Binhai Cong

612 total citations
23 papers, 470 citations indexed

About

Binhai Cong is a scholar working on Behavioral Neuroscience, Endocrinology, Diabetes and Metabolism and Cardiology and Cardiovascular Medicine. According to data from OpenAlex, Binhai Cong has authored 23 papers receiving a total of 470 indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Behavioral Neuroscience, 7 papers in Endocrinology, Diabetes and Metabolism and 5 papers in Cardiology and Cardiovascular Medicine. Recurrent topics in Binhai Cong's work include Stress Responses and Cortisol (8 papers), Hormonal Regulation and Hypertension (6 papers) and Adenosine and Purinergic Signaling (3 papers). Binhai Cong is often cited by papers focused on Stress Responses and Cortisol (8 papers), Hormonal Regulation and Hypertension (6 papers) and Adenosine and Purinergic Signaling (3 papers). Binhai Cong collaborates with scholars based in China, United Kingdom and United States. Binhai Cong's co-authors include Xin Ni, Xiaoyan Zhu, Jianqiang Lu, Lu Gao, Zhiping Tang, Xiaolu Tang, Long Wang, Qing Yu, Chang-Nan Wang and Long Wang and has published in prestigious journals such as Scientific Reports, The FASEB Journal and Free Radical Biology and Medicine.

In The Last Decade

Binhai Cong

21 papers receiving 466 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Binhai Cong China 14 154 84 84 77 69 23 470
Lai Jin China 16 284 1.8× 84 1.0× 88 1.0× 115 1.5× 23 0.3× 46 654
Fang Guo China 19 197 1.3× 90 1.1× 52 0.6× 110 1.4× 162 2.3× 42 816
Francesca Nardi Italy 13 172 1.1× 77 0.9× 90 1.1× 120 1.6× 13 0.2× 25 779
Beverley Balkau France 11 271 1.8× 55 0.7× 155 1.8× 79 1.0× 28 0.4× 15 695
Megan Partyka United States 10 167 1.1× 19 0.2× 96 1.1× 132 1.7× 104 1.5× 13 605
Mieke C. Louwe Norway 9 171 1.1× 30 0.4× 71 0.8× 51 0.7× 94 1.4× 19 442
Kyoichiro Tsuchiya Japan 12 132 0.9× 47 0.6× 85 1.0× 299 3.9× 96 1.4× 28 612
Yuichiro Izumi Japan 13 245 1.6× 30 0.4× 63 0.8× 58 0.8× 45 0.7× 61 564
Lifang Yang China 14 233 1.5× 60 0.7× 68 0.8× 32 0.4× 76 1.1× 27 496

Countries citing papers authored by Binhai Cong

Since Specialization
Citations

This map shows the geographic impact of Binhai Cong's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Binhai Cong with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Binhai Cong more than expected).

Fields of papers citing papers by Binhai Cong

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Binhai Cong. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Binhai Cong. The network helps show where Binhai Cong may publish in the future.

