Carlos A.A. Penatti

847 total citations
26 papers, 651 citations indexed

About

Carlos A.A. Penatti is a scholar working on Molecular Biology, Endocrinology, Diabetes and Metabolism and Cellular and Molecular Neuroscience. According to data from OpenAlex, Carlos A.A. Penatti has authored 26 papers receiving a total of 651 indexed citations (citations by other indexed papers that have themselves been cited), including 15 papers in Molecular Biology, 11 papers in Endocrinology, Diabetes and Metabolism and 9 papers in Cellular and Molecular Neuroscience. Recurrent topics in Carlos A.A. Penatti's work include Hormonal and reproductive studies (10 papers), Neuroscience and Neuropharmacology Research (9 papers) and Receptor Mechanisms and Signaling (6 papers). Carlos A.A. Penatti is often cited by papers focused on Hormonal and reproductive studies (10 papers), Neuroscience and Neuropharmacology Research (9 papers) and Receptor Mechanisms and Signaling (6 papers). Carlos A.A. Penatti collaborates with scholars based in United States, Brazil and France. Carlos A.A. Penatti's co-authors include Leslie Henderson, Donna M. Porter, Etelvino José Henriques Bechara, Marilene Demasi, Roberto DeLucia, Ann S. Clark, B Jones, Joseph G. Oberlander, Matthew C. Davis and Paul Yang and has published in prestigious journals such as Journal of Neuroscience, Brain Research and Free Radical Biology and Medicine.

In The Last Decade

Carlos A.A. Penatti

25 papers receiving 643 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Carlos A.A. Penatti United States	15	239	212	128	126	93	26	651
Kyeong‐Hoon Jeong United States	17	259 1.1×	141 0.7×	86 0.7×	69 0.5×	116 1.2×	23	679
Cora Cymeryng Argentina	17	290 1.2×	121 0.6×	199 1.6×	66 0.5×	91 1.0×	45	726
Jennifer K. Stewart United States	17	212 0.9×	135 0.6×	102 0.8×	150 1.2×	93 1.0×	38	763
Elaine S. Coleman United States	15	274 1.1×	124 0.6×	143 1.1×	133 1.1×	38 0.4×	26	783
James J. Mrotek United States	13	170 0.7×	168 0.8×	71 0.6×	84 0.7×	72 0.8×	25	551
Hussien Al‐Shamma United States	15	210 0.9×	127 0.6×	102 0.8×	212 1.7×	97 1.0×	20	814
Mulan He China	21	258 1.1×	95 0.4×	78 0.6×	172 1.4×	27 0.3×	47	946
B. Manz Germany	18	205 0.9×	95 0.4×	122 1.0×	61 0.5×	61 0.7×	56	761
Christine Gervy Decoster Belgium	14	272 1.1×	357 1.7×	243 1.9×	108 0.9×	71 0.8×	26	936
G.D. Snyder United States	16	239 1.0×	353 1.7×	240 1.9×	251 2.0×	132 1.4×	21	1.1k

Countries citing papers authored by Carlos A.A. Penatti

Since Specialization

Citations

This map shows the geographic impact of Carlos A.A. Penatti's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Carlos A.A. Penatti with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Carlos A.A. Penatti more than expected).

Fields of papers citing papers by Carlos A.A. Penatti

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Carlos A.A. Penatti. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Carlos A.A. Penatti. The network helps show where Carlos A.A. Penatti may publish in the future.

Co-authorship network of co-authors of Carlos A.A. Penatti

This figure shows the co-authorship network connecting the top 25 collaborators of Carlos A.A. Penatti. A scholar is included among the top collaborators of Carlos A.A. Penatti based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Carlos A.A. Penatti. Carlos A.A. Penatti is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Ribeiro, Miriam O., Mônica Akemi Sato, Carlos A.A. Penatti, et al.. (2022). Triiodothyronine Treatment reverses Depression-Like Behavior in a triple-transgenic animal model of Alzheimer’s Disease. Metabolic Brain Disease. 37(8). 2735–2750. 7 indexed citations

Cardoso, Vanessa Eid da Silva, Fernando Dutra, Estela Bevilacqua, et al.. (2016). Liver Damage Induced by Succinylacetone: A Shared Redox Imbalance Mechanism between Tyrosinemia and Hepatic Porphyrias. Journal of the Brazilian Chemical Society. 9 indexed citations

Oberlander, Joseph G., Carlos A.A. Penatti, Donna M. Porter, & Leslie Henderson. (2012). The Buzz about Anabolic Androgenic Steroids: Electrophysiological Effects in Excitable Tissues. Neuroendocrinology. 96(2). 141–151. 6 indexed citations

Oberlander, Joseph G., Donna M. Porter, Carlos A.A. Penatti, et al.. (2012). Estrous cycle variations in GABAA receptor phosphorylation enable rapid modulation by anabolic androgenic steroids in the medial preoptic area. Neuroscience. 226. 397–410. 8 indexed citations

