Binbin Ni

566 total citations
9 papers, 450 citations indexed

About

Binbin Ni is a scholar working on Epidemiology, Molecular Biology and Surgery. According to data from OpenAlex, Binbin Ni has authored 9 papers receiving a total of 450 indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Epidemiology, 5 papers in Molecular Biology and 4 papers in Surgery. Recurrent topics in Binbin Ni's work include Autophagy in Disease and Therapy (3 papers), Bioactive Natural Diterpenoids Research (3 papers) and Orthopaedic implants and arthroplasty (2 papers). Binbin Ni is often cited by papers focused on Autophagy in Disease and Therapy (3 papers), Bioactive Natural Diterpenoids Research (3 papers) and Orthopaedic implants and arthroplasty (2 papers). Binbin Ni collaborates with scholars based in China. Binbin Ni's co-authors include Lei‐Sheng Jiang, Sheng‐Dan Jiang, Yuehua Yang, Jiangwei Chen, Bo Li, Bo Li, Xin‐Feng Zheng, Yuan Sun, Haonan Zhang and Weiwei Mao and has published in prestigious journals such as Free Radical Biology and Medicine, The International Journal of Biochemistry & Cell Biology and Cytokine.

In The Last Decade

Binbin Ni

9 papers receiving 448 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Binbin Ni China 8 199 146 121 113 95 9 450
Chenyu Wu China 13 110 0.6× 250 1.7× 55 0.5× 80 0.7× 55 0.6× 32 473
Wen Geng China 7 237 1.2× 144 1.0× 56 0.5× 36 0.3× 141 1.5× 14 460
Huipeng Yin China 11 330 1.7× 223 1.5× 108 0.9× 43 0.4× 170 1.8× 20 590
Xunlin Li China 8 130 0.7× 96 0.7× 69 0.6× 28 0.2× 98 1.0× 12 309
Zhonghui Chen China 17 181 0.9× 174 1.2× 267 2.2× 30 0.3× 70 0.7× 38 594
Jialin Jiang China 7 105 0.5× 82 0.6× 46 0.4× 50 0.4× 55 0.6× 19 273
Yueyang Li China 12 110 0.6× 198 1.4× 54 0.4× 49 0.4× 47 0.5× 38 497

Countries citing papers authored by Binbin Ni

Since Specialization
Citations

This map shows the geographic impact of Binbin Ni's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Binbin Ni with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Binbin Ni more than expected).

Fields of papers citing papers by Binbin Ni

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Binbin Ni. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Binbin Ni. The network helps show where Binbin Ni may publish in the future.

Co-authorship network of co-authors of Binbin Ni

This figure shows the co-authorship network connecting the top 25 collaborators of Binbin Ni. A scholar is included among the top collaborators of Binbin Ni based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Binbin Ni. Binbin Ni is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

9 of 9 papers shown
1.
Yang, Yuehua, Yuan Sun, Weiwei Mao, et al.. (2021). Oxidative stress induces downregulation of TP53INP2 and suppresses osteogenic differentiation of BMSCs during osteoporosis through the autophagy degradation pathway. Free Radical Biology and Medicine. 166. 226–237. 55 indexed citations
2.
Ni, Binbin, et al.. (2020). Use of a locking intramedullary nail for the treatment of initial varus proximal humeral fracture: a prospective pilot study. Journal of International Medical Research. 48(6). 1220734838–1220734838. 5 indexed citations
3.
Ni, Binbin, Hao Shen, Wei Wang, Hua Lu, & Lei‐Sheng Jiang. (2019). TGF-β1 reduces the oxidative stress-induced autophagy and apoptosis in rat annulus fibrosus cells through the ERK signaling pathway. Journal of Orthopaedic Surgery and Research. 14(1). 241–241. 25 indexed citations
4.
Chen, Pengbo, et al.. (2017). Newly designed anterolateral and posterolateral locking anatomic plates for lateral tibial plateau fractures: a finite element study. Journal of Orthopaedic Surgery and Research. 12(1). 35–35. 22 indexed citations
5.
Chen, Pengbo, et al.. (2016). The morphological features of different Schatzker types of tibial plateau fractures: a three-dimensional computed tomography study. Journal of Orthopaedic Surgery and Research. 11(1). 94–94. 25 indexed citations
6.
7.
Chen, Jiangwei, Binbin Ni, Bo Li, et al.. (2014). The Responses of Autophagy and Apoptosis to Oxidative Stress in Nucleus Pulposus Cells: Implications for Disc Degeneration. Cellular Physiology and Biochemistry. 34(4). 1175–1189. 164 indexed citations
8.
Chen, Jiangwei, Binbin Ni, Xin‐Feng Zheng, et al.. (2014). Hypoxia facilitates the survival of nucleus pulposus cells in serum deprivation by down-regulating excessive autophagy through restricting ROS generation. The International Journal of Biochemistry & Cell Biology. 59. 1–10. 74 indexed citations
9.
Li, Bo, Yuehua Yang, Sheng‐Dan Jiang, et al.. (2012). Adenovirus-mediated overexpression of BMP-9 inhibits human osteosarcoma cell growth and migration through downregulation of the PI3K/AKT pathway. International Journal of Oncology. 41(5). 1809–1819. 29 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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