Cécile Boisson

1.3k total citations
7 papers, 599 citations indexed

About

Cécile Boisson is a scholar working on Cancer Research, Molecular Biology and Pathology and Forensic Medicine. According to data from OpenAlex, Cécile Boisson has authored 7 papers receiving a total of 599 indexed citations (citations by other indexed papers that have themselves been cited), including 5 papers in Cancer Research, 2 papers in Molecular Biology and 2 papers in Pathology and Forensic Medicine. Recurrent topics in Cécile Boisson's work include Cancer, Hypoxia, and Metabolism (3 papers), Epigenetics and DNA Methylation (2 papers) and Soft tissue tumor case studies (2 papers). Cécile Boisson is often cited by papers focused on Cancer, Hypoxia, and Metabolism (3 papers), Epigenetics and DNA Methylation (2 papers) and Soft tissue tumor case studies (2 papers). Cécile Boisson collaborates with scholars based in France and Germany. Cécile Boisson's co-authors include Sylviane Olschwang, Gilles Thomas, Albert J. Augustin, Marion Mandl, Gilles Thomas, Gabriela Möslein, T. Böker, Waltraut Friedl, Reiner Caspari and Martina Kadmon and has published in prestigious journals such as Human Molecular Genetics, International Journal of Cancer and Journal of Medical Genetics.

In The Last Decade

Cécile Boisson

7 papers receiving 575 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Cécile Boisson France 6 389 230 190 165 117 7 599
Shantie Jagmohan–Changur Netherlands 12 459 1.2× 249 1.1× 166 0.9× 287 1.7× 123 1.1× 17 681
Nel J. Kuipers‐Dijkshoorn Netherlands 15 196 0.5× 217 0.9× 343 1.8× 275 1.7× 190 1.6× 23 731
Viviana Gismondi Italy 17 692 1.8× 472 2.1× 253 1.3× 297 1.8× 168 1.4× 45 1.0k
Robyn Lukeis Australia 13 155 0.4× 202 0.9× 149 0.8× 273 1.7× 122 1.0× 25 580
John F. Herath United States 13 163 0.4× 136 0.6× 236 1.2× 280 1.7× 158 1.4× 14 765
Ulla‐Maria Flodérus Sweden 13 165 0.4× 102 0.4× 166 0.9× 185 1.1× 151 1.3× 14 746
Caroline Brennetot France 8 206 0.5× 215 0.9× 183 1.0× 295 1.8× 69 0.6× 8 620
Annette Gylling Finland 11 373 1.0× 245 1.1× 226 1.2× 188 1.1× 63 0.5× 15 599
Bénédicte Thuille France 5 345 0.9× 272 1.2× 159 0.8× 271 1.6× 33 0.3× 8 668
Denisa Ilenčíková Slovakia 15 438 1.1× 227 1.0× 257 1.4× 301 1.8× 190 1.6× 40 851

Countries citing papers authored by Cécile Boisson

Since Specialization
Citations

This map shows the geographic impact of Cécile Boisson's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Cécile Boisson with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Cécile Boisson more than expected).

Fields of papers citing papers by Cécile Boisson

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Cécile Boisson. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Cécile Boisson. The network helps show where Cécile Boisson may publish in the future.

Co-authorship network of co-authors of Cécile Boisson

This figure shows the co-authorship network connecting the top 25 collaborators of Cécile Boisson. A scholar is included among the top collaborators of Cécile Boisson based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Cécile Boisson. Cécile Boisson is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

7 of 7 papers shown
1.
Charbonnier, Françoise, Sylviane Olschwang, Qing Wang, et al.. (2002). MSH2 in contrast to MLH1 and MSH6 is frequently inactivated by exonic and promoter rearrangements in hereditary nonpolyposis colorectal cancer.. PubMed. 62(3). 848–53. 106 indexed citations
2.
Olschwang, Sylviane, Cécile Boisson, & Gilles Thomas. (2001). Peutz-Jeghers families unlinked toSTK11/LKB1 gene mutations are highly predisposed to primitive biliary adenocarcinoma. Journal of Medical Genetics. 38(6). 356–360. 65 indexed citations
3.
Olschwang, Sylviane, Richard J. Kahnoski, Cécile Boisson, et al.. (1998). Germline mutation profile of theVHL gene in von Hippel-Lindau disease and in sporadic hemangioblastoma. Human Mutation. 12(6). 424–430. 83 indexed citations
4.
Goutebroze, Laurence, Marco Giovannini, Cécile Boisson, et al.. (1998). Heterogeneity of mesothelioma cell lines as defined by altered genomic structure and expression of theNF2 gene. International Journal of Cancer. 77(4). 554–560. 50 indexed citations
5.
Olschwang, Sylviane, Richard J. Kahnoski, Cécile Boisson, et al.. (1998). Germline mutation profile of the VHL gene in von Hippel‐Lindau disease and in sporadic hemangioblastoma. Human Mutation. 12(6). 424–430. 7 indexed citations
6.
Caspari, Reiner, Waltraut Friedl, Marion Mandl, et al.. (1995). Familial adenomatous polyposis: desmoid tumours and lack of ophthalmic lesions (CHRPE) associated with APC mutations beyond codon 1444. Human Molecular Genetics. 4(3). 337–340. 283 indexed citations
7.
Olschwang, Sylviane, Cécile Boisson, Richard J. Kahnoski, F Resche, & Gilles Thomas. (1995). DNA-Based PresymptomaticDiagnosis for thevon Hippel-Lindau Disease byLinkage Analysis. European Journal of Human Genetics. 3(2). 108–115. 5 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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