Benjamin M. Matta

1.9k total citations
21 papers, 1.4k citations indexed

About

Benjamin M. Matta is a scholar working on Immunology, Molecular Biology and Pulmonary and Respiratory Medicine. According to data from OpenAlex, Benjamin M. Matta has authored 21 papers receiving a total of 1.4k indexed citations (citations by other indexed papers that have themselves been cited), including 19 papers in Immunology, 3 papers in Molecular Biology and 2 papers in Pulmonary and Respiratory Medicine. Recurrent topics in Benjamin M. Matta's work include Immune Cell Function and Interaction (11 papers), Immunotherapy and Immune Responses (9 papers) and Immune Response and Inflammation (6 papers). Benjamin M. Matta is often cited by papers focused on Immune Cell Function and Interaction (11 papers), Immunotherapy and Immune Responses (9 papers) and Immune Response and Inflammation (6 papers). Benjamin M. Matta collaborates with scholars based in United States, China and Germany. Benjamin M. Matta's co-authors include Angus W. Thomson, Juan B. Ochoa, Vishal Bansal, Hēth Turnquist, Lori Perez, Brian Rosborough, A. Castellaneta, Dietmar Schwarz, Bruce A. McPheron and Bruce R. Blazar and has published in prestigious journals such as Nature, Blood and The Journal of Immunology.

In The Last Decade

Benjamin M. Matta

21 papers receiving 1.4k citations

Peers

Benjamin M. Matta

IMMUN

SURGE

ONCOL

GENET

Sabine Urban Germany

Kazumi Umeki Japan

Laurent Boucontet France

M Takada Japan

Jörg Klug Germany

Nasima Muqim United States

Márcia Nascimento United States

John J. Thompson United States

David L. Lamar United States

A Konno Japan

Sabine Urban Germany

Benjamin M. Matta

312 ×0.3

IMMUN

327 ×1.6

SURGE

93 ×0.5

ONCOL

101 ×0.7

GENET

405 ×3.1

Citations per year, relative to Benjamin M. Matta Benjamin M. Matta (= 1×) peers Sabine Urban

Countries citing papers authored by Benjamin M. Matta

Since Specialization

Citations

This map shows the geographic impact of Benjamin M. Matta's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Benjamin M. Matta with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Benjamin M. Matta more than expected).

Fields of papers citing papers by Benjamin M. Matta

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Benjamin M. Matta. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Benjamin M. Matta. The network helps show where Benjamin M. Matta may publish in the future.

Co-authorship network of co-authors of Benjamin M. Matta

This figure shows the co-authorship network connecting the top 25 collaborators of Benjamin M. Matta. A scholar is included among the top collaborators of Benjamin M. Matta based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Benjamin M. Matta. Benjamin M. Matta is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Matta, Benjamin M., Dawn K. Reichenbach, Xiaoli Zhang, et al.. (2016). Peri-alloHCT IL-33 administration expands recipient T-regulatory cells that protect mice against acute GVHD. Blood. 128(3). 427–439. 90 indexed citations

Matta, Benjamin M., Dawn K. Reichenbach, Bruce R. Blazar, & Hēth Turnquist. (2016). Alarmins and Their Receptors as Modulators and Indicators of Alloimmune Responses. American Journal of Transplantation. 17(2). 320–327. 22 indexed citations

Matta, Benjamin M. & Hēth Turnquist. (2015). Expansion of Regulatory T Cells In Vitro and In Vivo by IL-33. Methods in molecular biology. 1371. 29–41. 32 indexed citations

Lott, Jeremy M., Quan Liu, Benjamin M. Matta, et al.. (2015). The iron sequestering protein Lipocalin 2 is critical to IL-33-exposed dendritic cell stimulation of Th2 responses and allergic airway disease (IRC7P.424). The Journal of Immunology. 194(1_Supplement). 128.5–128.5. 1 indexed citations

Yoshida, O, Shoko Kimura, Lei Dou, et al.. (2014). DAP12 Deficiency in Liver Allografts Results in Enhanced Donor DC Migration, Augmented Effector T Cell Responses and Abrogation of Transplant Tolerance. American Journal of Transplantation. 14(8). 1791–1805. 22 indexed citations

