Benny J. Evison

826 total citations
17 papers, 663 citations indexed

About

Benny J. Evison is a scholar working on Molecular Biology, Oncology and Organic Chemistry. According to data from OpenAlex, Benny J. Evison has authored 17 papers receiving a total of 663 indexed citations (citations by other indexed papers that have themselves been cited), including 15 papers in Molecular Biology, 5 papers in Oncology and 4 papers in Organic Chemistry. Recurrent topics in Benny J. Evison's work include Cancer therapeutics and mechanisms (10 papers), DNA and Nucleic Acid Chemistry (4 papers) and DNA Repair Mechanisms (4 papers). Benny J. Evison is often cited by papers focused on Cancer therapeutics and mechanisms (10 papers), DNA and Nucleic Acid Chemistry (4 papers) and DNA Repair Mechanisms (4 papers). Benny J. Evison collaborates with scholars based in Australia, United States and Israel. Benny J. Evison's co-authors include Suzanne M. Cutts, Don R. Phillips, Mas Jaffri Masarudin, Paul J. Pigram, Brad E. Sleebs, Keith G. Watson, Francis C. K. Chiu, Gabriella Pezzoni, Rebecca A. Bilardi and E. MENTA and has published in prestigious journals such as Nucleic Acids Research, Journal of Medicinal Chemistry and Journal of Lipid Research.

In The Last Decade

Benny J. Evison

17 papers receiving 658 citations

Peers

Countries citing papers authored by Benny J. Evison

Since Specialization

Citations

This map shows the geographic impact of Benny J. Evison's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Benny J. Evison with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Benny J. Evison more than expected).

Fields of papers citing papers by Benny J. Evison

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Benny J. Evison. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Benny J. Evison. The network helps show where Benny J. Evison may publish in the future.

Co-authorship network of co-authors of Benny J. Evison

This figure shows the co-authorship network connecting the top 25 collaborators of Benny J. Evison. A scholar is included among the top collaborators of Benny J. Evison based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Benny J. Evison. Benny J. Evison is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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17 of 17 papers shown

#	Title	Journal	Authors	Indexed citations
1	PCSK9 activation promotes early atherosclerosis in a vascular microphysiological system	APL Bioengineering	Joung-Hyun Lee, Kevin L. Shores et al.	7
2	An evaluation of the interaction of pixantrone with formaldehyde-releasing drugs in cancer cells	Cancer Chemotherapy and Pharmacology	Abraham Nudelman, Ada Rephaeli et al.	2
3	A Novel, Orally Bioavailable, Small-Molecule Inhibitor of PCSK9 With Significant Cholesterol-Lowering Properties In Vivo	Journal of Lipid Research	Alexandra K. Suchowerska, Geurt Stokman et al.	15
4	Development of an automated assay for accelerated in vitro detection of DNA adduct-inducing and crosslinking agents	Bioorganic & Medicinal Chemistry Letters	Brad E. Sleebs, Kurt Lackovic et al.	2
5	A small molecule inhibitor of PCSK9 that antagonizes LDL receptor binding via interaction with a cryptic PCSK9 binding groove	Bioorganic & Medicinal Chemistry	Benny J. Evison, James T. Palmer et al.	36
6	Formaldehyde-activated WEHI-150 induces DNA interstrand crosslinks with unique structural features	Bioorganic & Medicinal Chemistry	Benny J. Evison, J. Grant Collins et al.	4
7	Factors determining the stability, size distribution, and cellular accumulation of small, monodisperse chitosan nanoparticles as candidate vectors for anticancer drug delivery: application to the passive encapsulation of [14C]-doxorubicin	PubMed	Mas Jaffri Masarudin, Suzanne M. Cutts et al.	277
8	Mitoxantrone, More than Just Another Topoisomerase II Poison	Medicinal Research Reviews	Benny J. Evison, Brad E. Sleebs et al.	182
9	Design, synthesis, and DNA sequence selectivity of formaldehyde-mediated DNA-adducts of the novel N-(4-aminobutyl) acridine-4-carboxamide	Bioorganic & Medicinal Chemistry Letters	Benny J. Evison, Don R. Phillips et al.	2
10	The hydroxyl epimer of doxorubicin controls the rate of formation of cytotoxic anthracycline-DNA adducts	Cancer Chemotherapy and Pharmacology	Lonnie P. Swift, Benny J. Evison et al.	8
11	M2, a novel anthracenedione, elicits a potent DNA damage response that can be subverted through checkpoint kinase inhibition to generate mitotic catastrophe	Biochemical Pharmacology	Benny J. Evison, Rebecca A. Bilardi et al.	8
12	DNA binding by pixantrone	Organic & Biomolecular Chemistry	Damian P. Buck, Benny J. Evison et al.	12
13	New Anthracenedione Derivatives with Improved Biological Activity by Virtue of Stable Drug−DNA Adduct Formation	Journal of Medicinal Chemistry	Benny J. Evison, Brad E. Sleebs et al.	19
14	CpG methylation potentiates pixantrone and doxorubicin-induced DNA damage and is a marker of drug sensitivity	Nucleic Acids Research	Benny J. Evison, Rebecca A. Bilardi et al.	29
15	In vitro Transcription Assay for Resolution of Drug-DNA Interactions at Defined DNA Sequences	Methods in molecular biology	Benny J. Evison, Don R. Phillips et al.	2
16	Formaldehyde-Activated Pixantrone Is a Monofunctional DNA Alkylator That Binds Selectively to CpG and CpA Doublets	Molecular Pharmacology	Benny J. Evison, Francis C. K. Chiu et al.	22
17	Pixantrone can be activated by formaldehyde to generate a potent DNA adduct forming agent	Nucleic Acids Research	Benny J. Evison, E. MENTA et al.	36

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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