Boris Calderón

6.5k total citations · 2 hit papers
36 papers, 5.1k citations indexed

About

Boris Calderón is a scholar working on Immunology, Genetics and Surgery. According to data from OpenAlex, Boris Calderón has authored 36 papers receiving a total of 5.1k indexed citations (citations by other indexed papers that have themselves been cited), including 21 papers in Immunology, 18 papers in Genetics and 15 papers in Surgery. Recurrent topics in Boris Calderón's work include Diabetes and associated disorders (18 papers), Immune Cell Function and Interaction (16 papers) and Pancreatic function and diabetes (14 papers). Boris Calderón is often cited by papers focused on Diabetes and associated disorders (18 papers), Immune Cell Function and Interaction (16 papers) and Pancreatic function and diabetes (14 papers). Boris Calderón collaborates with scholars based in United States, Japan and Belgium. Boris Calderón's co-authors include Emil R. Unanue, Javier A. Carrero, Kenneth M. Murphy, Robert D. Schreiber, Michael Diamond, Brian T. Edelson, Masako Kohyama, Kai Hildner, Theresa L. Murphy and Whitney E. Purtha and has published in prestigious journals such as Nature, Science and Proceedings of the National Academy of Sciences.

In The Last Decade

Boris Calderón

36 papers receiving 5.0k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Batf3 Deficiency Reveals a Critical Role for CD8α ⁺ Dendritic Cells in Cytotoxic T Cell Immunity

2008 1.5k citations Kai Hildner, Brian T. Edelson et al. Science profile →
Embryonic and Adult-Derived Resident Cardiac Macrophages Are Maintained through Distinct Mechanisms at Steady State and during Inflammation

2014 1.1k citations Slava Epelman, Kory J. Lavine et al. Immunity profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Boris Calderón United States	27	3.4k	1.2k	1.1k	966	924	36	5.1k
Louis A. Matis United States	42	4.4k 1.3×	1.3k 1.1×	753 0.7×	1.1k 1.1×	698 0.8×	78	6.5k
Dirk Homann United States	33	2.5k 0.7×	623 0.5×	900 0.8×	591 0.6×	484 0.5×	75	4.0k
Richard J. Cornall United Kingdom	34	2.9k 0.8×	1.9k 1.6×	1.4k 1.4×	710 0.7×	724 0.8×	83	5.8k
Sally C. Kent United States	29	3.2k 0.9×	822 0.7×	1.8k 1.7×	1.3k 1.4×	544 0.6×	62	5.1k
Daniel L. Mueller United States	42	4.9k 1.4×	1.5k 1.3×	543 0.5×	362 0.4×	1.3k 1.4×	83	6.6k
Xuyu Zhou China	26	5.1k 1.5×	981 0.8×	689 0.6×	337 0.3×	1.1k 1.2×	64	6.4k
Yuqiong Liang United States	28	3.7k 1.1×	1.8k 1.5×	434 0.4×	198 0.2×	899 1.0×	36	6.2k
Julien C. Marie France	32	2.8k 0.8×	1.2k 1.0×	388 0.4×	290 0.3×	925 1.0×	62	4.5k
Susanne Berchtold Germany	30	2.3k 0.7×	1.1k 0.9×	683 0.6×	303 0.3×	1.1k 1.2×	66	4.0k
Paolo Monti Italy	28	2.2k 0.6×	909 0.8×	907 0.9×	793 0.8×	743 0.8×	55	4.1k

Countries citing papers authored by Boris Calderón

Since Specialization

Citations

This map shows the geographic impact of Boris Calderón's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Boris Calderón with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Boris Calderón more than expected).

Fields of papers citing papers by Boris Calderón

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Boris Calderón. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Boris Calderón. The network helps show where Boris Calderón may publish in the future.

Co-authorship network of co-authors of Boris Calderón

This figure shows the co-authorship network connecting the top 25 collaborators of Boris Calderón. A scholar is included among the top collaborators of Boris Calderón based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Boris Calderón. Boris Calderón is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Mullins, Garrett R., Michael E. Hodsdon, Greg Anglin, et al.. (2023). Tirzepatide Immunogenicity on Pharmacokinetics, Efficacy, and Safety: Analysis of Data From Phase 3 Studies. The Journal of Clinical Endocrinology & Metabolism. 109(2). 361–369. 4 indexed citations

Calderón, Boris, et al.. (2023). A Monovalent Fab Affinity-Capture and Elution Bridging Immunoassay Overcomes Rheumatoid Factor Interference while Accurately Detecting Antidrug Antibodies. The Journal of Applied Laboratory Medicine. 8(5). 896–908. 1 indexed citations

Harding, James J., Víctor Moreno, Yung‐Jue Bang, et al.. (2021). Blocking TIM-3 in Treatment-refractory Advanced Solid Tumors: A Phase Ia/b Study of LY3321367 with or without an Anti-PD-L1 Antibody. Clinical Cancer Research. 27(8). 2168–2178. 116 indexed citations

