Cecilia Bivik

889 total citations
18 papers, 716 citations indexed

About

Cecilia Bivik is a scholar working on Immunology, Molecular Biology and Dermatology. According to data from OpenAlex, Cecilia Bivik has authored 18 papers receiving a total of 716 indexed citations (citations by other indexed papers that have themselves been cited), including 12 papers in Immunology, 10 papers in Molecular Biology and 4 papers in Dermatology. Recurrent topics in Cecilia Bivik's work include Psoriasis: Treatment and Pathogenesis (10 papers), Dermatology and Skin Diseases (3 papers) and melanin and skin pigmentation (3 papers). Cecilia Bivik is often cited by papers focused on Psoriasis: Treatment and Pathogenesis (10 papers), Dermatology and Skin Diseases (3 papers) and melanin and skin pigmentation (3 papers). Cecilia Bivik collaborates with scholars based in Sweden, United States and United Kingdom. Cecilia Bivik's co-authors include Inger Rosdahl, Karin Öllinger, Charlotta Enerbäck, Anna‐Karin Ekman, Deepti Verma, Mats Fredrikson, Ingemar Rundquist, Ingrid Synnerstad, Peter Söderkvist and Ensieh Farahani and has published in prestigious journals such as Journal of Investigative Dermatology, Journal of the American Academy of Dermatology and Carcinogenesis.

In The Last Decade

Cecilia Bivik

18 papers receiving 707 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Cecilia Bivik Sweden 13 449 334 153 94 92 18 716
Elmar vom Baur France 9 598 1.3× 358 1.1× 143 0.9× 31 0.3× 159 1.7× 14 1.0k
Isabel Ruppen Spain 8 436 1.0× 167 0.5× 43 0.3× 69 0.7× 140 1.5× 14 688
Daniel DiSepio United States 11 431 1.0× 140 0.4× 65 0.4× 87 0.9× 75 0.8× 14 744
Yuki Sato Japan 7 144 0.3× 383 1.1× 198 1.3× 20 0.2× 94 1.0× 15 561
Motoshi Kinouchi Japan 13 210 0.5× 99 0.3× 95 0.6× 110 1.2× 55 0.6× 21 426
Yaxiong Deng China 7 160 0.4× 469 1.4× 143 0.9× 12 0.1× 100 1.1× 12 649
Walid Mahfouf France 14 452 1.0× 76 0.2× 74 0.5× 60 0.6× 86 0.9× 20 675
Jeffrey Hildesheim United States 12 362 0.8× 70 0.2× 75 0.5× 68 0.7× 169 1.8× 13 567
Jamal Dakour Canada 15 341 0.8× 209 0.6× 25 0.2× 89 0.9× 62 0.7× 27 750
Zhongjia Tan United States 8 354 0.8× 288 0.9× 35 0.2× 23 0.2× 205 2.2× 9 672

Countries citing papers authored by Cecilia Bivik

Since Specialization
Citations

This map shows the geographic impact of Cecilia Bivik's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Cecilia Bivik with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Cecilia Bivik more than expected).

Fields of papers citing papers by Cecilia Bivik

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Cecilia Bivik. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Cecilia Bivik. The network helps show where Cecilia Bivik may publish in the future.

Co-authorship network of co-authors of Cecilia Bivik

This figure shows the co-authorship network connecting the top 25 collaborators of Cecilia Bivik. A scholar is included among the top collaborators of Cecilia Bivik based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Cecilia Bivik. Cecilia Bivik is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

