Carl W. Steiner

883 total citations
11 papers, 689 citations indexed

About

Carl W. Steiner is a scholar working on Rheumatology, Oncology and Immunology. According to data from OpenAlex, Carl W. Steiner has authored 11 papers receiving a total of 689 indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Rheumatology, 6 papers in Oncology and 6 papers in Immunology. Recurrent topics in Carl W. Steiner's work include Systemic Lupus Erythematosus Research (6 papers), T-cell and B-cell Immunology (5 papers) and Cytokine Signaling Pathways and Interactions (4 papers). Carl W. Steiner is often cited by papers focused on Systemic Lupus Erythematosus Research (6 papers), T-cell and B-cell Immunology (5 papers) and Cytokine Signaling Pathways and Interactions (4 papers). Carl W. Steiner collaborates with scholars based in Austria, Germany and Finland. Carl W. Steiner's co-authors include Josef S Smolen, G Steiner, Martin Aringer, W. Graninger, Theresa Kapral, Daniel Aletaha, Daniela Seidinger, Sabine Steiner, Johannes Grisar and Günter Weigel and has published in prestigious journals such as Circulation, Diabetes and International Immunology.

In The Last Decade

Carl W. Steiner

11 papers receiving 675 citations

Peers

Carl W. Steiner

IMMUN

RHEUM

ONCOL

Johannes Grisar Austria

Takayuki Katsuyama Japan

Chikako Fukasawa Japan

Ágnes Szentpétery Ireland

Petra Reusch Germany

Martin Judex Germany

Mary Donnelly United Kingdom

Róisín Armstrong United Kingdom

Hongyu Yang United States

Linyu Geng China

Johannes Grisar Austria

Carl W. Steiner

235 ×0.8

IMMUN

391 ×1.5

RHEUM

379 ×1.5

181 ×0.8

ONCOL

51 ×0.9

Citations per year, relative to Carl W. Steiner Carl W. Steiner (= 1×) peers Johannes Grisar

Countries citing papers authored by Carl W. Steiner

Since Specialization

Citations

This map shows the geographic impact of Carl W. Steiner's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Carl W. Steiner with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Carl W. Steiner more than expected).

Fields of papers citing papers by Carl W. Steiner

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Carl W. Steiner. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Carl W. Steiner. The network helps show where Carl W. Steiner may publish in the future.

Co-authorship network of co-authors of Carl W. Steiner

This figure shows the co-authorship network connecting the top 25 collaborators of Carl W. Steiner. A scholar is included among the top collaborators of Carl W. Steiner based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Carl W. Steiner. Carl W. Steiner is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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11 of 11 papers shown

Karonitsch, Thomas, Victoria Saferding, Markus Kieler, et al.. (2023). Amino Acids Fueling Fibroblast‐Like Synoviocyte Activation and Arthritis By Regulating Chemokine Expression and Leukocyte Migration. Arthritis & Rheumatology. 76(4). 531–540. 3 indexed citations

Haemmerle, Monika, Thomas Keller, Gerda Egger, et al.. (2013). Enhanced Lymph Vessel Density, Remodeling, and Inflammation Are Reflected by Gene Expression Signatures in Dermal Lymphatic Endothelial Cells in Type 2 Diabetes. Diabetes. 62(7). 2509–2529. 43 indexed citations

Herman, Sonja, Michael Klimas, Michael Bonelli, et al.. (2012). Regulatory T cells form stable and long-lasting cell cluster with myeloid dendritic cells (DC). International Immunology. 24(7). 417–426. 14 indexed citations

Karonitsch, Thomas, K Dalwigk, Carl W. Steiner, et al.. (2011). Interferon signals and monocytic sensitization of the interferon‐γ signaling pathway in the peripheral blood of patients with rheumatoid arthritis. Arthritis & Rheumatism. 64(2). 400–408. 29 indexed citations

Karonitsch, Thomas, Eva Feierl, Carl W. Steiner, et al.. (2009). Activation of the interferon‐γ signaling pathway in systemic lupus erythematosus peripheral blood mononuclear cells. Arthritis & Rheumatism. 60(5). 1463–1471. 77 indexed citations

Feierl, Eva, Josef S Smolen, Thomas Karonitsch, et al.. (2007). Engulfed cell remnants, and not cells undergoing apoptosis, constitute the LE-cell phenomenon. Autoimmunity. 40(4). 315–321. 4 indexed citations

Wick, Nikolaus, Pipsa Saharinen, Juha Saharinen, et al.. (2007). Transcriptomal comparison of human dermal lymphatic endothelial cells ex vivo and in vitro. Physiological Genomics. 28(2). 179–192. 82 indexed citations

Aringer, Martin, G Steiner, W. Graninger, et al.. (2006). Effects of short‐term infliximab therapy on autoantibodies in systemic lupus erythematosus. Arthritis & Rheumatism. 56(1). 274–279. 84 indexed citations

Grisar, Johannes, Daniel Aletaha, Carl W. Steiner, et al.. (2005). Depletion of Endothelial Progenitor Cells in the Peripheral Blood of Patients With Rheumatoid Arthritis. Circulation. 111(2). 204–211. 218 indexed citations

10.

Redlich, Kurt, Hans P. Kiener, Georg Schett, et al.. (2001). Overexpression of transcription factor Ets-1 in rheumatoid arthritis synovial membrane: Regulation of expression and activation by interleukin-1 and tumor necrosis factor ?. Arthritis & Rheumatism. 44(2). 266–274. 55 indexed citations

11.

Aringer, Martin, Carl W. Steiner, Hans P. Kiener, et al.. (1994). High levels of bcl‐2 protein in circulating t lymphocytes, but not b lymphocytes, of patients with systemic lupus erythematosus. Arthritis & Rheumatism. 37(10). 1423–1430. 80 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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