Carlos Gonzalez‐Quesada

2.2k total citations
17 papers, 1.8k citations indexed

About

Carlos Gonzalez‐Quesada is a scholar working on Cardiology and Cardiovascular Medicine, Molecular Biology and Oncology. According to data from OpenAlex, Carlos Gonzalez‐Quesada has authored 17 papers receiving a total of 1.8k indexed citations (citations by other indexed papers that have themselves been cited), including 14 papers in Cardiology and Cardiovascular Medicine, 9 papers in Molecular Biology and 4 papers in Oncology. Recurrent topics in Carlos Gonzalez‐Quesada's work include Cardiac Fibrosis and Remodeling (8 papers), Signaling Pathways in Disease (4 papers) and Coronary Interventions and Diagnostics (3 papers). Carlos Gonzalez‐Quesada is often cited by papers focused on Cardiac Fibrosis and Remodeling (8 papers), Signaling Pathways in Disease (4 papers) and Coronary Interventions and Diagnostics (3 papers). Carlos Gonzalez‐Quesada collaborates with scholars based in United States, Mexico and Vietnam. Carlos Gonzalez‐Quesada's co-authors include Nikolaos G. Frangogiannis, Marcin Dobaczewski, Marcin Bujak, Ying Xia, Leonardo H. Mendoza, Na Li, Xiao‐Fan Wang, Dong-Wook Lee, Anna Biernacka and Ping Kong and has published in prestigious journals such as Circulation Research, Hypertension and American Journal Of Pathology.

In The Last Decade

Carlos Gonzalez‐Quesada

17 papers receiving 1.7k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Carlos Gonzalez‐Quesada United States 13 1.1k 791 435 278 265 17 1.8k
Paweł Zymek United States 7 912 0.8× 730 0.9× 382 0.9× 225 0.8× 212 0.8× 12 1.5k
Davy Vanhoutte United States 23 935 0.9× 946 1.2× 375 0.9× 286 1.0× 297 1.1× 33 2.1k
Anja M. van der Laan Netherlands 19 997 0.9× 694 0.9× 456 1.0× 157 0.6× 481 1.8× 39 1.9k
Marcin Bujak United States 15 1.6k 1.5× 1.2k 1.5× 568 1.3× 359 1.3× 387 1.5× 19 2.5k
Farhad Forudi United States 17 544 0.5× 800 1.0× 724 1.7× 241 0.9× 197 0.7× 25 2.0k
Mortimer Korf‐Klingebiel Germany 18 690 0.6× 760 1.0× 383 0.9× 142 0.5× 385 1.5× 25 1.8k
Bryan D. Maliken United States 14 850 0.8× 922 1.2× 447 1.0× 260 0.9× 124 0.5× 20 1.9k
Leonardo H. Mendoza United States 16 1.4k 1.3× 1.1k 1.3× 524 1.2× 335 1.2× 607 2.3× 19 2.5k
Anis Hanna United States 14 704 0.6× 569 0.7× 263 0.6× 156 0.6× 192 0.7× 23 1.3k
Emmanuel Chorianopoulos Germany 20 747 0.7× 749 0.9× 318 0.7× 275 1.0× 191 0.7× 47 1.8k

Countries citing papers authored by Carlos Gonzalez‐Quesada

Since Specialization
Citations

This map shows the geographic impact of Carlos Gonzalez‐Quesada's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Carlos Gonzalez‐Quesada with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Carlos Gonzalez‐Quesada more than expected).

Fields of papers citing papers by Carlos Gonzalez‐Quesada

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Carlos Gonzalez‐Quesada. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Carlos Gonzalez‐Quesada. The network helps show where Carlos Gonzalez‐Quesada may publish in the future.

Co-authorship network of co-authors of Carlos Gonzalez‐Quesada

This figure shows the co-authorship network connecting the top 25 collaborators of Carlos Gonzalez‐Quesada. A scholar is included among the top collaborators of Carlos Gonzalez‐Quesada based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Carlos Gonzalez‐Quesada. Carlos Gonzalez‐Quesada is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

