Bastian Opitz

7.7k total citations · 1 hit paper
89 papers, 5.4k citations indexed

About

Bastian Opitz is a scholar working on Immunology, Molecular Biology and Epidemiology. According to data from OpenAlex, Bastian Opitz has authored 89 papers receiving a total of 5.4k indexed citations (citations by other indexed papers that have themselves been cited), including 52 papers in Immunology, 28 papers in Molecular Biology and 23 papers in Epidemiology. Recurrent topics in Bastian Opitz's work include Immune Response and Inflammation (34 papers), Pneumonia and Respiratory Infections (19 papers) and interferon and immune responses (14 papers). Bastian Opitz is often cited by papers focused on Immune Response and Inflammation (34 papers), Pneumonia and Respiratory Infections (19 papers) and interferon and immune responses (14 papers). Bastian Opitz collaborates with scholars based in Germany, United States and United Kingdom. Bastian Opitz's co-authors include Norbert Suttorp, Stefan Hippenstiel, Bernd Schmeck, Julia Eitel, Andreas C. Hocke, Vincent van Laak, Philippe Dje N’Guessan, Martin Witzenrath, Ralf R. Schumann and Hortense Slevogt and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of the American Chemical Society and Journal of Biological Chemistry.

In The Last Decade

Bastian Opitz

85 papers receiving 5.3k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Differential requirement for the activation of the inflammasome for processing and release of IL-1β in monocytes and macrophages

2008 660 citations Mihai G. Netea, Bastian Opitz et al. profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Bastian Opitz Germany	39	3.0k	2.2k	1.4k	578	577	89	5.4k
Bernd Schmeck Germany	41	2.0k 0.7×	2.1k 1.0×	1.3k 0.9×	675 1.2×	609 1.1×	145	5.1k
Trude Helen Flo Norway	30	2.5k 0.8×	1.4k 0.6×	1.3k 0.9×	490 0.8×	447 0.8×	59	5.4k
Ae‐Kyung Yi United States	29	5.8k 2.0×	2.4k 1.1×	1.0k 0.7×	384 0.7×	761 1.3×	52	8.0k
Irina Smirnova United States	21	6.0k 2.0×	2.0k 0.9×	1.5k 1.1×	602 1.0×	907 1.6×	38	8.2k
Martha Triantafilou United Kingdom	41	3.6k 1.2×	2.4k 1.1×	1.2k 0.8×	286 0.5×	526 0.9×	82	6.6k
Thomas Miethke Germany	34	3.2k 1.1×	1.4k 0.7×	1.1k 0.8×	212 0.4×	821 1.4×	115	5.4k
Amal O. Amer United States	39	3.1k 1.0×	4.2k 1.9×	1.3k 0.9×	678 1.2×	260 0.5×	89	6.8k
Kathrin S. Michelsen United States	36	4.0k 1.3×	1.9k 0.9×	1.2k 0.9×	382 0.7×	655 1.1×	64	6.8k
Shawn Skerrett United States	44	2.0k 0.7×	1.5k 0.7×	1.6k 1.1×	1.1k 1.9×	364 0.6×	95	5.5k
Letícia A. M. Carneiro Brazil	22	2.3k 0.8×	1.7k 0.8×	1.1k 0.8×	177 0.3×	480 0.8×	30	4.2k

Countries citing papers authored by Bastian Opitz

Since Specialization

Citations

This map shows the geographic impact of Bastian Opitz's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Bastian Opitz with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Bastian Opitz more than expected).

Fields of papers citing papers by Bastian Opitz

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Bastian Opitz. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Bastian Opitz. The network helps show where Bastian Opitz may publish in the future.

Co-authorship network of co-authors of Bastian Opitz

This figure shows the co-authorship network connecting the top 25 collaborators of Bastian Opitz. A scholar is included among the top collaborators of Bastian Opitz based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Bastian Opitz. Bastian Opitz is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Drude, Natascha, Stephan P. Rosshart, Andreas Diefenbach, et al.. (2024). A facility for laboratory mice with a natural microbiome at Charité – Universitätsmedizin Berlin. Lab Animal. 53(12). 351–354. 1 indexed citations

Shen, Dacheng, Ling Yao, Bastian Opitz, et al.. (2024). Semisynthetic Glycoconjugate Vaccine Lead against Klebsiella pneumoniae Serotype O2afg Induces Functional Antibodies and Reduces the Burden of Acute Pneumonia. Journal of the American Chemical Society. 146(51). 35356–35366. 2 indexed citations

Lippmann, Juliane, Katja Hönzke, Andreas C. Hocke, et al.. (2023). CLEC12A Binds to Legionella pneumophila but Has No Impact on the Host’s Antibacterial Response. International Journal of Molecular Sciences. 24(4). 3891–3891. 7 indexed citations

Lingscheid, Tilman, Lena J. Lippert, David Hillus, et al.. (2022). Characterization of antimicrobial use and co-infections among hospitalized patients with COVID-19: a prospective observational cohort study. Infection. 50(6). 1441–1452. 7 indexed citations

