Brooke Snetsinger

978 total citations
18 papers, 664 citations indexed

About

Brooke Snetsinger is a scholar working on Molecular Biology, Hematology and Genetics. According to data from OpenAlex, Brooke Snetsinger has authored 18 papers receiving a total of 664 indexed citations (citations by other indexed papers that have themselves been cited), including 9 papers in Molecular Biology, 9 papers in Hematology and 6 papers in Genetics. Recurrent topics in Brooke Snetsinger's work include Acute Myeloid Leukemia Research (9 papers), Epigenetics and DNA Methylation (5 papers) and Myeloproliferative Neoplasms: Diagnosis and Treatment (5 papers). Brooke Snetsinger is often cited by papers focused on Acute Myeloid Leukemia Research (9 papers), Epigenetics and DNA Methylation (5 papers) and Myeloproliferative Neoplasms: Diagnosis and Treatment (5 papers). Brooke Snetsinger collaborates with scholars based in Canada, Brazil and Saudi Arabia. Brooke Snetsinger's co-authors include Michael J. Rauh, Rena Buckstein, Alyssa Cull, Richard A. Wells, Xiaolong Yang, Yawei Hao, Ling Min, Helena J. Janse van Rensburg, Taha Azad and Charles H. Graham and has published in prestigious journals such as SHILAP Revista de lepidopterología, Blood and Cancer Research.

In The Last Decade

Brooke Snetsinger

17 papers receiving 659 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Brooke Snetsinger Canada 8 287 242 175 174 141 18 664
Larissa Lordier France 14 303 1.1× 428 1.8× 74 0.4× 169 1.0× 141 1.0× 21 788
Marloes R. Tijssen United Kingdom 16 431 1.5× 430 1.8× 143 0.8× 100 0.6× 166 1.2× 29 852
Baskar Ramdas United States 14 367 1.3× 337 1.4× 235 1.3× 51 0.3× 182 1.3× 33 723
Tara MacRae Canada 14 466 1.6× 374 1.5× 159 0.9× 46 0.3× 123 0.9× 25 735
Philip E. Boulais United States 9 230 0.8× 285 1.2× 237 1.4× 49 0.3× 158 1.1× 15 643
Floortje L. Kessler Netherlands 11 214 0.7× 168 0.7× 158 0.9× 119 0.7× 48 0.3× 19 512
Tiphaine Bouriez‐Jones United Kingdom 10 166 0.6× 150 0.6× 278 1.6× 77 0.4× 52 0.4× 14 547
Caroline Wallace United States 11 237 0.8× 79 0.3× 232 1.3× 79 0.5× 53 0.4× 13 628
Ganesan Keerthivasan United States 9 283 1.0× 80 0.3× 89 0.5× 70 0.4× 65 0.5× 13 489
Camelia Iancu‐Rubin United States 15 292 1.0× 342 1.4× 76 0.4× 50 0.3× 281 2.0× 36 655

Countries citing papers authored by Brooke Snetsinger

Since Specialization
Citations

This map shows the geographic impact of Brooke Snetsinger's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Brooke Snetsinger with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Brooke Snetsinger more than expected).

Fields of papers citing papers by Brooke Snetsinger

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Brooke Snetsinger. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Brooke Snetsinger. The network helps show where Brooke Snetsinger may publish in the future.

Co-authorship network of co-authors of Brooke Snetsinger

This figure shows the co-authorship network connecting the top 25 collaborators of Brooke Snetsinger. A scholar is included among the top collaborators of Brooke Snetsinger based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Brooke Snetsinger. Brooke Snetsinger is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

