Burkhard Kneitz

7.4k total citations · 1 hit paper
72 papers, 5.8k citations indexed

About

Burkhard Kneitz is a scholar working on Molecular Biology, Cancer Research and Pulmonary and Respiratory Medicine. According to data from OpenAlex, Burkhard Kneitz has authored 72 papers receiving a total of 5.8k indexed citations (citations by other indexed papers that have themselves been cited), including 38 papers in Molecular Biology, 29 papers in Cancer Research and 26 papers in Pulmonary and Respiratory Medicine. Recurrent topics in Burkhard Kneitz's work include Prostate Cancer Treatment and Research (22 papers), Prostate Cancer Diagnosis and Treatment (17 papers) and MicroRNA in disease regulation (12 papers). Burkhard Kneitz is often cited by papers focused on Prostate Cancer Treatment and Research (22 papers), Prostate Cancer Diagnosis and Treatment (17 papers) and MicroRNA in disease regulation (12 papers). Burkhard Kneitz collaborates with scholars based in Germany, United States and Belgium. Burkhard Kneitz's co-authors include Winfried Edelmann, Harry Hou, Martin Spahn, Michael P. Lisanti, Xiao Lan Zhang, Richard D. Kolodner, Anneliese Schimpl, Robert G. Russell, Paula E. Cohen and Maomi Li and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Nature Communications.

In The Last Decade

Burkhard Kneitz

69 papers receiving 5.7k citations

Hit Papers

Caveolin-1 Null Mice Are Viable but Show Evidence of Hype... 2001 2026 2009 2017 2001 250 500 750

Peers

Burkhard Kneitz
Alexei Protopopov United States
Dennis K. Watson United States
Andrea Morrione United States
Janusz Puc United States
Danny Liaw United States
Ralf Lesche Germany
Raymond S. Yeung United States
Alexei Protopopov United States
Burkhard Kneitz
Citations per year, relative to Burkhard Kneitz Burkhard Kneitz (= 1×) peers Alexei Protopopov

Countries citing papers authored by Burkhard Kneitz

Since Specialization
Citations

This map shows the geographic impact of Burkhard Kneitz's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Burkhard Kneitz with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Burkhard Kneitz more than expected).

Fields of papers citing papers by Burkhard Kneitz

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Burkhard Kneitz. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Burkhard Kneitz. The network helps show where Burkhard Kneitz may publish in the future.