Thibeault, Charlotte, Norbert Suttorp, & Bastian Opitz. (2021). The microbiota in pneumonia: From protection to predisposition. Science Translational Medicine. 13(576). 72 indexed citations

Wee, Bryan A., Joana Alves, Diane Lindsay, et al.. (2021). Population analysis of Legionella pneumophila reveals a basis for resistance to complement-mediated killing. Nature Communications. 12(1). 7165–7165. 13 indexed citations

Pei, Gang, Teresa Domaszewska, Joanna Żyła, et al.. (2020). Platelets Restrict the Oxidative Burst in Phagocytes and Facilitate Primary Progressive Tuberculosis. American Journal of Respiratory and Critical Care Medicine. 202(5). 730–744. 13 indexed citations

Suttorp, Norbert, et al.. (2019). Microbiota-Dependent Regulation of Antimicrobial Immunity in the Lung. American Journal of Respiratory Cell and Molecular Biology. 61(3). 284–289. 14 indexed citations

Caronni, Nicoletta, Corrado Guarnaccia, Bastian Opitz, et al.. (2018). Downregulation of Membrane Trafficking Proteins and Lactate Conditioning Determine Loss of Dendritic Cell Function in Lung Cancer. Cancer Research. 78(7). 1685–1699. 103 indexed citations

10.

Hamann, Lutz, Javeed A. Shah, Annelies Verbon, et al.. (2018). The common HAQ STING variant impairs cGAS-dependent antibacterial responses and is associated with susceptibility to Legionnaires’ disease in humans. PLoS Pathogens. 14(1). e1006829–e1006829. 43 indexed citations

11.

Zahlten, Janine, Juliane Lippmann, Philippe Dje N’Guessan, et al.. (2010). Legionella pneumophila-induced IκBζ-dependent expression of interleukin-6 in lung epithelium. European Respiratory Journal. 37(3). 648–657. 20 indexed citations

12.

Buß, Claudia, Bastian Opitz, Andreas C. Hocke, et al.. (2010). Essential Role of Mitochondrial Antiviral Signaling, IFN Regulatory Factor (IRF)3, and IRF7 in Chlamydophila pneumoniae -Mediated IFN-β Response and Control of Bacterial Replication in Human Endothelial Cells. The Journal of Immunology. 184(6). 3072–3078. 31 indexed citations

13.

Meixenberger, Karolin, Florence Pache, Julia Eitel, et al.. (2009). Listeria monocytogenes -Infected Human Peripheral Blood Mononuclear Cells Produce IL-1β, Depending on Listeriolysin O and NLRP3. The Journal of Immunology. 184(2). 922–930. 157 indexed citations

14.

Vinzing, Maya, Julia Eitel, Juliane Lippmann, et al.. (2008). NAIP and Ipaf Control Legionella pneumophila Replication in Human Cells. The Journal of Immunology. 180(10). 6808–6815. 102 indexed citations

15.

Schmeck, Bernd, Philippe Dje N’Guessan, Bastian Opitz, et al.. (2008). Histone Acetylation and Flagellin Are Essential for Legionella pneumophila -Induced Cytokine Expression. The Journal of Immunology. 181(2). 940–947. 66 indexed citations

16.

Eitel, Julia, Matthias Krüll, Andreas C. Hocke, et al.. (2008). β-PIX and Rac1 GTPase Mediate Trafficking and Negative Regulation of NOD2. The Journal of Immunology. 181(4). 2664–2671. 53 indexed citations

17.

N’Guessan, Philippe Dje, Sebastian Bachmann, Bastian Opitz, et al.. (2007). The UspA1 Protein ofMoraxella catarrhalisInduces CEACAM‐1–Dependent Apoptosis in Alveolar Epithelial Cells. The Journal of Infectious Diseases. 195(11). 1651–1660. 27 indexed citations

18.

Belyi, Yury, Ricarda Niggeweg, Bastian Opitz, et al.. (2006). Legionella pneumophila glucosyltransferase inhibits host elongation factor 1A. Proceedings of the National Academy of Sciences. 103(45). 16953–16958. 128 indexed citations

19.

Opitz, Bastian, Wiebke Beermann, Andreas C. Hocke, et al.. (2006). Listeria monocytogenes Activated p38 MAPK and Induced IL-8 Secretion in a Nucleotide-Binding Oligomerization Domain 1-Dependent Manner in Endothelial Cells. The Journal of Immunology. 176(1). 484–490. 151 indexed citations

20.

Schröder, Nicolas W.J., Bastian Opitz, Norbert Lamping, et al.. (2000). Involvement of Lipopolysaccharide Binding Protein, CD14, and Toll-Like Receptors in the Initiation of Innate Immune Responses by Treponema Glycolipids. The Journal of Immunology. 165(5). 2683–2693. 121 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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