Beth S. Lee

1.6k total citations
47 papers, 1.3k citations indexed

About

Beth S. Lee is a scholar working on Molecular Biology, Oncology and Cell Biology. According to data from OpenAlex, Beth S. Lee has authored 47 papers receiving a total of 1.3k indexed citations (citations by other indexed papers that have themselves been cited), including 43 papers in Molecular Biology, 11 papers in Oncology and 7 papers in Cell Biology. Recurrent topics in Beth S. Lee's work include ATP Synthase and ATPases Research (16 papers), Bone Metabolism and Diseases (16 papers) and Bone health and treatments (11 papers). Beth S. Lee is often cited by papers focused on ATP Synthase and ATPases Research (16 papers), Bone Metabolism and Diseases (16 papers) and Bone health and treatments (11 papers). Beth S. Lee collaborates with scholars based in United States, Germany and France. Beth S. Lee's co-authors include Stephen L. Gluck, L. Shannon Holliday, Ming Lu, Raoul D. Nelson, Li Zhang, Irina Krits, Selvi C. Jeyaraj, Richard E. Cheney, Mamata Singh and Duaa Dakhlallah and has published in prestigious journals such as Journal of Biological Chemistry, Blood and PLoS ONE.

In The Last Decade

Beth S. Lee

44 papers receiving 1.3k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Beth S. Lee United States 24 1.0k 211 204 131 109 47 1.3k
Masaki Yoda Japan 20 516 0.5× 137 0.6× 191 0.9× 153 1.2× 88 0.8× 44 1.0k
Adi D. Dubash United States 17 759 0.7× 466 2.2× 134 0.7× 80 0.6× 76 0.7× 22 1.2k
Venkaiah Betapudi United States 15 622 0.6× 368 1.7× 104 0.5× 105 0.8× 76 0.7× 21 1.2k
Agnieszka Strzelecka‐Kiliszek Poland 20 539 0.5× 104 0.5× 71 0.3× 179 1.4× 124 1.1× 45 1.0k
Marc Thiry Belgium 17 707 0.7× 98 0.5× 212 1.0× 74 0.6× 173 1.6× 32 1.2k
Renato Bareggi Italy 22 929 0.9× 261 1.2× 131 0.6× 133 1.0× 91 0.8× 80 1.4k
Johbu Itoh Japan 25 749 0.7× 202 1.0× 301 1.5× 159 1.2× 134 1.2× 92 1.6k
S. Park-Snyder United States 10 492 0.5× 117 0.6× 82 0.4× 167 1.3× 49 0.4× 10 784
Rainer Will Germany 19 763 0.7× 114 0.5× 202 1.0× 140 1.1× 311 2.9× 50 1.3k

Countries citing papers authored by Beth S. Lee

Since Specialization
Citations

This map shows the geographic impact of Beth S. Lee's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Beth S. Lee with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Beth S. Lee more than expected).

Fields of papers citing papers by Beth S. Lee

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Beth S. Lee. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Beth S. Lee. The network helps show where Beth S. Lee may publish in the future.

