Benjamin Primack

1.3k total citations · 1 hit paper
10 papers, 748 citations indexed

About

Benjamin Primack is a scholar working on Oncology, Molecular Biology and Immunology. According to data from OpenAlex, Benjamin Primack has authored 10 papers receiving a total of 748 indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Oncology, 4 papers in Molecular Biology and 3 papers in Immunology. Recurrent topics in Benjamin Primack's work include DNA Repair Mechanisms (3 papers), CAR-T cell therapy research (3 papers) and Advanced Breast Cancer Therapies (2 papers). Benjamin Primack is often cited by papers focused on DNA Repair Mechanisms (3 papers), CAR-T cell therapy research (3 papers) and Advanced Breast Cancer Therapies (2 papers). Benjamin Primack collaborates with scholars based in United States, China and United Kingdom. Benjamin Primack's co-authors include Alan D. D’Andrea, Ravindra Amunugama, Simon J. Boulton, Mark I.R. Petalcorin, Timur Yusufzai, Raphaël Ceccaldi, Panagiotis A. Konstantinopoulos, Stephen J. Elledge, Jessie Liu and Ildikó Hajdú and has published in prestigious journals such as Nature, Molecular Cell and Molecular and Cellular Biology.

In The Last Decade

Benjamin Primack

10 papers receiving 744 citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Homologous-recombination-deficient tumours are dependent on Polθ-mediated repair

2015 648 citations Raphaël Ceccaldi, Jessie Liu et al. Nature profile →

Peers

Benjamin Primack

MB

ONCOL

GENET

CR

PS

Niraj Joshi Canada

Ewa Gogola Netherlands

Giuseppe Leuzzi United States

Nidhi Nair Denmark

Arne Nedergaard Kousholt Denmark

Hatice Yücel Netherlands

Kamakoti P. Bhat United States

Amanda Day United States

Connor S. Clairmont United States

Jordan R. Becker United States

Niraj Joshi Canada

Benjamin Primack

621 ×0.9

MB

271 ×0.7

ONCOL

123 ×1.3

GENET

100 ×1.0

CR

46 ×0.8

PS

Citations per year, relative to Benjamin Primack Benjamin Primack (= 1×) peers Niraj Joshi

Countries citing papers authored by Benjamin Primack

Since Specialization

Citations

This map shows the geographic impact of Benjamin Primack's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Benjamin Primack with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Benjamin Primack more than expected).

Fields of papers citing papers by Benjamin Primack

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Benjamin Primack. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Benjamin Primack. The network helps show where Benjamin Primack may publish in the future.

Co-authorship network of co-authors of Benjamin Primack

This figure shows the co-authorship network connecting the top 25 collaborators of Benjamin Primack. A scholar is included among the top collaborators of Benjamin Primack based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Benjamin Primack. Benjamin Primack is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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10 of 10 papers shown

1.

Inniss, Mara C., Sean G. Smith, Benjamin Primack, et al.. (2025). Carbonic anhydrase 2-derived drug-responsive domain regulates membrane-bound cytokine expression and function in engineered T cells. Communications Biology. 8(1). 28–28. 2 indexed citations

2.

Burga, Rachel A., Bülent Arman Aksoy, Zheng Ao, et al.. (2025). IL-2-independent expansion, persistence, and antitumor activity in TIL expressing regulatable membrane-bound IL-15. Molecular Therapy. 33(8). 3605–3623. 4 indexed citations

3.

Koscsó, Balázs, Zheng Ao, Carmela Passaro, et al.. (2024). Abstract LB065: Tumor-infiltrating lymphocytes (TIL) engineered with regulatable membrane-bound IL15 (mbIL15) and LIGHT (TNFSF14) show enhanced efficacy in fibroblast-containing cold tumors. Cancer Research. 84(7_Supplement). LB065–LB065. 2 indexed citations

4.

Pedro, Kyle D., Rachel A. Burga, Gauri Kulkarni, et al.. (2023). Abstract LB096: IL15-engineered tumor infiltrating lymphocytes (cytoTIL15TM) exhibit activity against autologous tumor cells from multiple solid tumor indications without IL2. Cancer Research. 83(8_Supplement). LB096–LB096. 4 indexed citations

5.

Tchaicha, Jeremy H., Rachel A. Burga, Benjamin Primack, et al.. (2022). Abstract LB212: Allogeneic, IL-2-independent tumor-infiltrating lymphocytes expressing membrane-bound IL-15 (cytoTIL15) eradicate tumors in a melanoma PDX model through recognition of shared tumor antigens. Cancer Research. 82(12_Supplement). LB212–LB212. 1 indexed citations

6.

Ceccaldi, Raphaël, Jessie Liu, Ravindra Amunugama, et al.. (2015). Homologous-recombination-deficient tumours are dependent on Polθ-mediated repair. Nature. 518(7538). 258–262. 648 indexed citations breakdown →

7.

Johnson, Sara E., Neil F. Johnson, Benjamin Primack, et al.. (2014). 238 Reversal of primary and acquired PARP-inhibitor resistance in BRCA-mutated triple-negative breast cancers by inhibition of transcriptional cyclin-dependent kinases (CDKs). European Journal of Cancer. 50. 80–80. 1 indexed citations

8.

Johnson, Shawn F., Neil Johnson, David Chi, et al.. (2013). Abstract 1788: The CDK inhibitor dinaciclib sensitizes triple-negative breast cancer cells to PARP inhibition.. Cancer Research. 73(8_Supplement). 1788–1788. 2 indexed citations

9.

Park, Eunmi, Hyungjin Kim, Benjamin Primack, et al.. (2013). FANCD2 Activates Transcription of TAp63 and Suppresses Tumorigenesis. Molecular Cell. 50(6). 908–918. 51 indexed citations

10.

Park, Eunmi, Jeong‐Ki Min, Benjamin Primack, et al.. (2013). Inactivation of Uaf1 Causes Defective Homologous Recombination and Early Embryonic Lethality in Mice. Molecular and Cellular Biology. 33(22). 4360–4370. 33 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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