Benjamin C. Lewis

1.2k total citations
27 papers, 938 citations indexed

About

Benjamin C. Lewis is a scholar working on Pharmacology, Oncology and Molecular Biology. According to data from OpenAlex, Benjamin C. Lewis has authored 27 papers receiving a total of 938 indexed citations (citations by other indexed papers that have themselves been cited), including 18 papers in Pharmacology, 14 papers in Oncology and 11 papers in Molecular Biology. Recurrent topics in Benjamin C. Lewis's work include Pharmacogenetics and Drug Metabolism (18 papers), Drug Transport and Resistance Mechanisms (13 papers) and Neonatal Health and Biochemistry (3 papers). Benjamin C. Lewis is often cited by papers focused on Pharmacogenetics and Drug Metabolism (18 papers), Drug Transport and Resistance Mechanisms (13 papers) and Neonatal Health and Biochemistry (3 papers). Benjamin C. Lewis collaborates with scholars based in Australia, United Kingdom and United States. Benjamin C. Lewis's co-authors include John O. Miners, David J. Elliot, Peter I. Mackenzie, Robyn Meech, Sara Tommasi, Arduino A. Mangoni, Thomas M. Polasek, Krongtong Yoovathaworn, Elizabeth M. J. Gillam and Julie-Ann Hulin and has published in prestigious journals such as Journal of Biological Chemistry, SHILAP Revista de lepidopterología and Scientific Reports.

In The Last Decade

Benjamin C. Lewis

27 papers receiving 910 citations

Peers

Benjamin C. Lewis
Catherine Spire France
Kazutomi Kusano Japan
Alison E.M. Vickers United States
Kajsa P. Kanebratt Sweden
Tetsuya Kamataki Japan
Yingjie Guo China
Ylva Terelius Sweden
Ajay Madan United States
Stephen Fowler Switzerland
Xavier Boulenc France
Catherine Spire France
Benjamin C. Lewis
Citations per year, relative to Benjamin C. Lewis Benjamin C. Lewis (= 1×) peers Catherine Spire

Countries citing papers authored by Benjamin C. Lewis

Since Specialization
Citations

This map shows the geographic impact of Benjamin C. Lewis's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Benjamin C. Lewis with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Benjamin C. Lewis more than expected).

Fields of papers citing papers by Benjamin C. Lewis

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Benjamin C. Lewis. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Benjamin C. Lewis. The network helps show where Benjamin C. Lewis may publish in the future.

