Bingxi Lei

889 total citations
28 papers, 602 citations indexed

About

Bingxi Lei is a scholar working on Molecular Biology, Cancer Research and Genetics. According to data from OpenAlex, Bingxi Lei has authored 28 papers receiving a total of 602 indexed citations (citations by other indexed papers that have themselves been cited), including 17 papers in Molecular Biology, 14 papers in Cancer Research and 7 papers in Genetics. Recurrent topics in Bingxi Lei's work include Cancer-related molecular mechanisms research (10 papers), MicroRNA in disease regulation (8 papers) and Glioma Diagnosis and Treatment (7 papers). Bingxi Lei is often cited by papers focused on Cancer-related molecular mechanisms research (10 papers), MicroRNA in disease regulation (8 papers) and Glioma Diagnosis and Treatment (7 papers). Bingxi Lei collaborates with scholars based in China, Macao and Canada. Bingxi Lei's co-authors include Songtao Qi, Yawei Liu, Hai Wang, Zhongjun Li, Jun Pan, Chao Li, Wei Xiang, Zhiwei Zhou, Ziyang Chen and Yuntao Lu and has published in prestigious journals such as Cell, Brain and Journal of Hazardous Materials.

In The Last Decade

Bingxi Lei

27 papers receiving 600 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Bingxi Lei China 13 405 283 128 70 65 28 602
Fangcheng Li China 12 397 1.0× 291 1.0× 76 0.6× 77 1.1× 51 0.8× 31 578
Nanxiang Xiong China 16 475 1.2× 326 1.2× 118 0.9× 117 1.7× 58 0.9× 65 887
Chunfa Qian China 15 365 0.9× 263 0.9× 94 0.7× 85 1.2× 38 0.6× 34 632
Shigang Lv China 15 396 1.0× 223 0.8× 83 0.6× 38 0.5× 57 0.9× 32 576
Bo Pang China 15 451 1.1× 256 0.9× 88 0.7× 93 1.3× 78 1.2× 22 641
Renjun Peng China 17 567 1.4× 478 1.7× 45 0.4× 63 0.9× 51 0.8× 39 804
Yutian Wei China 11 677 1.7× 566 2.0× 71 0.6× 56 0.8× 70 1.1× 19 839
Sekyung Oh South Korea 14 526 1.3× 154 0.5× 97 0.8× 59 0.8× 47 0.7× 31 733
Xinke Xu China 12 280 0.7× 227 0.8× 58 0.5× 58 0.8× 64 1.0× 23 430
Adriana Cruvinel Carloni Brazil 9 231 0.6× 101 0.4× 135 1.1× 101 1.4× 51 0.8× 14 392

Countries citing papers authored by Bingxi Lei

Since Specialization
Citations

This map shows the geographic impact of Bingxi Lei's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Bingxi Lei with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Bingxi Lei more than expected).

Fields of papers citing papers by Bingxi Lei

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Bingxi Lei. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Bingxi Lei. The network helps show where Bingxi Lei may publish in the future.

