Caiping Ren

3.7k total citations
116 papers, 2.6k citations indexed

About

Caiping Ren is a scholar working on Molecular Biology, Cancer Research and Oncology. According to data from OpenAlex, Caiping Ren has authored 116 papers receiving a total of 2.6k indexed citations (citations by other indexed papers that have themselves been cited), including 81 papers in Molecular Biology, 38 papers in Cancer Research and 26 papers in Oncology. Recurrent topics in Caiping Ren's work include RNA modifications and cancer (26 papers), Cancer-related molecular mechanisms research (22 papers) and MicroRNA in disease regulation (18 papers). Caiping Ren is often cited by papers focused on RNA modifications and cancer (26 papers), Cancer-related molecular mechanisms research (22 papers) and MicroRNA in disease regulation (18 papers). Caiping Ren collaborates with scholars based in China, Saudi Arabia and United States. Caiping Ren's co-authors include Xingjun Jiang, Weidong Liu, Bin Zhu, Ming Zhao, Xiangling Feng, Kai‐Tai Yao, Hecheng Zhu, Kai‐Tai Yao, Hongjuan Xu and Yuxiang Chen and has published in prestigious journals such as Oncogene, Scientific Reports and The Lancet Oncology.

In The Last Decade

Caiping Ren

112 papers receiving 2.6k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Caiping Ren China 31 1.7k 896 543 296 281 116 2.6k
Xiaoli Wu China 30 1.8k 1.0× 741 0.8× 438 0.8× 206 0.7× 181 0.6× 82 2.8k
Tiziana Annese Italy 30 1.7k 1.0× 772 0.9× 800 1.5× 326 1.1× 548 2.0× 99 3.2k
Xudong Wang China 33 2.2k 1.3× 1.4k 1.5× 620 1.1× 229 0.8× 323 1.1× 162 3.3k
Wen Yang China 24 2.0k 1.2× 1.2k 1.3× 887 1.6× 193 0.7× 342 1.2× 42 3.2k
Baocun Sun China 38 2.1k 1.3× 1.3k 1.5× 974 1.8× 294 1.0× 267 1.0× 64 2.9k
Wenxiu Zhao China 28 1.4k 0.8× 596 0.7× 577 1.1× 186 0.6× 462 1.6× 67 2.4k
Go J. Yoshida Japan 21 1.5k 0.9× 949 1.1× 931 1.7× 313 1.1× 248 0.9× 25 2.5k
John G. Clohessy United States 28 2.9k 1.7× 1.4k 1.6× 756 1.4× 371 1.3× 411 1.5× 50 3.7k
Kou‐Juey Wu Taiwan 30 2.5k 1.5× 894 1.0× 661 1.2× 413 1.4× 259 0.9× 59 3.6k
Jan Bouchal Czechia 27 1.5k 0.9× 613 0.7× 939 1.7× 449 1.5× 204 0.7× 86 2.6k

Countries citing papers authored by Caiping Ren

Since Specialization
Citations

This map shows the geographic impact of Caiping Ren's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Caiping Ren with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Caiping Ren more than expected).

Fields of papers citing papers by Caiping Ren

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Caiping Ren. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Caiping Ren. The network helps show where Caiping Ren may publish in the future.