Rosborough, Brian, Lisa Mathews, Benjamin M. Matta, et al.. (2014). Cutting Edge: Flt3 Ligand Mediates STAT3-Independent Expansion but STAT3-Dependent Activation of Myeloid-Derived Suppressor Cells. The Journal of Immunology. 192(8). 3470–3473. 19 indexed citations

Matta, Benjamin M., Jeremy M. Lott, Lisa Mathews, et al.. (2014). IL-33 Is an Unconventional Alarmin That Stimulates IL-2 Secretion by Dendritic Cells To Selectively Expand IL-33R/ST2+ Regulatory T Cells. The Journal of Immunology. 193(8). 4010–4020. 181 indexed citations

Matta, Benjamin M., Giorgio Raimondi, Brian Rosborough, Tina L. Sumpter, & Angus W. Thomson. (2012). IL-27 Production and STAT3-Dependent Upregulation of B7-H1 Mediate Immune Regulatory Functions of Liver Plasmacytoid Dendritic Cells. The Journal of Immunology. 188(11). 5227–5237. 87 indexed citations

Sumpter, Tina L., Anil Dangi, Benjamin M. Matta, et al.. (2012). Hepatic Stellate Cells Undermine the Allostimulatory Function of Liver Myeloid Dendritic Cells via STAT3-Dependent Induction of IDO. The Journal of Immunology. 189(8). 3848–3858. 57 indexed citations

10.

Matta, Benjamin M.. (2012). Plasmacytoid Dendritic Cells Are Key Regulators of Immune Tolerance. D-Scholarship@Pitt (University of Pittsburgh). 1 indexed citations

11.

Matta, Benjamin M., Tina L. Sumpter, Giorgio Raimondi, & Angus W. Thomson. (2010). Liver DC modulate Foxp3 expression in allogeneic CD4+ T cells via IL-27 (145.3). The Journal of Immunology. 184(Supplement_1). 145.3–145.3. 1 indexed citations

12.

Matta, Benjamin M., A. Castellaneta, & Angus W. Thomson. (2010). Tolerogenic plasmacytoid DC. European Journal of Immunology. 40(10). 2667–2676. 148 indexed citations

13.

Matta, Benjamin M., Tina L. Sumpter, Bridget L. Colvin, & Angus W. Thomson. (2009). Host-derived plasmacytoid dendritic cells prolong cardiac allograft survival and regulate alloreactive T cell responses via B7 homolog-1 (141.28). The Journal of Immunology. 182(Supplement_1). 141.28–141.28. 1 indexed citations

14.

Raimondi, Giorgio, Tina L. Sumpter, Benjamin M. Matta, et al.. (2009). Mammalian Target of Rapamycin Inhibition and Alloantigen-Specific Regulatory T Cells Synergize To Promote Long-Term Graft Survival in Immunocompetent Recipients. The Journal of Immunology. 184(2). 624–636. 87 indexed citations

15.

Popovič, P., et al.. (2009). Stat 6-Dependent Induction of Myeloid Derived Suppressor Cells After Physical Injury Regulates Nitric Oxide Response to Endotoxin. Annals of Surgery. 251(1). 120–126. 49 indexed citations

16.

Colvin, Bridget L., Benjamin M. Matta, & Angus W. Thomson. (2008). Dendritic Cells and Chemokine-Directed Migration in Transplantation: Where Are We Headed?. Clinics in Laboratory Medicine. 28(3). 375–384. 10 indexed citations

17.

Popovič, P., Benjamin M. Matta, José M. Prince, & Juan B. Ochoa. (2007). 64. Journal of Surgical Research. 137(2). 177–177. 1 indexed citations

18.

Bansal, Vishal, et al.. (2006). CD11b+/Gr-1+ Myeloid Suppressor Cells Cause T Cell Dysfunction after Traumatic Stress. The Journal of Immunology. 176(4). 2085–2094. 282 indexed citations

19.

Bansal, Vishal, et al.. (2005). Interactions Between Fatty Acids and Arginine Metabolism: Implications for the Design of Immune‐Enhancing Diets. Journal of Parenteral and Enteral Nutrition. 29(1S). S75–80. 53 indexed citations

20.

Schwarz, Dietmar, et al.. (2005). Host shift to an invasive plant triggers rapid animal hybrid speciation. Nature. 436(7050). 546–549. 196 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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