Beatrice B. Magee

458 total citations
13 papers, 362 citations indexed

About

Beatrice B. Magee is a scholar working on Infectious Diseases, Molecular Biology and Epidemiology. According to data from OpenAlex, Beatrice B. Magee has authored 13 papers receiving a total of 362 indexed citations (citations by other indexed papers that have themselves been cited), including 10 papers in Infectious Diseases, 8 papers in Molecular Biology and 8 papers in Epidemiology. Recurrent topics in Beatrice B. Magee's work include Antifungal resistance and susceptibility (10 papers), Fungal Infections and Studies (8 papers) and Fungal and yeast genetics research (8 papers). Beatrice B. Magee is often cited by papers focused on Antifungal resistance and susceptibility (10 papers), Fungal Infections and Studies (8 papers) and Fungal and yeast genetics research (8 papers). Beatrice B. Magee collaborates with scholars based in United States, Australia and New Zealand. Beatrice B. Magee's co-authors include P. T. Magee, Paul T. Magee, Paul Lephart, Mélanie Legrand, Thomas J. Walsh, Anja Forche, Alejandro Cassola, Susana Silberstein, Susana Passeron and Luc Giasson and has published in prestigious journals such as Genetics, Molecular Microbiology and Gene.

In The Last Decade

Beatrice B. Magee

13 papers receiving 353 citations

Peers

Beatrice B. Magee
Beatrice B. Magee
Citations per year, relative to Beatrice B. Magee Beatrice B. Magee (= 1×) peers Raquel Martínez‐López

Countries citing papers authored by Beatrice B. Magee

Since Specialization
Citations

This map shows the geographic impact of Beatrice B. Magee's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Beatrice B. Magee with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Beatrice B. Magee more than expected).

Fields of papers citing papers by Beatrice B. Magee

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Beatrice B. Magee. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Beatrice B. Magee. The network helps show where Beatrice B. Magee may publish in the future.

Co-authorship network of co-authors of Beatrice B. Magee

This figure shows the co-authorship network connecting the top 25 collaborators of Beatrice B. Magee. A scholar is included among the top collaborators of Beatrice B. Magee based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Beatrice B. Magee. Beatrice B. Magee is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

13 of 13 papers shown
1.
Schmid, Jan, Paul T. Magee, Barbara R. Holland, et al.. (2015). Last hope for the doomed? Thoughts on the importance of a parasexual cycle for the yeast Candida albicans. Current Genetics. 62(1). 81–85. 6 indexed citations
2.
Zhang, Ningxin, Beatrice B. Magee, Paul T. Magee, et al.. (2015). Selective Advantages of a Parasexual Cycle for the YeastCandida albicans. Genetics. 200(4). 1117–1132. 18 indexed citations
3.
Thewes, Sascha, Gary P. Moran, Beatrice B. Magee, et al.. (2008). Phenotypic screening, transcriptional profiling, and comparative genomic analysis of an invasive and non-invasive strain of Candida albicans. BMC Microbiology. 8(1). 187–187. 36 indexed citations
4.
Gómez‐Raja, Jonathan, Encarnación Andaluz, Beatrice B. Magee, Richard Calderone, & Germán Larriba. (2007). A single SNP, G929T (Gly310Val), determines the presence of a functional and a non-functional allele of HIS4 in Candida albicans SC5314: Detection of the non-functional allele in laboratory strains. Fungal Genetics and Biology. 45(4). 527–541. 19 indexed citations
5.
Magee, Beatrice B. & Paul T. Magee. (2005). Recent advances in the genomic analysis of Candida albicans. Revista Iberoamericana de Micología. 22(4). 187–193. 7 indexed citations
6.
Legrand, Mélanie, Paul Lephart, Anja Forche, et al.. (2004). Homozygosity at the MTL locus in clinical strains of Candida albicans: karyotypic rearrangements and tetraploid formation. Molecular Microbiology. 52(5). 1451–1462. 81 indexed citations
7.
Magee, P. T. & Beatrice B. Magee. (2004). Through a glass opaquely: the biological significance of mating in Candida albicans. Current Opinion in Microbiology. 7(6). 661–665. 34 indexed citations
8.
Cassola, Alejandro, Susana Silberstein, Beatrice B. Magee, et al.. (2004). Candida albicans Lacking the Gene Encoding the Regulatory Subunit of Protein Kinase A Displays a Defect in Hyphal Formation and an Altered Localization of the Catalytic Subunit. Eukaryotic Cell. 3(1). 190–199. 61 indexed citations
11.
Raymond, Martine, et al.. (1998). A Ste6p/P‐glycoprotein homologue from the asexual yeast Candida albicans transports the a‐factor mating pheromone in Saccharomyces cerevisiae. Molecular Microbiology. 27(3). 587–598. 42 indexed citations
12.
Hoyer, Lois L., et al.. (1994). The ARG4 gene of Candida albicans. Gene. 142(2). 213–218. 18 indexed citations
13.
Stein, Gary E., et al.. (1991). Use of rDNA restriction fragment length polymorphisms to differentiate strains of Candida albicans in women with vulvovaginal candidiasis. Diagnostic Microbiology and Infectious Disease. 14(6). 459–464. 23 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

Explore authors with similar magnitude of impact

Rankless by CCL
2026