Co-authorship network of co-authors of Binhai Cong

This figure shows the co-authorship network connecting the top 25 collaborators of Binhai Cong. A scholar is included among the top collaborators of Binhai Cong based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Binhai Cong. Binhai Cong is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
2.
Song, Jin‐Chao, et al.. (2024). Bradykinin attenuates endothelial-mesenchymal transition following cardiac ischemia-reperfusion injury. European Journal of Pharmacology. 971. 176556–176556. 1 indexed citations
3.
4.
Song, Jin‐Chao, Xing Tan, Qing Yu, et al.. (2023). Tissue kallikrein-related peptidase8 accentuates cardiac fibrosis after myocardial ischemia-reperfusion injury via regulation of cardiac fibroblasts. Life Sciences. 329. 121973–121973. 5 indexed citations
5.
Song, Jin‐Chao, et al.. (2021). Dexmedetomidine protects the heart against ischemia reperfusion injury via regulation of the bradykinin receptors. European Journal of Pharmacology. 911. 174493–174493. 13 indexed citations
6.
Wang, Yifei, Shiyu Ma, Jing Shang, et al.. (2019). Tissue kallikrein-related peptidase8 protects rat heart against acute ischemia reperfusion injury. International Journal of Biological Macromolecules. 140. 1126–1133. 11 indexed citations
7.
Cong, Binhai, Xiaoyan Zhu, & Xin Ni. (2017). The roles of microRNA-22 in myocardial infarction.. PubMed. 69(5). 571–578. 5 indexed citations
8.
Cong, Binhai, Qing Yu, Long Wang, et al.. (2016). Upregulation of microRNA-22 contributes to myocardial ischemia-reperfusion injury by interfering with the mitochondrial function. Free Radical Biology and Medicine. 96. 406–417. 87 indexed citations
9.
Yu, Qing, Wei Zhao, Zhiping Tang, et al.. (2016). Kallikrein-related peptidase 8 is expressed in myocardium and induces cardiac hypertrophy. Scientific Reports. 6(1). 20024–20024. 17 indexed citations
10.
Tang, Zhiping, Yujun Wang, Xiaoyan Zhu, et al.. (2015). Reduced expression of CRHR2 and Sp-1 in myocardium of ovariectomized rats is improved by exercise training. Gynecological Endocrinology. 31(9). 742–746. 2 indexed citations
11.
Cong, Binhai, Long Wang, Xiaoyan Zhu, et al.. (2014). SGK1 Is Involved in Cardioprotection of Urocortin-1 Against Hypoxia/Reoxygenation in Cardiomyocytes. Canadian Journal of Cardiology. 30(6). 687–695. 15 indexed citations
12.
Zhu, Xiaoyan, Zhiping Tang, Binhai Cong, et al.. (2013). Estrogens increase cystathionine-γ-lyase expression and decrease inflammation and oxidative stress in the myocardium of ovariectomized rats. Menopause The Journal of The North American Menopause Society. 20(10). 1084–1091. 59 indexed citations
13.
Cong, Binhai, Yongjun Xu, Hui Sheng, et al.. (2013). Cardioprotection of 17β-estradiol against hypoxia/reoxygenation in cardiomyocytes is partly through up-regulation of CRH receptor type 2. Molecular and Cellular Endocrinology. 382(1). 17–25. 16 indexed citations
14.
Cong, Binhai, et al.. (2013). Estrogens protect myocardium against ischemia/reperfusion insult by up-regulation of CRH receptor type 2 in female rats. International Journal of Cardiology. 168(5). 4755–4760. 21 indexed citations
15.
Ma, Bei, Li-hua Yu, Binhai Cong, et al.. (2011). Estrogen modulation of peripheral pain signal transduction: involvement of P2X3 receptors. Purinergic Signalling. 7(1). 73–83. 40 indexed citations
16.
Cong, Binhai, et al.. (2009). Reduced expression of CRH receptor type 1 in upper segment human myometrium during labour. Reproductive Biology and Endocrinology. 7(1). 43–43. 22 indexed citations
17.
Gao, Lu, et al.. (2009). Expression of the calcium-activated potassium channel in upper and lower segment human myometrium during pregnancy and parturition. Reproductive Biology and Endocrinology. 7(1). 27–27. 28 indexed citations
18.
Sheng, Hui, Tingting Sun, Binhai Cong, et al.. (2008). Corticotropin-releasing hormone stimulates SGK-1 kinase expression in cultured hippocampal neurons via CRH-R1. American Journal of Physiology-Endocrinology and Metabolism. 295(4). E938–E946. 17 indexed citations
19.
Zhou, Mingyue, Binhai Cong, Ping He, et al.. (2008). Transient changes in P2X3 but not TRPV1 mRNA expression in rat after prenatal exposure to glucocorticoids. Autonomic Neuroscience. 141(1-2). 112–116. 6 indexed citations
20.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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