C. David Wood

1.4k total citations
20 papers, 1.1k citations indexed

About

C. David Wood is a scholar working on Molecular Biology, Oncology and Pathology and Forensic Medicine. According to data from OpenAlex, C. David Wood has authored 20 papers receiving a total of 1.1k indexed citations (citations by other indexed papers that have themselves been cited), including 10 papers in Molecular Biology, 8 papers in Oncology and 6 papers in Pathology and Forensic Medicine. Recurrent topics in C. David Wood's work include Viral-associated cancers and disorders (8 papers), Lymphoma Diagnosis and Treatment (5 papers) and Histiocytic Disorders and Treatments (4 papers). C. David Wood is often cited by papers focused on Viral-associated cancers and disorders (8 papers), Lymphoma Diagnosis and Treatment (5 papers) and Histiocytic Disorders and Treatments (4 papers). C. David Wood collaborates with scholars based in United Kingdom, United States and Germany. C. David Wood's co-authors include Tina M. Thornton, Mercedes Rincón, Roger J. Davis, Guadalupe Sabio, Michelle J. West, James L. Clements, Bin Deng, Dwight E. Matthews, Gustavo Pedraza‐Alva and Bradley W. Doble and has published in prestigious journals such as Science, Nucleic Acids Research and Journal of Biological Chemistry.

In The Last Decade

C. David Wood

19 papers receiving 1.1k citations

Peers

C. David Wood

ONCOL

IMMUN

PFM

Emma Hickman United States

Hideyuki Komekado Japan

Raymond Yu United States

Laurent Pouyet France

Caroline Bouchard Canada

Ulrich A. K. Betz Germany

George S.W. Tsao Hong Kong

Xuan Qu United States

Jagan Muppidi United States

Emma Hickman United States

C. David Wood

805 ×1.2

605 ×1.9

ONCOL

236 ×0.9

IMMUN

127 ×0.8

PFM

162 ×1.1

Citations per year, relative to C. David Wood C. David Wood (= 1×) peers Emma Hickman

Countries citing papers authored by C. David Wood

Since Specialization

Citations

This map shows the geographic impact of C. David Wood's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by C. David Wood with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites C. David Wood more than expected).

Fields of papers citing papers by C. David Wood

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by C. David Wood. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by C. David Wood. The network helps show where C. David Wood may publish in the future.

Co-authorship network of co-authors of C. David Wood

This figure shows the co-authorship network connecting the top 25 collaborators of C. David Wood. A scholar is included among the top collaborators of C. David Wood based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with C. David Wood. C. David Wood is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Wood, C. David, et al.. (2025). Leveraging the role of the microbiome in endometriosis: novel non-invasive and therapeutic approaches. Frontiers in Immunology. 16. 1631522–1631522.

Günther, Thomas, C. David Wood, Steven Wingett, et al.. (2025). Repression of CADM1 transcription by HPV type 18 is mediated by three-dimensional rearrangement of promoter-enhancer interactions. PLoS Pathogens. 21(1). e1012506–e1012506. 2 indexed citations

Mitchell, Simon, et al.. (2022). Dissecting the impact of bromodomain inhibitors on the Interferon Regulatory Factor 4‐MYC oncogenic axis in multiple myeloma. Hematological Oncology. 40(3). 417–429. 5 indexed citations

Wood, C. David, et al.. (2022). Regulation of B cell receptor signalling by Epstein–Barr virus nuclear antigens. Biochemical Journal. 479(23). 2395–2417. 9 indexed citations

Khatri, Ritika, et al.. (2019). Increased association between Epstein-Barr virus EBNA2 from type 2 strains and the transcriptional repressor BS69 restricts EBNA2 activity. PLoS Pathogens. 15(7). e1007458–e1007458. 15 indexed citations

Wood, C. David, et al.. (2018). Enhancer Control of MicroRNA miR-155 Expression in Epstein-Barr Virus-Infected B Cells. Journal of Virology. 92(19). 40 indexed citations

Cotic, Marius, C. David Wood, Ian J. Groves, et al.. (2018). Disruption of CTCF-YY1–dependent looping of the human papillomavirus genome activates differentiation-induced viral oncogene transcription. PLoS Biology. 16(10). e2005752–e2005752. 57 indexed citations

Brocard, Michèle, et al.. (2018). Pumilio directs deadenylation-associated translational repression of the cyclin-dependent kinase 1 activator RGC-32. Nucleic Acids Research. 46(7). 3707–3725. 16 indexed citations

Zhang, Helen, et al.. (2017). The miR-23a~27a~24-2 microRNA cluster buffers transcription and signaling pathways during hematopoiesis. PLoS Genetics. 13(7). e1006887–e1006887. 37 indexed citations

10.

Webb, Helen, C. David Wood, Bettina Kempkes, et al.. (2016). RUNXsuper-enhancer control through the Notch pathway by Epstein-Barr virus transcription factors regulates B cell growth. Nucleic Acids Research. 44(10). 4636–4650. 52 indexed citations

11.

Zhang, Helen, et al.. (2016). Regulation of Hematopoietic Stem and Progenitor Cell Differentiation By Mirn23a/b Micrornas. Blood. 128(22). 3880–3880. 1 indexed citations

12.

Wood, C. David, Helen Webb, Claire Shannon‐Lowe, et al.. (2016). MYC activation and BCL2L11 silencing by a tumour virus through the large-scale reconfiguration of enhancer-promoter hubs. eLife. 5. 81 indexed citations

13.

Wood, C. David, Tim J. Cooper, Aditi Kanhere, et al.. (2013). Modulation of Enhancer Looping and Differential Gene Targeting by Epstein-Barr Virus Transcription Factors Directs Cellular Reprogramming. PLoS Pathogens. 9(9). e1003636–e1003636. 73 indexed citations

14.

Wood, C. David, et al.. (2012). Downregulation of Integrin Receptor-Signaling Genes by Epstein-Barr Virus EBNA 3C via Promoter-Proximal and -Distal Binding Elements. Journal of Virology. 86(9). 5165–5178. 40 indexed citations

15.

Wood, C. David, et al.. (2011). Upregulation of the Cell-Cycle Regulator RGC-32 in Epstein-Barr Virus-Immortalized Cells. PLoS ONE. 6(12). e28638–e28638. 22 indexed citations

16.

Wood, C. David, Tina M. Thornton, Guadalupe Sabio, Roger J. Davis, & Mercedes Rincón. (2009). Nuclear Localization of p38 MAPK in Response to DNA Damage. International Journal of Biological Sciences. 5(5). 428–437. 122 indexed citations

17.

Thornton, Tina M., Gustavo Pedraza‐Alva, Bin Deng, et al.. (2008). Phosphorylation by p38 MAPK as an Alternative Pathway for GSK3β Inactivation. Science. 320(5876). 667–670. 402 indexed citations

18.

Wood, C. David, April Kelly, Sharon A. Matthews, & Doreen A. Cantrell. (2007). Phosphoinositide‐dependent protein kinase‐1 (PDK1)‐independent activation of the protein kinase C substrate, protein kinase D. FEBS Letters. 581(18). 3494–3498. 6 indexed citations

19.

Spitaler, Martin, et al.. (2006). Diacylglycerol and Protein Kinase D Localization during T Lymphocyte Activation. Immunity. 24(5). 535–546. 95 indexed citations

20.

Wood, C. David, Ulrica Marklund, & Doreen A. Cantrell. (2004). Dual Phospholipase C/Diacylglycerol Requirement for Protein Kinase D1 Activation in Lymphocytes. Journal of Biological Chemistry. 280(7). 6245–6251. 26 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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