18 of 18 papers shown
1.
Köhler, Ines, et al.. (2024). NOS2-derived low levels of NO drive psoriasis pathogenesis. Cell Death and Disease. 15(6). 449–449. 5 indexed citations
2.
Verma, Deepti, et al.. (2023). Differential DNA Methylation of MicroRNA-Encoding Genes in Psoriatic Epidermis Highlights the Wnt Pathway. Journal of Investigative Dermatology. 143(8). 1594–1597.e14. 2 indexed citations
3.
Bivik, Cecilia, Ines Köhler, Deepti Verma, et al.. (2021). MTH1 Inhibitors for the Treatment of Psoriasis. Journal of Investigative Dermatology. 141(8). 2037–2048.e4. 15 indexed citations
4.
Verma, Deepti, et al.. (2020). Enhanced Inflammasome Activity in Patients with Psoriasis Promotes Systemic Inflammation. Journal of Investigative Dermatology. 141(3). 586–595.e5. 74 indexed citations
5.
Ekman, Anna‐Karin, Cecilia Bivik, Ingemar Rundquist, & Charlotta Enerbäck. (2019). IL-17 and IL-22 Promote Keratinocyte Stemness in the Germinative Compartment in Psoriasis. Journal of Investigative Dermatology. 139(7). 1564–1573.e8. 54 indexed citations
6.
Bivik, Cecilia, et al.. (2019). 222 IL-17 and IL-22 promote keratinocyte stemness in the germinative compartment in psoriasis. Journal of Investigative Dermatology. 139(9). S252–S252. 3 indexed citations
7.
Ekman, Anna‐Karin, Cecilia Bivik, Ingemar Rundquist, & Charlotta Enerbäck. (2019). 292 IL-17 and IL-22 promote keratinocyte stemness in the germinative compartment in psoriasis. Journal of Investigative Dermatology. 139(5). S51–S51. 4 indexed citations
8.
Verma, Deepti, Anna‐Karin Ekman, Cecilia Bivik, & Charlotta Enerbäck. (2017). Genome-Wide DNA Methylation Profiling Identifies Differential Methylation in Uninvolved Psoriatic Epidermis. Journal of Investigative Dermatology. 138(5). 1088–1093. 47 indexed citations
9.
Ekman, Anna‐Karin, et al.. (2016). Psoriasin (S100A7) promotes stress-induced angiogenesis. British Journal of Dermatology. 175(6). 1263–1273. 32 indexed citations
10.
Bostner, Josefine, Elin Karlsson, Cecilia Bivik, et al.. (2015). S6 kinase signaling: tamoxifen response and prognostic indication in two breast cancer cohorts. Endocrine Related Cancer. 22(3). 331–343. 24 indexed citations
11.
Ekman, Anna‐Karin, et al.. (2014). Systemic treatment and narrowband ultraviolet B differentially affect cardiovascular risk markers in psoriasis. Journal of the American Academy of Dermatology. 70(6). 1067–1075. 32 indexed citations
12.
Bivik, Cecilia, Petra Wäster, Mats Fredrikson, et al.. (2014). MDM2 SNP309 promoter polymorphism confers risk for hereditary melanoma. Melanoma Research. 24(3). 190–197. 9 indexed citations
13.
Ekman, Anna‐Karin, et al.. (2014). Genetic variations ofNLRP1: susceptibility in psoriasis. British Journal of Dermatology. 171(6). 1517–1520. 69 indexed citations
14.
Verma, Deepti, Cecilia Bivik, Ensieh Farahani, et al.. (2012). Inflammasome polymorphisms confer susceptibility to sporadic malignant melanoma. Pigment Cell & Melanoma Research. 25(4). 506–513. 104 indexed citations
15.
Bivik, Cecilia & Karin Öllinger. (2008). JNK mediates UVB-induced apoptosis upstream lysosomal membrane permeabilization and Bcl-2 family proteins. APOPTOSIS. 13(9). 1111–1120. 34 indexed citations
16.
Bivik, Cecilia, et al.. (2006). UVA/B-Induced Apoptosis in Human Melanocytes Involves Translocation of Cathepsins and Bcl-2 Family Members. Journal of Investigative Dermatology. 126(5). 1119–1127. 89 indexed citations
17.
Bivik, Cecilia, Inger Rosdahl, & Karin Öllinger. (2006). Hsp70 protects against UVB induced apoptosis by preventing release of cathepsins and cytochrome c in human melanocytes. Carcinogenesis. 28(3). 537–544. 101 indexed citations
18.
Bivik, Cecilia, et al.. (2005). Wavelength-specific effects on UVB-induced apoptosis in melanocytes. A study of Bcl-2/Bax expression and keratinocyte rescue effects. Melanoma Research. 15(1). 7–13. 18 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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