and Redox Balance in Human Prostate Cancer Cell Lines. Antioxidants. 13(11). 1340–1340. 4 indexed citations
4.
Reglinski, Katharina, Celien Lismont, Joseph L. Costello, et al.. (2023). Peroxisomes : novel findings and future directions. Histochemistry and Cell Biology. 159(5). 379–387. 1 indexed citations
5.
Lismont, Celien, et al.. (2023). Functional Analysis of GSTK1 in Peroxisomal Redox Homeostasis in HEK-293 Cells. Antioxidants. 12(6). 1236–1236. 5 indexed citations
6.
Li, Hongli, et al.. (2023). Enhanced Levels of Peroxisome-Derived H2O2 Do Not Induce Pexophagy but Impair Autophagic Flux in HEK-293 and HeLa Cells. Antioxidants. 12(3). 613–613. 6 indexed citations
7.
Lismont, Celien, Janet Koster, Hongli Li, et al.. (2023). The solute carrier SLC25A17 sustains peroxisomal redox homeostasis in diverse mammalian cell lines. Free Radical Biology and Medicine. 212. 241–254. 2 indexed citations
8.
Li, Hongli, et al.. (2023). Assessment of the Peroxisomal Redox State in Living Cells Using NADPH- and NAD+/NADH-Specific Fluorescent Protein Sensors. Methods in molecular biology. 2643. 183–197. 3 indexed citations
9.
Lismont, Celien, Iulia Revenco, Hongli Li, et al.. (2022). Peroxisome-Derived Hydrogen Peroxide Modulates the Sulfenylation Profiles of Key Redox Signaling Proteins in Flp-In T-REx 293 Cells. Frontiers in Cell and Developmental Biology. 10. 888873–888873. 11 indexed citations
10.
Ramazani, Yasaman, Noël Knops, Celien Lismont, et al.. (2021). Therapeutic concentrations of calcineurin inhibitors do not deregulate glutathione redox balance in human renal proximal tubule cells. PLoS ONE. 16(4). e0250996–e0250996. 8 indexed citations
11.
Li, Hongli, et al.. (2021). The Peroxisome-Autophagy Redox Connection: A Double-Edged Sword?. Frontiers in Cell and Developmental Biology. 9. 814047–814047. 19 indexed citations
12.
Passmore, Josiah B., Ruth E. Carmichael, Tina A. Schrader, et al.. (2020). Mitochondrial fission factor (MFF) is a critical regulator of peroxisome maturation. Biochimica et Biophysica Acta (BBA) - Molecular Cell Research. 1867(7). 118709–118709. 30 indexed citations
13.
Fransen, Marc, et al.. (2020). Peroxisomal Dysfunction and Oxidative Stress in Neurodegenerative Disease: A Bidirectional Crosstalk. Advances in experimental medicine and biology. 1299. 19–30. 13 indexed citations
14.
Lismont, Celien, Janet Koster, Myriam Baes, et al.. (2019). Deciphering the potential involvement of PXMP2 and PEX11B in hydrogen peroxide permeation across the peroxisomal membrane reveals a role for PEX11B in protein sorting. Biochimica et Biophysica Acta (BBA) - Biomembranes. 1861(10). 182991–182991. 28 indexed citations
15.
Fransen, Marc & Celien Lismont. (2018). Redox Signaling from and to Peroxisomes: Progress, Challenges, and Prospects. Antioxidants and Redox Signaling. 30(1). 95–112. 60 indexed citations
16.
Lismont, Celien, Marcus Nordgren, Chantal Brees, et al.. (2017). Peroxisomes as Modulators of Cellular Protein Thiol Oxidation: A New Model System. Antioxidants and Redox Signaling. 30(1). 22–39. 30 indexed citations
17.
Walton, Paul A., Chantal Brees, Celien Lismont, Oksana Apanasets, & Marc Fransen. (2017). The peroxisomal import receptor PEX5 functions as a stress sensor, retaining catalase in the cytosol in times of oxidative stress. Biochimica et Biophysica Acta (BBA) - Molecular Cell Research. 1864(10). 1833–1843. 63 indexed citations
18.
Lismont, Celien, Paul A. Walton, & Marc Fransen. (2017). Quantitative Monitoring of Subcellular Redox Dynamics in Living Mammalian Cells Using RoGFP2-Based Probes. Methods in molecular biology. 1595. 151–164. 15 indexed citations
19.
Lismont, Celien, et al.. (2015). Peroxisome biogenesis in mammalian cells: The impact of genes and environment. Biochimica et Biophysica Acta (BBA) - Molecular Cell Research. 1863(5). 1049–1060. 30 indexed citations
20.
Lismont, Celien, Marcus Nordgren, Paul P. Van Veldhoven, & Marc Fransen. (2015). Redox interplay between mitochondria and peroxisomes. Frontiers in Cell and Developmental Biology. 3. 35–35. 151 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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