Benjamin Cieply

3.2k total citations
30 papers, 2.4k citations indexed

About

Benjamin Cieply is a scholar working on Molecular Biology, Oncology and Cancer Research. According to data from OpenAlex, Benjamin Cieply has authored 30 papers receiving a total of 2.4k indexed citations (citations by other indexed papers that have themselves been cited), including 24 papers in Molecular Biology, 6 papers in Oncology and 6 papers in Cancer Research. Recurrent topics in Benjamin Cieply's work include Wnt/β-catenin signaling in development and cancer (13 papers), RNA Research and Splicing (9 papers) and Cancer-related gene regulation (8 papers). Benjamin Cieply is often cited by papers focused on Wnt/β-catenin signaling in development and cancer (13 papers), RNA Research and Splicing (9 papers) and Cancer-related gene regulation (8 papers). Benjamin Cieply collaborates with scholars based in United States and China. Benjamin Cieply's co-authors include Satdarshan P. Monga, Steven M. Frisch, Udayan Apte, Russ P. Carstens, Xinping Tan, Michael D. Schaller, George K. Michalopoulos, Gang Zeng, Jaideep Behari and James Denvir and has published in prestigious journals such as Journal of Biological Chemistry, Gastroenterology and PLoS ONE.

In The Last Decade

Benjamin Cieply

28 papers receiving 2.4k citations

Peers

Benjamin Cieply
Aaron Bell United States
Mario Mikula Austria
Chunyue Yin United States
Polina Iakova United States
Radha P. Narsimhan United States
Efi E. Massasa United States
Susanne N. Weber Germany
Vincent W. Keng Hong Kong
Janel L. Kopp United States
Yasufumi Omori Japan
Aaron Bell United States
Benjamin Cieply
Citations per year, relative to Benjamin Cieply Benjamin Cieply (= 1×) peers Aaron Bell

Countries citing papers authored by Benjamin Cieply

Since Specialization
Citations

This map shows the geographic impact of Benjamin Cieply's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Benjamin Cieply with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Benjamin Cieply more than expected).

Fields of papers citing papers by Benjamin Cieply

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Benjamin Cieply. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Benjamin Cieply. The network helps show where Benjamin Cieply may publish in the future.