Co-authorship network of co-authors of Beth S. Lee

This figure shows the co-authorship network connecting the top 25 collaborators of Beth S. Lee. A scholar is included among the top collaborators of Beth S. Lee based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Beth S. Lee. Beth S. Lee is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Clements, Richard, Radmila Terentyeva, Shanna Hamilton, et al.. (2023). Sexual dimorphism in bidirectional SR-mitochondria crosstalk in ventricular cardiomyocytes. Basic Research in Cardiology. 118(1). 15–15. 12 indexed citations
2.
Liu, Jie, Keiichiro Watanabe, Shareef M. Dabdoub, Beth S. Lee, & Do‐Gyoon Kim. (2022). Site-specific characteristics of bone and progenitor cells in control and ovariectomized rats. Bone. 163. 116501–116501. 3 indexed citations
3.
Bhatti, Fazal‐Ur‐Rehman, Anastasios Karydis, Beth S. Lee, et al.. (2021). Understanding Early-Stage Posttraumatic Osteoarthritis for Future Prospects of Diagnosis: from Knee to Temporomandibular Joint. Current Osteoporosis Reports. 19(2). 166–174. 7 indexed citations
4.
Watanabe, Keiichiro, Xiaohan Guo, Ai Ni, et al.. (2020). Regional variations of jaw bone characteristics in an ovariectomized rat model. Journal of the mechanical behavior of biomedical materials. 110. 103952–103952. 9 indexed citations
5.
Knapik, Derrick M., P. Perera, Jin Nam, et al.. (2014). Mechanosignaling in bone health, trauma and inflammation. RWTH Publications (RWTH Aachen). 1 indexed citations
6.
Collaco, Anne, et al.. (2013). Functional vacuolar ATPase (V-ATPase) proton pumps traffic to the enterocyte brush border membrane and require CFTR. American Journal of Physiology-Cell Physiology. 305(9). C981–C996. 17 indexed citations
7.
Knapik, Derrick M., P. Perera, Jin Nam, et al.. (2013). Mechanosignaling in Bone Health, Trauma and Inflammation. Antioxidants and Redox Signaling. 20(6). 970–985. 43 indexed citations
8.
Lee, Beth S., et al.. (2013). Adaptive and maladaptive expression of the Mrna regulatory protein HuR. World Journal of Biological Chemistry. 4(4). 111–111. 34 indexed citations
9.
Singh, Mamata, et al.. (2012). HuR inhibits apoptosis by amplifying Akt signaling through a positive feedback loop. Journal of Cellular Physiology. 228(1). 182–189. 18 indexed citations
10.
Lu, Jingwei, Suman Kanji, Matthew Joseph, et al.. (2012). Human Umbilical Cord Blood-Derived CD34+ Cells Reverse Osteoporosis in NOD/SCID Mice by Altering Osteoblastic and Osteoclastic Activities. PLoS ONE. 7(6). e39365–e39365. 32 indexed citations
11.
Meyer, Stephanie, et al.. (2010). c-Src-mediated Phosphorylation of Thyroid Hormone Receptor-interacting Protein 6 (TRIP6) Promotes Osteoclast Sealing Zone Formation. Journal of Biological Chemistry. 285(34). 26641–26651. 26 indexed citations
12.
Wysolmerski, Robert B., et al.. (2009). Regulated Proteolysis of Nonmuscle Myosin IIA Stimulates Osteoclast Fusion. Journal of Biological Chemistry. 284(18). 12266–12275. 30 indexed citations
13.
Jeyaraj, Selvi C., et al.. (2009). Transcriptional Control of Human Antigen R by Bone Morphogenetic Protein. Journal of Biological Chemistry. 285(7). 4432–4440. 23 indexed citations
14.
Ayupova, Dina, Mamata Singh, Ellen C. Leonard, David P. Basile, & Beth S. Lee. (2009). Expression of the RNA-stabilizing protein HuR in ischemia-reperfusion injury of rat kidney. American Journal of Physiology-Renal Physiology. 297(1). F95–F105. 30 indexed citations
15.
Lee, Beth S., et al.. (2008). High molecular weight tropomyosins regulate osteoclast cytoskeletal morphology. Bone. 43(5). 951–960. 15 indexed citations
16.
Holliday, L. Shannon, et al.. (2006). Tropomyosin isoforms localize to distinct microfilament populations in osteoclasts. Bone. 39(4). 694–705. 54 indexed citations
17.
Jeyaraj, Selvi C., et al.. (2005). HuR Stabilizes Vacuolar H+-translocating ATPase mRNA during Cellular Energy Depletion. Journal of Biological Chemistry. 280(45). 37957–37964. 27 indexed citations
18.
Holliday, L. Shannon, Ming Lu, Beth S. Lee, et al.. (2000). The Amino-terminal Domain of the B Subunit of Vacuolar H+-ATPase Contains a Filamentous Actin Binding Site. Journal of Biological Chemistry. 275(41). 32331–32337. 153 indexed citations
19.
Gluck, Stephen L., et al.. (1996). Distal urinary acidification from Homer Smith to the present. Kidney International. 49(6). 1660–1664. 11 indexed citations
20.
Lee, Beth S., et al.. (1995). Transcriptional Regulation of the Vacuolar H+-ATPase B2 Subunit Gene in Differentiating THP-1 Cells. Journal of Biological Chemistry. 270(13). 7320–7329. 40 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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