Bolong Lin

538 total citations
9 papers, 278 citations indexed

About

Bolong Lin is a scholar working on Molecular Biology, Immunology and Neurology. According to data from OpenAlex, Bolong Lin has authored 9 papers receiving a total of 278 indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Molecular Biology, 6 papers in Immunology and 2 papers in Neurology. Recurrent topics in Bolong Lin's work include Inflammasome and immune disorders (5 papers), Sphingolipid Metabolism and Signaling (3 papers) and Immune Cell Function and Interaction (3 papers). Bolong Lin is often cited by papers focused on Inflammasome and immune disorders (5 papers), Sphingolipid Metabolism and Signaling (3 papers) and Immune Cell Function and Interaction (3 papers). Bolong Lin collaborates with scholars based in China and Poland. Bolong Lin's co-authors include Rongbin Zhou, Wei Jiang, Xianming Deng, Hongbin He, Wei Jiang, Yun Chen, Xiaqiong Wang, Cong Li, Ming Ma and Yubo Zhou and has published in prestigious journals such as Immunity, Nature Immunology and Cell Death and Disease.

In The Last Decade

Bolong Lin

8 papers receiving 277 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Bolong Lin China	5	166	116	35	31	29	9	278
Xiaoxia Zuo China	8	205 1.2×	89 0.8×	15 0.4×	59 1.9×	12 0.4×	26	384
Yuxiong Lai China	11	165 1.0×	171 1.5×	10 0.3×	34 1.1×	52 1.8×	13	403
Mehdi Tourani Iran	8	138 0.8×	107 0.9×	37 1.1×	26 0.8×	4 0.1×	14	349
Emma E. Hornick United States	9	178 1.1×	157 1.4×	20 0.6×	25 0.8×	13 0.4×	15	309
Marc A. Napoleon United States	6	134 0.8×	93 0.8×	17 0.5×	164 5.3×	27 0.9×	9	372
Xiangyu Wang China	10	158 1.0×	30 0.3×	20 0.6×	16 0.5×	24 0.8×	33	291
Yunyun Jin China	14	184 1.1×	41 0.4×	58 1.7×	98 3.2×	9 0.3×	30	446
Shuwen Su China	7	90 0.5×	89 0.8×	20 0.6×	60 1.9×	7 0.2×	17	387
Vivien I. Maltez United States	7	291 1.8×	184 1.6×	19 0.5×	18 0.6×	35 1.2×	7	473
Xiaochen Guo China	10	235 1.4×	66 0.6×	33 0.9×	14 0.5×	5 0.2×	23	371

Countries citing papers authored by Bolong Lin

Since Specialization

Citations

This map shows the geographic impact of Bolong Lin's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Bolong Lin with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Bolong Lin more than expected).

Fields of papers citing papers by Bolong Lin

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Bolong Lin. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Bolong Lin. The network helps show where Bolong Lin may publish in the future.

Co-authorship network of co-authors of Bolong Lin

This figure shows the co-authorship network connecting the top 25 collaborators of Bolong Lin. A scholar is included among the top collaborators of Bolong Lin based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Bolong Lin. Bolong Lin is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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9 of 9 papers shown

1.

Zhou, Yubo, Bolong Lin, Ming Ma, et al.. (2025). Demyelination-derived lysophosphatidylserine promotes microglial dysfunction and neuropathology in a mouse model of Alzheimer’s disease. Cellular and Molecular Immunology. 22(2). 134–149. 2 indexed citations

2.

Cao, Rui, Bolong Lin, Hongbin He, et al.. (2025). CRM1 mediates ASC nuclear export and inflammasome activation. International Immunopharmacology. 153. 114503–114503.

3.

He, Hongbin, Ming‐Hui Qi, Bolong Lin, et al.. (2025). A small molecule directly targets NLRP3 to promote inflammasome activation and antitumor immunity. Cell Death and Disease. 16(1). 252–252. 3 indexed citations

4.

Li, Guo‐Yang, et al.. (2024). Ascorbyl palmitate ameliorates inflammatory diseases by inhibition of NLRP3 inflammasome. International Immunopharmacology. 131. 111915–111915. 3 indexed citations

5.

Lin, Bolong, Yubo Zhou, Ming Ma, et al.. (2024). GPR34 senses demyelination to promote neuroinflammation and pathologies. Cellular and Molecular Immunology. 21(10). 1131–1144. 7 indexed citations

6.

Yang, Mingyang, Cong Li, Bolong Lin, et al.. (2022). An RRx-001 Analogue With Potent Anti-NLRP3 Inflammasome Activity but Without High-Energy Nitro Functional Groups. Frontiers in Pharmacology. 13. 822833–822833. 9 indexed citations

7.

Wang, Xiaqiong, Juan Cai, Bolong Lin, et al.. (2021). GPR34-mediated sensing of lysophosphatidylserine released by apoptotic neutrophils activates type 3 innate lymphoid cells to mediate tissue repair. Immunity. 54(6). 1123–1136.e8. 77 indexed citations

8.

He, Hongbin, Bolong Lin, Yun Chen, et al.. (2021). RRx-001 ameliorates inflammatory diseases by acting as a potent covalent NLRP3 inhibitor. Cellular and Molecular Immunology. 18(6). 1425–1436. 108 indexed citations

9.

Liu, Lei, Tao Gong, Wanyin Tao, et al.. (2019). Commensal viruses maintain intestinal intraepithelial lymphocytes via noncanonical RIG-I signaling. Nature Immunology. 20(12). 1681–1691. 69 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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