Co-authorship network of co-authors of Benjamin Cieply

This figure shows the co-authorship network connecting the top 25 collaborators of Benjamin Cieply. A scholar is included among the top collaborators of Benjamin Cieply based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Benjamin Cieply. Benjamin Cieply is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Cieply, Benjamin, et al.. (2018). Esrp1-Regulated Splicing of Arhgef11 Isoforms Is Required for Epithelial Tight Junction Integrity. Cell Reports. 25(9). 2417–2430.e5. 22 indexed citations
2.
Bebee, Thomas W., Caleb M. Radens, Chris McDermott‐Roe, et al.. (2017). ESRP1 Mutations Cause Hearing Loss due to Defects in Alternative Splicing that Disrupt Cochlear Development. Developmental Cell. 43(3). 318–331.e5. 41 indexed citations
3.
Cieply, Benjamin, Juw Won Park, Angela Nakauka‐Ddamba, et al.. (2016). Multiphasic and Dynamic Changes in Alternative Splicing during Induction of Pluripotency Are Coordinated by Numerous RNA-Binding Proteins. Cell Reports. 15(2). 247–255. 56 indexed citations
4.
Murphy, Daniel, Benjamin Cieply, Russ P. Carstens, Visvanathan Ramamurthy, & Peter Stoilov. (2016). The Musashi 1 Controls the Splicing of Photoreceptor-Specific Exons in the Vertebrate Retina. PLoS Genetics. 12(8). e1006256–e1006256. 52 indexed citations
5.
Cieply, Benjamin, et al.. (2015). CD44S-hyaluronan interactions protect cells resulting from EMT against anoikis. Matrix Biology. 48. 55–65. 36 indexed citations
6.
Bebee, Thomas W., Benjamin Cieply, & Russ P. Carstens. (2014). Genome-Wide Activities of RNA Binding Proteins That Regulate Cellular Changes in the Epithelial to Mesenchymal Transition (EMT). Advances in experimental medicine and biology. 825. 267–302. 19 indexed citations
7.
Cieply, Benjamin, Joshua C. Farris, James Denvir, Heide L. Ford, & Steven M. Frisch. (2013). Epithelial–Mesenchymal Transition and Tumor Suppression Are Controlled by a Reciprocal Feedback Loop between ZEB1 and Grainyhead-like-2. Cancer Research. 73(20). 6299–6309. 151 indexed citations
8.
Heinicke, Laurie A., Behnam Nabet, Shihao Shen, et al.. (2013). The RNA Binding Protein RBM38 (RNPC1) Regulates Splicing during Late Erythroid Differentiation. PLoS ONE. 8(10). e78031–e78031. 39 indexed citations
9.
Cieply, Benjamin, P. A. Riley, Phillip M. Pifer, et al.. (2012). Suppression of the Epithelial–Mesenchymal Transition by Grainyhead-like-2. Cancer Research. 72(9). 2440–2453. 168 indexed citations
10.
Kumar, Sanjeev, Sun Hee Park, Benjamin Cieply, et al.. (2011). A Pathway for the Control of Anoikis Sensitivity by E-Cadherin and Epithelial-to-Mesenchymal Transition. Molecular and Cellular Biology. 31(19). 4036–4051. 102 indexed citations
11.
Behari, Jaideep, Benjamin Cieply, Udayan Apte, et al.. (2009). Liver-Specific β-Catenin Knockout Mice Exhibit Defective Bile Acid and Cholesterol Homeostasis and Increased Susceptibility to Diet-Induced Steatohepatitis. American Journal Of Pathology. 176(2). 744–753. 101 indexed citations
12.
Nejak‐Bowen, Kari, Gang Zeng, Xinping Tan, Benjamin Cieply, & Satdarshan P. Monga. (2009). β-Catenin Regulates Vitamin C Biosynthesis and Cell Survival in Murine Liver. Journal of Biological Chemistry. 284(41). 28115–28127. 36 indexed citations
13.
Apte, Udayan, Sucha Singh, Gang Zeng, et al.. (2009). Beta-Catenin Activation Promotes Liver Regeneration after Acetaminophen-Induced Injury. American Journal Of Pathology. 175(3). 1056–1065. 130 indexed citations
14.
Tan, Xinping, Youzhong Yuan, Gang Zeng, et al.. (2008). β-Catenin deletion in hepatoblasts disrupts hepatic morphogenesis and survival during mouse development. Hepatology. 47(5). 1667–1679. 148 indexed citations
15.
Cieply, Benjamin, Gang Zeng, Tracy Proverbs‐Singh, David A. Geller, & Satdarshan P. Monga. (2008). Unique phenotype of hepatocellular cancers with exon‐3 mutations in beta‐catenin gene†. Hepatology. 49(3). 821–831. 126 indexed citations
16.
Apte, Udayan, Gang Zeng, Michael D. Thompson, et al.. (2007). β-Catenin is critical for early postnatal liver growth. American Journal of Physiology-Gastrointestinal and Liver Physiology. 292(6). G1578–G1585. 86 indexed citations
17.
Apte, Udayan, Michael D. Thompson, Shanshan Cui, et al.. (2007). Wnt/β-catenin signaling mediates oval cell response in rodents. Hepatology. 47(1). 288–295. 130 indexed citations
18.
Zeng, Gang, Udayan Apte, Benjamin Cieply, Sucha Singh, & Satdarshan P. Monga. (2007). siRNA-Mediated β-Catenin Knockdown in Human Hepatoma Cells Results in Decreased Growth and Survival. Neoplasia. 9(11). 951–959. 103 indexed citations
19.
Apte, Udayan, Gang Zeng, Peggy Müller, et al.. (2006). Activation of Wnt/β‐catenin pathway during hepatocyte growth factor–induced hepatomegaly in mice†. Hepatology. 44(4). 992–1002. 77 indexed citations
20.
Tan, Xinping, Jaideep Behari, Benjamin Cieply, George K. Michalopoulos, & Satdarshan P. Monga. (2006). Conditional Deletion of β-Catenin Reveals Its Role in Liver Growth and Regeneration. Gastroenterology. 131(5). 1561–1572. 288 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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