Co-authorship network of co-authors of Burkhard Kneitz

This figure shows the co-authorship network connecting the top 25 collaborators of Burkhard Kneitz. A scholar is included among the top collaborators of Burkhard Kneitz based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Burkhard Kneitz. Burkhard Kneitz is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Cossa, Giacomo, Christina Schülein‐Völk, Francisco Montesinos, et al.. (2024). PAF1c links S-phase progression to immune evasion and MYC function in pancreatic carcinoma. Nature Communications. 15(1). 1446–1446. 7 indexed citations
2.
Kalogirou, Charis, Daniel W. Schafer, Markus Krebs, et al.. (2015). Metformin-Derived Growth Inhibition in Renal Cell Carcinoma Depends on miR-21-Mediated <b><i>PTEN</i></b> Expression. Urologia Internationalis. 96(1). 106–115. 30 indexed citations
3.
Briganti, Alberto, R. Jeffrey Karnes, Giorgio Gandaglia, et al.. (2015). Natural history of surgically treated high-risk prostate cancer. Urologic Oncology Seminars and Original Investigations. 33(4). 163.e7–163.e13. 98 indexed citations
4.
Kneitz, Burkhard, Markus Krebs, Charis Kalogirou, et al.. (2014). Survival in Patients with High-Risk Prostate Cancer Is Predicted by miR-221, Which Regulates Proliferation, Apoptosis, and Invasion of Prostate Cancer Cells by Inhibiting IRF2 and SOCS3. Cancer Research. 74(9). 2591–2603. 106 indexed citations
5.
Schmid, Michael, Claus Steinlein, Qi Tian, et al.. (2014). Mosaic variegated aneuploidy in mouse BubR1 deficient embryos and pregnancy loss in human. Chromosome Research. 22(3). 375–392. 8 indexed citations
6.
Vergho, Daniel, Susanne Kneitz, Charis Kalogirou, et al.. (2014). Impact of miR-21, miR-126 and miR-221 as Prognostic Factors of Clear Cell Renal Cell Carcinoma with Tumor Thrombus of the Inferior Vena Cava. PLoS ONE. 9(10). e109877–e109877. 29 indexed citations
7.
Schubert, Maria, Martin Spahn, Susanne Kneitz, et al.. (2013). Distinct microRNA Expression Profile in Prostate Cancer Patients with Early Clinical Failure and the Impact of let-7 as Prognostic Marker in High-Risk Prostate Cancer. PLoS ONE. 8(6). e65064–e65064. 64 indexed citations
9.
Reiß, Cora, et al.. (2010). Conditional inactivation of MLH1 in thymic and naive T-cells in mice leads to a limited incidence of lymphoblastic T-cell lymphomas. Leukemia & lymphoma. 51(10). 1875–1886. 10 indexed citations
10.
Spahn, Martin, Susanne Kneitz, Claus Jürgen Scholz, et al.. (2009). Expression of microRNA‐221 is progressively reduced in aggressive prostate cancer and metastasis and predicts clinical recurrence. International Journal of Cancer. 127(2). 394–403. 189 indexed citations
11.
Wagner, Toni U., et al.. (2008). STAT3 and SMAD1 Signaling in Medaka Embryonic Stem-Like Cells and Blastula Embryos. Stem Cells and Development. 18(1). 151–160. 7 indexed citations
12.
Kan, Rui, Nadine K. Kolas, Elena Avdievich, et al.. (2007). Comparative Analysis of Meiotic Progression in Female Mice Bearing Mutations in Genes of the DNA Mismatch Repair Pathway1. Biology of Reproduction. 78(3). 462–471. 31 indexed citations
13.
Hoffmann, Anja, Sheena C. Kerr, Julia Jellusova, et al.. (2007). Siglec-G is a B1 cell–inhibitory receptor that controls expansion and calcium signaling of the B1 cell population. Nature Immunology. 8(7). 695–704. 160 indexed citations
14.
Ju, Wenjun, Petra Knaus, Raju Kucherlapati, et al.. (2006). B Cell-Specific Deficiency for Smad2 In Vivo Leads to Defects in TGF-β-Directed IgA Switching and Changes in B Cell Fate. The Journal of Immunology. 176(4). 2389–2396. 37 indexed citations
15.
Wei, Kaichun, Allan Clark, Michael Kane, et al.. (2003). Inactivation of Exonuclease 1 in mice results in DNA mismatch repair defects, increased cancer susceptibility, and male and female sterility. Genes & Development. 17(5). 603–614. 261 indexed citations
16.
Razani, Babak, Jeffery A. Engelman, Xiaobo Wang, et al.. (2001). Caveolin-1 Null Mice Are Viable but Show Evidence of Hyperproliferative and Vascular Abnormalities. Journal of Biological Chemistry. 276(41). 38121–38138. 975 indexed citations breakdown →
17.
Galbiati, Ferruccio, Jeffrey A. Engelman, Daniela Volonté, et al.. (2001). Caveolin-3 Null Mice Show a Loss of Caveolae, Changes in the Microdomain Distribution of the Dystrophin-Glycoprotein Complex, and T-tubule Abnormalities. Journal of Biological Chemistry. 276(24). 21425–21433. 367 indexed citations
18.
Wiesendanger, Margrit, Burkhard Kneitz, Winfried Edelmann, & Matthew D. Scharff. (2000). Somatic Hypermutation in Muts Homologue (Msh)3-, Msh6-, and Msh3/Msh6-Deficient Mice Reveals a Role for the Msh2–Msh6 Heterodimer in Modulating the Base Substitution Pattern. The Journal of Experimental Medicine. 191(3). 579–584. 159 indexed citations
19.
Edelmann, Winfried, Paula E. Cohen, Burkhard Kneitz, et al.. (1999). Mammalian MutS homologue 5 is required for chromosome pairing in meiosis. Nature Genetics. 21(1). 123–127. 312 indexed citations
20.
Schuh, Kai, et al.. (1998). The Interleukin 2 Receptor α Chain/CD25 Promoter Is a Target for Nuclear Factor of Activated T Cells. The Journal of Experimental Medicine. 188(7). 1369–1373. 73 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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