18 of 18 papers shown
1.
Bentley, R. E., Charles C.T. Hindmarch, Kimberly J. Dunham‐Snary, et al.. (2021). The comprehensive transcriptome of human ductus arteriosus smooth muscle cells (hDASMC). SHILAP Revista de lepidopterología. 40. 107736–107736. 1 indexed citations
2.
Snetsinger, Brooke, Xiao Zhang, Guillaume Richard‐Carpentier, et al.. (2021). Validation, Implementation, and Clinical Impact of the Oncomine Myeloid Targeted-Amplicon DNA and RNA Ion Semiconductor Sequencing Assay. Journal of Molecular Diagnostics. 23(10). 1292–1305. 7 indexed citations
3.
Bentley, R. E., Charles C.T. Hindmarch, Kimberly J. Dunham‐Snary, et al.. (2021). The molecular mechanisms of oxygen-sensing in human ductus arteriosus smooth muscle cells: A comprehensive transcriptome profile reveals a central role for mitochondria. Genomics. 113(5). 3128–3140. 7 indexed citations
4.
Potus, François, Ashley Martin, Brooke Snetsinger, & Stephen L. Archer. (2020). Biventricular Assessment of Cardiac Function and Pressure-Volume Loops by Closed-Chest Catheterization in Mice. Journal of Visualized Experiments. 6 indexed citations
5.
Snetsinger, Brooke, et al.. (2019). Targeted, Amplicon-Based, Next-Generation Sequencing to Detect Age-Related Clonal Hematopoiesis. Methods in molecular biology. 2045. 167–180. 7 indexed citations
6.
Cook, Elina K., Mina Jamali, Liying Zhang, et al.. (2019). Comorbid and inflammatory characteristics of genetic subtypes of clonal hematopoiesis. Blood Advances. 3(16). 2482–2486. 97 indexed citations
7.
Siqueira, Lúcia H., et al.. (2019). Longitudinal sequencing of RUNX1 familial platelet disorder: new insights into genetic mechanisms of transformation to myeloid malignancies. British Journal of Haematology. 186(5). 724–734. 7 indexed citations
8.
Rensburg, Helena J. Janse van, Taha Azad, Ling Min, et al.. (2018). The Hippo Pathway Component TAZ Promotes Immune Evasion in Human Cancer through PD-L1. Cancer Research. 78(6). 1457–1470. 244 indexed citations
9.
Snetsinger, Brooke, Xiao Zhang, David Good, et al.. (2018). Validation and Clinical Impact of the Oncomine Myeloid Targeted DNA and RNA Ion Semiconductor Sequencing Assay. Blood. 132(Supplement 1). 5523–5523. 4 indexed citations
10.
11.
Cull, Alyssa, Brooke Snetsinger, Rena Buckstein, Richard A. Wells, & Michael J. Rauh. (2017). Tet2 restrains inflammatory gene expression in macrophages. Experimental Hematology. 55. 56–70.e13. 222 indexed citations
12.
Cull, Alyssa, Brooke Snetsinger, David Good, et al.. (2017). Overexpression of Arginase 1 is linked to DNMT3A and TET2 mutations in lower-grade myelodysplastic syndromes and chronic myelomonocytic leukemia. Leukemia Research. 65. 5–13. 15 indexed citations
13.
Hao, Yawei, et al.. (2017). TAZ induces lung cancer stem cell properties and tumorigenesis by up-regulating ALDH1A1. Oncotarget. 8(24). 38426–38443. 32 indexed citations
14.
Cull, Alyssa, Brooke Snetsinger, & Michael J. Rauh. (2016). Tet2 Deficiency Leads to an Increased Inflammatory Phenotype in Murine Macrophages. Blood. 128(22). 708–708. 2 indexed citations
15.
Cull, Alyssa, Brooke Snetsinger, David Good, et al.. (2016). Microenvironmental Links Between TET2/DNMT3A Mutations and Arginase 1 Overexpression in Human MDS/CMML. Blood. 128(22). 3164–3164.
16.
Cull, Alyssa, Brooke Snetsinger, & Michael J. Rauh. (2015). Tet2 Is a Novel Regulator of Murine Macrophage Differentiation and Polarization. Blood. 126(23). 646–646. 5 indexed citations
17.
Snetsinger, Brooke, Emily Heath, & Michael J. Rauh. (2015). Suspicious, Non-MDS-Diagnostic Bone Marrows Have a High Incidence of Clonal Hematopoiesis (CHIP), with MDS-like Clone Size but Restricted Mutation Burden. Blood. 126(23). 1668–1668. 1 indexed citations
18.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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