Co-authorship network of co-authors of Bingxi Lei

This figure shows the co-authorship network connecting the top 25 collaborators of Bingxi Lei. A scholar is included among the top collaborators of Bingxi Lei based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Bingxi Lei. Bingxi Lei is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Zhou, Xiangjun, Jun Li, Shizhen Qiu, et al.. (2025). Bclaf1 mediates super-enhancer-driven activation of POLR2A to enhance chromatin accessibility in nitrosamine-induced esophageal carcinogenesis. Journal of Hazardous Materials. 492. 138218–138218. 1 indexed citations
2.
Chen, Caiyun, et al.. (2024). Depletion of the N6-Methyladenosine (m6A) reader protein IGF2BP3 induces ferroptosis in glioma by modulating the expression of GPX4. Cell Death and Disease. 15(3). 181–181. 12 indexed citations
3.
Cheng, Xiaopeng, Bowen Xu, Bingxi Lei, & Shanfeng Wang. (2024). Opposite Mechanical Preference of Bone/Nerve Regeneration in 3D‐Printed Bioelastomeric Scaffolds/Conduits Consistently Correlated with YAP‐Mediated Stem Cell Osteo/Neuro‐Genesis. Advanced Healthcare Materials. 13(7). e2301158–e2301158. 9 indexed citations
4.
Li, Ling, et al.. (2023). Identification of a copper metabolism‐related gene signature for predicting prognosis and immune response in glioma. Cancer Medicine. 12(8). 10123–10137. 11 indexed citations
5.
Lei, Bingxi, Chao Yang, Marta Silva, et al.. (2023). Artemisia annua Extract Improves the Cognitive Deficits and Reverses the Pathological Changes of Alzheimer’s Disease via Regulating YAP Signaling. International Journal of Molecular Sciences. 24(6). 5259–5259. 10 indexed citations
6.
Di, Can, Shi‐Jian Song, Yubo Zhang, et al.. (2023). Choroid plexus mast cells drive tumor-associated hydrocephalus. Cell. 186(26). 5719–5738.e28. 21 indexed citations
7.
Que, Zhongyou, et al.. (2023). Dihydroartemisinin inhibits EMT of glioma via gene BASP1 in extrachromosomal DNA. Biochemical and Biophysical Research Communications. 675. 130–138. 5 indexed citations
8.
Li, Jiaqian, Di Huang, Bingxi Lei, et al.. (2022). VLA-4 suppression by senescence signals regulates meningeal immunity and leptomeningeal metastasis. eLife. 11. 7 indexed citations
9.
10.
Lei, Bingxi, et al.. (2021). Whole-exome sequencing identifies somatic mutations associated with lung cancer metastasis to the brain. Annals of Translational Medicine. 9(8). 694–694. 10 indexed citations
11.
Wang, Yuhao, Aijun Shan, Zhiwei Zhou, et al.. (2021). LncRNA TCONS_00004099-derived microRNA regulates oncogenesis through PTPRF in gliomas. Annals of Translational Medicine. 9(12). 1023–1023. 12 indexed citations
12.
13.
Yi, Guozhong, Guanglong Huang, Xian Zhang, et al.. (2019). Acquired temozolomide resistance in MGMT-deficient glioblastoma cells is associated with regulation of DNA repair by DHC2. Brain. 142(8). 2352–2366. 107 indexed citations
14.
Lei, Bingxi, et al.. (2018). Circular RNA hsa_circ_0076248 promotes oncogenesis of glioma by sponging miR‐181a to modulate SIRT1 expression. Journal of Cellular Biochemistry. 120(4). 6698–6708. 61 indexed citations
15.
Lei, Bingxi, et al.. (2017). Prognostic factors in patients treated with surgery for brain metastases: A single-center retrospective analysis of 125 patients. International Journal of Surgery. 44. 204–209. 7 indexed citations
16.
Deng, Shengze, Yaomin Li, Guozhong Yi, et al.. (2017). Overexpression of COX7A2 is associated with a good prognosis in patients with glioma. Journal of Neuro-Oncology. 136(1). 41–50. 14 indexed citations
17.
Wang, Hai, Yuntao Lu, Hezhen Li, et al.. (2016). Expression of dynein, cytoplasmic 2, heavy chain 1 (DHC2) associated with glioblastoma cell resistance to temozolomide. Scientific Reports. 6(1). 28948–28948. 17 indexed citations
18.
Lei, Bingxi, et al.. (2016). A simple classification of cranial–nasal–orbital communicating tumors that facilitate choice of surgical approaches: analysis of a series of 32 cases. European Archives of Oto-Rhino-Laryngology. 273(8). 2239–2248. 3 indexed citations
19.
Li, Chao, et al.. (2015). H19 derived microRNA-675 regulates cell proliferation and migration through CDK6 in glioma.. PubMed. 7(10). 1747–64. 42 indexed citations
20.
Wang, Hai, Xiang Peng, Yawei Liu, et al.. (2014). Inhibition of EZH2 reverses chemotherapeutic drug TMZ chemosensitivity in glioblastoma.. PubMed Central. 7(10). 6662–70. 62 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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