Beyza Vurusaner

496 total citations
8 papers, 417 citations indexed

About

Beyza Vurusaner is a scholar working on Surgery, Molecular Biology and Immunology. According to data from OpenAlex, Beyza Vurusaner has authored 8 papers receiving a total of 417 indexed citations (citations by other indexed papers that have themselves been cited), including 5 papers in Surgery, 5 papers in Molecular Biology and 3 papers in Immunology. Recurrent topics in Beyza Vurusaner's work include Cholesterol and Lipid Metabolism (5 papers), Receptor Mechanisms and Signaling (2 papers) and Sphingolipid Metabolism and Signaling (2 papers). Beyza Vurusaner is often cited by papers focused on Cholesterol and Lipid Metabolism (5 papers), Receptor Mechanisms and Signaling (2 papers) and Sphingolipid Metabolism and Signaling (2 papers). Beyza Vurusaner collaborates with scholars based in Italy, Türkiye and United States. Beyza Vurusaner's co-authors include Giuseppe Poli, Hüveyda Başağa, Paola Gamba, Gabriella Leonarduzzi, Gabriella Testa, Simona Gargiulo, Edward A. Fisher, Lara K. Mahal, Shiyu Chen and James W. Canary and has published in prestigious journals such as Analytical Chemistry, Scientific Reports and Free Radical Biology and Medicine.

In The Last Decade

Beyza Vurusaner

8 papers receiving 414 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Beyza Vurusaner Italy 7 231 91 83 64 52 8 417
Ashok Reddy Dinasarapu United States 10 240 1.0× 31 0.3× 70 0.8× 53 0.8× 29 0.6× 26 447
Yanbin Ma China 15 396 1.7× 104 1.1× 86 1.0× 59 0.9× 68 1.3× 26 702
Lisa A. Vettore United Kingdom 6 342 1.5× 44 0.5× 246 3.0× 42 0.7× 32 0.6× 9 548
Ninder Panesar United States 13 167 0.7× 73 0.8× 41 0.5× 85 1.3× 29 0.6× 23 469
Berta Schulz Chile 7 264 1.1× 77 0.8× 56 0.7× 23 0.4× 63 1.2× 8 479
T. I. Torkhovskaya Russia 10 198 0.9× 50 0.5× 24 0.3× 35 0.5× 46 0.9× 53 388
Michel Nauze France 13 247 1.1× 167 1.8× 31 0.4× 32 0.5× 50 1.0× 18 482
Heinric Williams United States 10 284 1.2× 109 1.2× 63 0.8× 24 0.4× 19 0.4× 25 492
Guennadi A. Khoudoli United Kingdom 11 688 3.0× 75 0.8× 76 0.9× 35 0.5× 108 2.1× 12 810
Lixia Pu United States 9 400 1.7× 77 0.8× 40 0.5× 31 0.5× 54 1.0× 15 506

Countries citing papers authored by Beyza Vurusaner

Since Specialization
Citations

This map shows the geographic impact of Beyza Vurusaner's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Beyza Vurusaner with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Beyza Vurusaner more than expected).

Fields of papers citing papers by Beyza Vurusaner

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Beyza Vurusaner. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Beyza Vurusaner. The network helps show where Beyza Vurusaner may publish in the future.

Co-authorship network of co-authors of Beyza Vurusaner

This figure shows the co-authorship network connecting the top 25 collaborators of Beyza Vurusaner. A scholar is included among the top collaborators of Beyza Vurusaner based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Beyza Vurusaner. Beyza Vurusaner is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

8 of 8 papers shown
1.
Vurusaner, Beyza, et al.. (2022). Loss of PRMT2 in myeloid cells in normoglycemic mice phenocopies impaired regression of atherosclerosis in diabetic mice. Scientific Reports. 12(1). 12031–12031. 8 indexed citations
2.
Chen, Shiyu, et al.. (2021). Two-Photon, Ratiometric, Quantitative Fluorescent Probe Reveals Fluctuation of Peroxynitrite Regulated by Arginase 1. Analytical Chemistry. 93(29). 10090–10098. 45 indexed citations
3.
Vurusaner, Beyza, Simona Gargiulo, Gabriella Testa, et al.. (2018). The role of autophagy in survival response induced by 27-hydroxycholesterol in human promonocytic cells. Redox Biology. 17. 400–410. 26 indexed citations
4.
Vurusaner, Beyza, Gabriella Leonarduzzi, Paola Gamba, Giuseppe Poli, & Hüveyda Başağa. (2016). Oxysterols and mechanisms of survival signaling. Molecular Aspects of Medicine. 49. 8–22. 34 indexed citations
5.
Vurusaner, Beyza, Paola Gamba, Gabriella Testa, et al.. (2016). Molecular Mechanism of Oxysterol Induced Survival Response: The Role of Autophagy and ROS. Free Radical Biology and Medicine. 100. S46–S46. 1 indexed citations
6.
Vurusaner, Beyza, Paola Gamba, Simona Gargiulo, et al.. (2015). Nrf2 antioxidant defense is involved in survival signaling elicited by 27-hydroxycholesterol in human promonocytic cells. Free Radical Biology and Medicine. 91. 93–104. 24 indexed citations
7.
Vurusaner, Beyza, Paola Gamba, Gabriella Testa, et al.. (2014). Survival signaling elicited by 27-hydroxycholesterol through the combined modulation of cellular redox state and ERK/Akt phosphorylation. Free Radical Biology and Medicine. 77. 376–385. 38 indexed citations
8.
Vurusaner, Beyza, Giuseppe Poli, & Hüveyda Başağa. (2011). Tumor suppressor genes and ROS: complex networks of interactions. Free Radical Biology and Medicine. 52(1). 7–18. 241 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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