Co-authorship network of co-authors of Caiping Ren

This figure shows the co-authorship network connecting the top 25 collaborators of Caiping Ren. A scholar is included among the top collaborators of Caiping Ren based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Caiping Ren. Caiping Ren is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Liu, Weidong, et al.. (2025). The role of non-coding RNA regulates stem cell programmed death in disease therapy. Non-coding RNA Research. 13. 57–70. 4 indexed citations
2.
Yin, Wen, et al.. (2023). A prognostic signature based on snoRNA predicts the overall survival of lower-grade glioma patients. Frontiers in Immunology. 14. 1138363–1138363. 1 indexed citations
3.
Li, Shasha, Hui Liu, Weidong Liu, et al.. (2023). ESRG is critical to maintain the cell survival and self-renewal/pluripotency of hPSCs by collaborating with MCM2 to suppress p53 pathway. International Journal of Biological Sciences. 19(3). 916–935. 11 indexed citations
4.
Xu, Hongjuan, Yihan Li, Ning Shi, et al.. (2023). FLOT2 promotes nasopharyngeal carcinoma progression through suppression of TGF-β pathway via facilitating CD109 expression. iScience. 27(1). 108580–108580. 4 indexed citations
5.
6.
Zhu, Hecheng, Ming Zhao, Quanwei Zhou, et al.. (2022). Potential mechanisms underlying the promoting effects of 3D collagen scaffold culture on stemness and drug resistance of glioma cells. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1868(11). 166522–166522. 7 indexed citations
7.
8.
Liu, Hui, Shasha Li, Caiping Ren, et al.. (2020). Generation of an ESRG Pr-tdTomato reporter human embryonic stem cell line, CSUe011-A, using CRISPR/Cas9 editing. Stem Cell Research. 48. 101983–101983. 4 indexed citations
9.
Ren, Caiping, et al.. (2020). Advances in targeted therapy mainly based on signal pathways for nasopharyngeal carcinoma. Signal Transduction and Targeted Therapy. 5(1). 245–245. 70 indexed citations
10.
Li, Jian, et al.. (2018). Research Progress on Reversing Multidrug Resistance in Tumors by Using Chinese Medicine. Chinese Journal of Integrative Medicine. 24(6). 474–480. 17 indexed citations
11.
Lin, Ling, Zhang Yang-de, Ziyu Chen, Yuxiang Chen, & Caiping Ren. (2016). The clinicopathological significance of miR-149 and PARP-2 in hepatocellular carcinoma and their roles in chemo/radiotherapy. Tumor Biology. 37(9). 12339–12346. 24 indexed citations
12.
Zeng, Fei, Min Xue, Ting Xiao, et al.. (2016). MiR-200b promotes the cell proliferation and metastasis of cervical cancer by inhibiting FOXG1. Biomedicine & Pharmacotherapy. 79. 294–301. 34 indexed citations
13.
Huang, Wei, Jie Liu, Xiangling Feng, et al.. (2015). DLC-1 induces mitochondrial apoptosis and epithelial mesenchymal transition arrest in nasopharyngeal carcinoma by targeting EGFR/Akt/NF-κB pathway. Medical Oncology. 32(4). 115–115. 26 indexed citations
14.
15.
Liu, Jie, Wei Huang, Caiping Ren, et al.. (2015). Flotillin-2 promotes metastasis of nasopharyngeal carcinoma by activating NF-κB and PI3K/Akt3 signaling pathways. Scientific Reports. 5(1). 11614–11614. 47 indexed citations
16.
Shi, Jia, Caiping Ren, Hui Liu, et al.. (2015). An ESRG-interacting protein, COXII, is involved in pro-apoptosis of human embryonic stem cells. Biochemical and Biophysical Research Communications. 460(2). 130–135. 6 indexed citations
17.
Zuo, Jianhong, Chunshun Zhang, Caiping Ren, et al.. (2014). Secretory leukocyte protease inhibitor is a proliferation and survival factor for pancreatic cancer cells. Clinical & Translational Oncology. 17(4). 314–321. 12 indexed citations
18.
Zhou, Wen, Xiangling Feng, Caiping Ren, et al.. (2013). Over-expression of BCAT1, a c-Myc target gene, induces cell proliferation, migration and invasion in nasopharyngeal carcinoma. Molecular Cancer. 12(1). 53–53. 121 indexed citations
19.
Zhang, Hongbo, Weidong Liu, Xiangling Feng, et al.. (2011). Identification of ABCG2+ cells in nasopharyngeal carcinoma cells. Oncology Reports. 27(4). 1177–1187. 11 indexed citations
20.
Feng, Xiangling, Wen Zhou, Hong Li, et al.. (2008). The DLC-1 −29A/T Polymorphism Is Not Associated with Nasopharyngeal Carcinoma Risk in Chinese Population. Genetic Testing. 12(3). 345–349. 10 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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