17 of 17 papers shown
1.
Guerrero, Mayra, Ammar M. Killu, Carlos Gonzalez‐Quesada, et al.. (2020). Pre-Emptive Radiofrequency Septal Ablation to Decrease the Risk of Left Ventricular Outflow Tract Obstruction After TMVR. JACC: Cardiovascular Interventions. 13(9). 1129–1132. 7 indexed citations
2.
Biernacka, Anna, Michele Cavalera, Junhong Wang, et al.. (2015). Smad3 Signaling Promotes Fibrosis While Preserving Cardiac and Aortic Geometry in Obese Diabetic Mice. Circulation Heart Failure. 8(4). 788–798. 109 indexed citations
3.
Gonzalez‐Quesada, Carlos & Robert P. Giugliano. (2014). Comparison of the Phase III Clinical Trial Designs of Novel Oral Anticoagulants Versus Warfarin for the Treatment of Nonvalvular Atrial Fibrillation: Implications for Clinical Practice. American Journal of Cardiovascular Drugs. 14(2). 111–127. 17 indexed citations
4.
Saxena, Amit, Marcin Bujak, Marcin Dobaczewski, et al.. (2014). CXCR3-independent actions of the CXC chemokine CXCL10 in the infarcted myocardium and in isolated cardiac fibroblasts are mediated through proteoglycans. Cardiovascular Research. 103(2). 217–227. 59 indexed citations
5.
Kong, Ping, et al.. (2013). Thrombospondin-1 regulates adiposity and metabolic dysfunction in diet-induced obesity enhancing adipose inflammation and stimulating adipocyte proliferation. American Journal of Physiology-Endocrinology and Metabolism. 305(3). E439–E450. 78 indexed citations
6.
Christia, Panagiota, Marcin Bujak, Carlos Gonzalez‐Quesada, et al.. (2013). Systematic Characterization of Myocardial Inflammation, Repair, and Remodeling in a Mouse Model of Reperfused Myocardial Infarction. Journal of Histochemistry & Cytochemistry. 61(8). 555–570. 99 indexed citations
7.
Gonzalez‐Quesada, Carlos, Michele Cavalera, Anna Biernacka, et al.. (2013). Thrombospondin-1 Induction in the Diabetic Myocardium Stabilizes the Cardiac Matrix in Addition to Promoting Vascular Rarefaction Through Angiopoietin-2 Upregulation. Circulation Research. 113(12). 1331–1344. 111 indexed citations
8.
Xia, Ying, Marcin Dobaczewski, Carlos Gonzalez‐Quesada, et al.. (2011). Endogenous Thrombospondin 1 Protects the Pressure-Overloaded Myocardium by Modulating Fibroblast Phenotype and Matrix Metabolism. Hypertension. 58(5). 902–911. 114 indexed citations
9.
Dobaczewski, Marcin, Ying Xia, Marcin Bujak, Carlos Gonzalez‐Quesada, & Nikolaos G. Frangogiannis. (2010). CCR5 Signaling Suppresses Inflammation and Reduces Adverse Remodeling of the Infarcted Heart, Mediating Recruitment of Regulatory T Cells. American Journal Of Pathology. 176(5). 2177–2187. 239 indexed citations
10.
Dobaczewski, Marcin, Marcin Bujak, Na Li, et al.. (2010). Smad3 Signaling Critically Regulates Fibroblast Phenotype and Function in Healing Myocardial Infarction. Circulation Research. 107(3). 418–428. 318 indexed citations
11.
Bujak, Marcin, Marcin Dobaczewski, Carlos Gonzalez‐Quesada, et al.. (2009). Induction of the CXC Chemokine Interferon-γ-Inducible Protein 10 Regulates the Reparative Response Following Myocardial Infarction. Circulation Research. 105(10). 973–983. 103 indexed citations
12.
Martínez‐Ríos, Marco Antonio, et al.. (2009). Left main coronary artery stenosis treatment with two paclitaxel-eluting stents in a patient with cardiac allograft vasculopathy.. PubMed. 78(4). 407–12. 1 indexed citations
13.
Gonzalez‐Quesada, Carlos & Nikolaos G. Frangogiannis. (2009). Monocyte chemoattractant protein-1/CCL2 as a biomarker in acute coronary syndromes. Current Atherosclerosis Reports. 11(2). 131–138. 61 indexed citations
14.
Dobaczewski, Marcin, Carlos Gonzalez‐Quesada, & Nikolaos G. Frangogiannis. (2009). The extracellular matrix as a modulator of the inflammatory and reparative response following myocardial infarction. Journal of Molecular and Cellular Cardiology. 48(3). 504–511. 421 indexed citations
15.
Martínez‐Ríos, Marco Antonio, Marco Antonio Peña‐Duque, José Manuel Fragoso, et al.. (2008). No association found between the insertion/deletion of a 287-bp alu repeat sequence within intron 16 of the angiotensin-I-converting enzyme (ACE) gene in Mexican patients and binary restenosis after coronary stenting. Clinica Chimica Acta. 397(1-2). 65–67. 7 indexed citations
16.
Martínez‐Ríos, Marco Antonio, José Manuel Fragoso, José Manuel Rodrı́guez-Pérez, et al.. (2008). The interleukin 1B–511 polymorphism is associated with the risk of developing restenosis after coronary stenting in Mexican patients. Human Immunology. 69(2). 116–121. 17 indexed citations
17.
Castellanos, José A., et al.. (2008). Predictors of mortality and adverse outcome in elderly high-risk patients undergoing percutaneous coronary intervention.. PubMed. 77(3). 194–9. 7 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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