Oberlander, Joseph G., Donna M. Porter, Carlos A.A. Penatti, & Leslie Henderson. (2011). Anabolic Androgenic Steroid Abuse: Multiple Mechanisms of Regulation of GABAergic Synapses in Neuroendocrine Control Regions of the Rodent Forebrain. Journal of Neuroendocrinology. 24(1). 202–214. 24 indexed citations

Maue, Robert A., Robert W. Burgess, Bing Wang, et al.. (2011). A novel mouse model of Niemann–Pick type C disease carrying a D1005G-Npc1 mutation comparable to commonly observed human mutations. Human Molecular Genetics. 21(4). 730–750. 103 indexed citations

Robinson, Siobhan, Carlos A.A. Penatti, & Ann S. Clark. (2011). The role of the androgen receptor in anabolic androgenic steroid-induced aggressive behavior in C57BL/6J and Tfm mice. Hormones and Behavior. 61(1). 67–75. 23 indexed citations

Penatti, Carlos A.A., Joseph G. Oberlander, Matthew C. Davis, Donna M. Porter, & Leslie Henderson. (2011). Chronic exposure to anabolic androgenic steroids alters activity and synaptic function in neuroendocrine control regions of the female mouse. Neuropharmacology. 61(4). 653–664. 22 indexed citations

Costine, Beth A., Joseph G. Oberlander, Matthew C. Davis, et al.. (2010). Chronic anabolic androgenic steroid exposure alters corticotropin releasing factor expression and anxiety-like behaviors in the female mouse. Psychoneuroendocrinology. 35(10). 1473–1485. 30 indexed citations

10.

Penatti, Carlos A.A., Matthew C. Davis, Donna M. Porter, & Leslie Henderson. (2010). Altered GABA _A Receptor-Mediated Synaptic Transmission Disrupts the Firing of Gonadotropin-Releasing Hormone Neurons in Male Mice under Conditions That Mimic Steroid Abuse. Journal of Neuroscience. 30(19). 6497–6506. 29 indexed citations

11.

Penatti, Carlos A.A., Beth A. Costine, Donna M. Porter, & Leslie Henderson. (2009). Effects of chronic exposure to an anabolic androgenic steroid cocktail on α5-receptor-mediated GABAergic transmission and neural signaling in the forebrain of female mice. Neuroscience. 161(2). 526–537. 21 indexed citations

12.

Penatti, Carlos A.A., Donna M. Porter, & Leslie Henderson. (2009). Chronic Exposure to Anabolic Androgenic Steroids Alters Neuronal Function in the Mammalian Forebrain via Androgen Receptor- and Estrogen Receptor-Mediated Mechanisms. Journal of Neuroscience. 29(40). 12484–12496. 39 indexed citations

13.

Adhikari, Avishek, Carlos A.A. Penatti, Rodrigo R. Resende, et al.. (2006). 5-Aminolevulinate and 4, 5-dioxovalerate ions decrease GABAA receptor density in neuronal cells, synaptosomes and rat brain. Brain Research. 1093(1). 95–104. 31 indexed citations

14.

Bechara, Etelvino José Henriques, Fernando Dutra, Kelly Polido Kaneshiro Olympio, et al.. (2006). The dual face of endogenous α-aminoketones: Pro-oxidizing metabolic weapons. Comparative Biochemistry and Physiology Part C Toxicology & Pharmacology. 146(1-2). 88–110. 45 indexed citations

15.

Clark, Ann S., Beth A. Costine, B Jones, et al.. (2006). Sex- and age-specific effects of anabolic androgenic steroids on reproductive behaviors and on GABAergic transmission in neuroendocrine control regions. Brain Research. 1126(1). 122–138. 37 indexed citations

16.

Henderson, Leslie, Carlos A.A. Penatti, B Jones, Paul Yang, & Ann S. Clark. (2005). Anabolic androgenic steroids and forebrain GABAergic transmission. Neuroscience. 138(3). 793–799. 47 indexed citations

17.

Penatti, Carlos A.A., Donna M. Porter, B Jones, & Leslie Henderson. (2005). Sex-specific effects of chronic anabolic androgenic steroid treatment on GABAA receptor expression and function in adolescent mice. Neuroscience. 135(2). 533–543. 23 indexed citations

18.

Penatti, Carlos A.A., Sonia A. Gurgueira, Etelvino José Henriques Bechara, & Marilene Demasi. (1998). Neuroleptic drug-stimulated iron uptake by synaptosome preparations of rat cerebral cortex. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1407(1). 61–68. 2 indexed citations

19.

Demasi, Marilene, Carlos A.A. Penatti, Roberto DeLucia, & Etelvino José Henriques Bechara. (1996). The prooxidant effect of 5-aminolevulinic acid in the brain tissue of rats: Implications in neuropsychiatric manifestations in porphyrias. Free Radical Biology and Medicine. 20(3). 291–299. 93 indexed citations

20.

Penatti, Carlos A.A., Etelvino José Henriques Bechara, & Marilene Demasi. (1996). δ-Aminolevulinic Acid-Induced Synaptosomal Ca2+Uptake and Mitochondrial Permeabilization. Archives of Biochemistry and Biophysics. 335(1). 53–60. 10 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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