Hellmann, Matthew D., Nicoletta Bivi, Boris Calderón, et al.. (2021). Safety and Immunogenicity of LY3415244, a Bispecific Antibody Against TIM-3 and PD-L1, in Patients With Advanced Solid Tumors. Clinical Cancer Research. 27(10). 2773–2781. 84 indexed citations

Falchook, Gerald S., Marc Peeters, Sylvie Rottey, et al.. (2021). A phase 1a/1b trial of CSF-1R inhibitor LY3022855 in combination with durvalumab or tremelimumab in patients with advanced solid tumors. Investigational New Drugs. 39(5). 1284–1297. 47 indexed citations

Bivi, Nicoletta, Terry L. Moore, George H. Rodgers, et al.. (2019). Investigation of pre-existing reactivity to biotherapeutics can uncover potential immunogenic epitopes and predict immunogenicity risk. mAbs. 11(5). 861–869. 19 indexed citations

Calderón, Boris, et al.. (2017). Discrepancies between two immunoassays for the determination of MPO and PR3 autoantibodies. Clinica Chimica Acta. 470. 93–96. 1 indexed citations

Calderón, Boris, Javier A. Carrero, Stephen T. Ferris, et al.. (2015). The pancreas anatomy conditions the origin and properties of resident macrophages. The Journal of Experimental Medicine. 212(10). 1497–1512. 222 indexed citations

Vomund, Anthony N., Bernd H. Zinselmeyer, Boris Calderón, et al.. (2015). Beta cells transfer vesicles containing insulin to phagocytes for presentation to T cells. Proceedings of the National Academy of Sciences. 112(40). E5496–502. 81 indexed citations

10.

Ferris, Stephen T., Javier A. Carrero, James F. Mohan, et al.. (2014). A Minor Subset of Batf3-Dependent Antigen-Presenting Cells in Islets of Langerhans Is Essential for the Development of Autoimmune Diabetes. Immunity. 41(4). 657–669. 115 indexed citations

11.

Epelman, Slava, Kory J. Lavine, Anna E. Beaudin, et al.. (2014). Embryonic and Adult-Derived Resident Cardiac Macrophages Are Maintained through Distinct Mechanisms at Steady State and during Inflammation. Immunity. 40(1). 91–104. 1092 indexed citations breakdown →

12.

Mohan, James F., Boris Calderón, Mark S. Anderson, & Emil R. Unanue. (2013). Pathogenic CD4+ T cells recognizing an unstable peptide of insulin are directly recruited into islets bypassing local lymph nodes. The Journal of Experimental Medicine. 210(11). 2403–2414. 47 indexed citations

13.

Carrero, Javier A., Boris Calderón, Fadi Towfic, Maxim N. Artyomov, & Emil R. Unanue. (2013). Defining the Transcriptional and Cellular Landscape of Type 1 Diabetes in the NOD Mouse. PLoS ONE. 8(3). e59701–e59701. 93 indexed citations

14.

Calderón, Boris, Javier A. Carrero, Mark J. Miller, & Emil R. Unanue. (2011). Cellular and molecular events in the localization of diabetogenic T cells to islets of Langerhans. Proceedings of the National Academy of Sciences. 108(4). 1561–1566. 80 indexed citations

15.

McCartney, Stephen A., William Vermi, Silvia Lonardi, et al.. (2011). RNA sensor–induced type I IFN prevents diabetes caused by a β cell–tropic virus in mice. Journal of Clinical Investigation. 121(4). 1497–1507. 57 indexed citations

16.

Mohan, James F., Matteo Levisetti, Boris Calderón, et al.. (2010). Unique autoreactive T cells recognize insulin peptides generated within the islets of Langerhans in autoimmune diabetes. Nature Immunology. 11(4). 350–354. 147 indexed citations

17.

Hildner, Kai, Brian T. Edelson, Whitney E. Purtha, et al.. (2008). Batf3 Deficiency Reveals a Critical Role for CD8α ⁺ Dendritic Cells in Cytotoxic T Cell Immunity. Science. 322(5904). 1097–1100. 1543 indexed citations breakdown →

18.

Calderón, Boris, et al.. (2008). IFN-γ-Dependent Regulatory Circuits in Immune Inflammation Highlighted in Diabetes. The Journal of Immunology. 181(10). 6964–6974. 37 indexed citations

19.

Bredemeyer, Andrea, Beth A. Helmink, Cynthia L. Innes, et al.. (2008). DNA double-strand breaks activate a multi-functional genetic program in developing lymphocytes. Nature. 456(7223). 819–823. 115 indexed citations

20.

Calderón, Boris, Anish Suri, & Emil R. Unanue. (2006). In CD4+ T-Cell-Induced Diabetes, Macrophages Are the Final Effector Cells that Mediate Islet β-Cell Killing. American Journal Of Pathology. 169(6). 2137–2147. 96 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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