Celeste Owen

761 total citations
8 papers, 180 citations indexed

About

Celeste Owen is a scholar working on Molecular Biology, Epidemiology and Rheumatology. According to data from OpenAlex, Celeste Owen has authored 8 papers receiving a total of 180 indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Molecular Biology, 2 papers in Epidemiology and 2 papers in Rheumatology. Recurrent topics in Celeste Owen's work include ATP Synthase and ATPases Research (5 papers), Autophagy in Disease and Therapy (2 papers) and RNA modifications and cancer (2 papers). Celeste Owen is often cited by papers focused on ATP Synthase and ATPases Research (5 papers), Autophagy in Disease and Therapy (2 papers) and RNA modifications and cancer (2 papers). Celeste Owen collaborates with scholars based in Canada, China and United States. Celeste Owen's co-authors include Irina Voronov, Jane E. Aubin, Morris F. Manolson, Luciene R. Carraro‐Lacroix, John H. Brumell, Paul Corey, Andrew Wang, Yingwei Hu, Tayyab Khan and Ann M. Flenniken and has published in prestigious journals such as Scientific Reports, Journal of Bone and Mineral Research and Journal of Cellular Biochemistry.

In The Last Decade

Celeste Owen

8 papers receiving 178 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Celeste Owen Canada 7 118 44 31 28 23 8 180
William H. Bradford United States 9 142 1.2× 31 0.7× 7 0.2× 16 0.6× 34 1.5× 11 282
Sebastiaan Vanuytven Belgium 2 89 0.8× 24 0.5× 24 0.8× 7 0.3× 5 0.2× 3 141
Dacheng Zhao China 10 167 1.4× 18 0.4× 27 0.9× 7 0.3× 11 0.5× 17 289
David R. Amici United States 9 137 1.2× 83 1.9× 16 0.5× 18 0.6× 31 1.3× 13 214
Lutz Fleischhauer Germany 8 59 0.5× 7 0.2× 22 0.7× 11 0.4× 35 1.5× 11 155
Monica Schwartzman United States 7 58 0.5× 22 0.5× 5 0.2× 34 1.2× 54 2.3× 12 175
Alexander Bernt Germany 9 152 1.3× 21 0.5× 26 0.8× 13 0.5× 31 1.3× 10 199
Laura M Yerges United States 6 135 1.1× 8 0.2× 45 1.5× 49 1.8× 6 0.3× 7 178
F. Dicső Hungary 5 44 0.4× 39 0.9× 59 1.9× 19 0.7× 18 0.8× 9 178
William Sullivan United States 5 122 1.0× 6 0.1× 38 1.2× 38 1.4× 71 3.1× 9 253

Countries citing papers authored by Celeste Owen

Since Specialization
Citations

This map shows the geographic impact of Celeste Owen's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Celeste Owen with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Celeste Owen more than expected).

Fields of papers citing papers by Celeste Owen

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Celeste Owen. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Celeste Owen. The network helps show where Celeste Owen may publish in the future.

Co-authorship network of co-authors of Celeste Owen

This figure shows the co-authorship network connecting the top 25 collaborators of Celeste Owen. A scholar is included among the top collaborators of Celeste Owen based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Celeste Owen. Celeste Owen is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

8 of 8 papers shown
1.
Wang, Andrew, Luciene R. Carraro‐Lacroix, Celeste Owen, et al.. (2017). Activity-independent targeting of mTOR to lysosomes in primary osteoclasts. Scientific Reports. 7(1). 3005–3005. 10 indexed citations
2.
Ganss, Bernhard, Andrew Wang, Ralph Zirngibl, et al.. (2017). V‐ATPases Containing a3 Subunit Play a Direct Role in Enamel Development in Mice. Journal of Cellular Biochemistry. 118(10). 3328–3340. 5 indexed citations
3.
Hu, Yingwei, Luciene R. Carraro‐Lacroix, Andrew Wang, et al.. (2015). Lysosomal pH Plays a Key Role in Regulation of mTOR Activity in Osteoclasts. Journal of Cellular Biochemistry. 117(2). 413–425. 46 indexed citations
4.
Voronov, Irina, et al.. (2013). The R740S mutation in the V‐ATPase a3 subunit results in osteoclast apoptosis and defective early‐stage autophagy. Journal of Cellular Biochemistry. 114(12). 2823–2833. 23 indexed citations
5.
Owen, Celeste, Frieda Chen, Ann M. Flenniken, et al.. (2012). A novel Phex mutation in a new mouse model of hypophosphatemic rickets. Journal of Cellular Biochemistry. 113(7). 2432–2441. 15 indexed citations
6.
Voronov, Irina, et al.. (2012). The R740S mutation in the V-ATPase a3 subunit increases lysosomal pH, impairs NFATc1 translocation, and decreases in vitro osteoclastogenesis. Journal of Bone and Mineral Research. 28(1). 108–118. 22 indexed citations
7.
Flenniken, Ann M., Celeste Owen, Irina Voronov, et al.. (2011). The V–ATPase a3 subunit mutation R740S is dominant negative and results in osteopetrosis in mice. Journal of Bone and Mineral Research. 26(7). 1484–1493. 30 indexed citations
8.
Holmes, Christina, et al.. (2007). Longitudinal Analysis of Mesenchymal Progenitors and Bone Quality in the Stem Cell Antigen-1–Null Osteoporotic Mouse. Journal of Bone and Mineral Research. 22(9). 1373–1386. 29 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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