Co-authorship network of co-authors of Benjamin C. Lewis

This figure shows the co-authorship network connecting the top 25 collaborators of Benjamin C. Lewis. A scholar is included among the top collaborators of Benjamin C. Lewis based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Benjamin C. Lewis. Benjamin C. Lewis is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Tommasi, Sara, David J. Elliot, Mariasole Da Boit, et al.. (2018). Homoarginine and inhibition of human arginase activity: kinetic characterization and biological relevance. Scientific Reports. 8(1). 3697–3697. 44 indexed citations
2.
3.
Hulin, Julie-Ann, Sara Tommasi, David J. Elliot, et al.. (2017). MiR-193b regulates breast cancer cell migration and vasculogenic mimicry by targeting dimethylarginine dimethylaminohydrolase 1. Scientific Reports. 7(1). 13996–13996. 61 indexed citations
4.
Tommasi, Sara, David J. Elliot, Julie-Ann Hulin, et al.. (2017). Human dimethylarginine dimethylaminohydrolase 1 inhibition by proton pump inhibitors and the cardiovascular risk marker asymmetric dimethylarginine: in vitro and in vivo significance. Scientific Reports. 7(1). 2871–2871. 16 indexed citations
5.
Lewis, Benjamin C., et al.. (2016). Impaired dacarbazine activation and 7-ethoxyresorufin deethylation in vitro by polymorphic variants of CYP1A1 and CYP1A2. Pharmacogenetics and Genomics. 26(10). 453–461. 7 indexed citations
6.
Lewis, Benjamin C., Pramod C. Nair, Andrew A. Somogyi, et al.. (2015). Warfarin resistance associated with genetic polymorphism of VKORC1. Pharmacogenetics and Genomics. 26(1). 44–50. 9 indexed citations
7.
Lewis, Benjamin C., et al.. (2014). Cross-sections for (p,x) reactions on natural chromium for the production of 52,52m,54Mn radioisotopes. Applied Radiation and Isotopes. 96. 154–161. 34 indexed citations
8.
Chau, Nuy, David J. Elliot, Benjamin C. Lewis, et al.. (2014). Morphine Glucuronidation and Glucosidation Represent Complementary Metabolic Pathways That Are Both Catalyzed by UDP-Glucuronosyltransferase 2B7: Kinetic, Inhibition, and Molecular Modeling Studies. Journal of Pharmacology and Experimental Therapeutics. 349(1). 126–137. 46 indexed citations
9.
Lewis, Benjamin C. & John O. Miners. (2013). Generation, Validation, and Application of a P450 Homology Model. Current Topics in Medicinal Chemistry. 13(18). 2233–2240. 2 indexed citations
10.
Meech, Robyn, et al.. (2012). Identification of Residues That Confer Sugar Selectivity to UDP-Glycosyltransferase 3A (UGT3A) Enzymes. Journal of Biological Chemistry. 287(29). 24122–24130. 28 indexed citations
11.
Meech, Robyn, John O. Miners, Benjamin C. Lewis, & Peter I. Mackenzie. (2012). The glycosidation of xenobiotics and endogenous compounds: Versatility and redundancy in the UDP glycosyltransferase superfamily. Pharmacology & Therapeutics. 134(2). 200–218. 104 indexed citations
12.
Lewis, Benjamin C., Peter I. Mackenzie, & John O. Miners. (2011). Application of Homology Modeling to Generate CYP1A1 Mutants with Enhanced Activation of the Cancer Chemotherapeutic Prodrug Dacarbazine. Molecular Pharmacology. 80(5). 879–888. 18 indexed citations
13.
Lewis, Benjamin C., Peter I. Mackenzie, & John O. Miners. (2011). Homodimerization of UDP-glucuronosyltransferase 2B7 (UGT2B7) and identification of a putative dimerization domain by protein homology modeling. Biochemical Pharmacology. 82(12). 2016–2023. 26 indexed citations
14.
Voelcker, Nicolas H., et al.. (2009). AFM study of the interaction of cytochrome P450 2C9 with phospholipid bilayers. Chemistry and Physics of Lipids. 163(2). 182–189. 15 indexed citations
15.
16.
Lewis, Benjamin C., Peter I. Mackenzie, & John O. Miners. (2007). Comparative homology modeling of human cytochrome P4501A1 (CYP1A1) and confirmation of residues involved in 7-ethoxyresorufin O-deethylation by site-directed mutagenesis and enzyme kinetic analysis. Archives of Biochemistry and Biophysics. 468(1). 58–69. 30 indexed citations
17.
Kubota, Takahiro, Benjamin C. Lewis, David J. Elliot, Peter I. Mackenzie, & John O. Miners. (2007). Critical Roles of Residues 36 and 40 in the Phenol and Tertiary Amine Aglycone Substrate Selectivities of UDP-Glucuronosyltransferases 1A3 and 1A4. Molecular Pharmacology. 72(4). 1054–1062. 49 indexed citations
18.
Lewis, Benjamin C., Peter I. Mackenzie, David J. Elliot, et al.. (2006). Amino terminal domains of human UDP-glucuronosyltransferases (UGT) 2B7 and 2B15 associated with substrate selectivity and autoactivation. Biochemical Pharmacology. 73(9). 1463–1473. 41 indexed citations
19.
Johnson, Daniel L., Benjamin C. Lewis, David J. Elliot, John O. Miners, & Lisandra L. Martin. (2005). Electrochemical characterisation of the human cytochrome P450 CYP2C9. Biochemical Pharmacology. 69(10). 1533–1541. 61 indexed citations
20.
Polasek, Thomas M., David J. Elliot, Benjamin C. Lewis, & John O. Miners. (2004). Mechanism-Based Inactivation of Human Cytochrome P4502C8 by Drugs in Vitro. Journal of Pharmacology and Experimental Therapeutics. 311(3). 996–1007. 76 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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