Bart Lammers

600 total citations
15 papers, 473 citations indexed

About

Bart Lammers is a scholar working on Surgery, Molecular Biology and Endocrinology, Diabetes and Metabolism. According to data from OpenAlex, Bart Lammers has authored 15 papers receiving a total of 473 indexed citations (citations by other indexed papers that have themselves been cited), including 12 papers in Surgery, 8 papers in Molecular Biology and 5 papers in Endocrinology, Diabetes and Metabolism. Recurrent topics in Bart Lammers's work include Cholesterol and Lipid Metabolism (11 papers), Peroxisome Proliferator-Activated Receptors (6 papers) and Diabetes, Cardiovascular Risks, and Lipoproteins (3 papers). Bart Lammers is often cited by papers focused on Cholesterol and Lipid Metabolism (11 papers), Peroxisome Proliferator-Activated Receptors (6 papers) and Diabetes, Cardiovascular Risks, and Lipoproteins (3 papers). Bart Lammers collaborates with scholars based in Netherlands, China and United States. Bart Lammers's co-authors include Miranda Van Eck, Ying Zhao, Illiana Meurs, Theo J.C. van Berkel, Menno Hoekstra, Reeni B. Hildebrand, Dan Ye, Ruud Out, Johan Kuiper and Patrick C.N. Rensen and has published in prestigious journals such as PLoS ONE, Arteriosclerosis Thrombosis and Vascular Biology and Journal of Lipid Research.

In The Last Decade

Bart Lammers

15 papers receiving 469 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Bart Lammers Netherlands 10 255 187 108 104 97 15 473
Zhaosha Li Netherlands 8 202 0.8× 147 0.8× 115 1.1× 53 0.5× 49 0.5× 9 409
Brian Fulp United States 7 196 0.8× 189 1.0× 184 1.7× 61 0.6× 86 0.9× 7 472
Nicole Herijgers Netherlands 10 218 0.9× 200 1.1× 63 0.6× 85 0.8× 189 1.9× 14 518
Jerry Angdisen United States 12 187 0.7× 300 1.6× 98 0.9× 73 0.7× 49 0.5× 23 583
Yuji Matsuzawa Japan 11 150 0.6× 259 1.4× 60 0.6× 71 0.7× 110 1.1× 17 499
Debi K. Swertfeger United States 16 253 1.0× 306 1.6× 131 1.2× 168 1.6× 143 1.5× 23 773
Kosuke Tsukamoto Japan 10 250 1.0× 237 1.3× 129 1.2× 52 0.5× 56 0.6× 19 438
Minjia Zhu China 7 198 0.8× 299 1.6× 86 0.8× 113 1.1× 38 0.4× 11 569
Inês Cebola United Kingdom 10 215 0.8× 379 2.0× 68 0.6× 96 0.9× 52 0.5× 22 674
Chiaki Ikegami Japan 7 246 1.0× 319 1.7× 125 1.2× 60 0.6× 60 0.6× 9 576

Countries citing papers authored by Bart Lammers

Since Specialization
Citations

This map shows the geographic impact of Bart Lammers's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Bart Lammers with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Bart Lammers more than expected).

Fields of papers citing papers by Bart Lammers

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Bart Lammers. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Bart Lammers. The network helps show where Bart Lammers may publish in the future.

Co-authorship network of co-authors of Bart Lammers

This figure shows the co-authorship network connecting the top 25 collaborators of Bart Lammers. A scholar is included among the top collaborators of Bart Lammers based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Bart Lammers. Bart Lammers is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

15 of 15 papers shown
1.
Zhao, Ying, Laura Calpe‐Berdiel, Bart Lammers, et al.. (2021). Impact of bone marrow ATP-binding cassette transporter A1 deficiency on atherogenesis is independent of the presence of the low-density lipoprotein receptor. Atherosclerosis. 319. 79–85. 4 indexed citations
2.
Hoekstra, Menno, Ronald J. van der Sluis, Reeni B. Hildebrand, et al.. (2020). Disruption of Phospholipid Transfer Protein–Mediated High-Density Lipoprotein Maturation Reduces Scavenger Receptor BI Deficiency–Driven Atherosclerosis Susceptibility Despite Unexpected Metabolic Complications. Arteriosclerosis Thrombosis and Vascular Biology. 40(3). 611–623. 6 indexed citations
3.
Louwe, Mieke C., Bart Lammers, Miguel A. Frias, et al.. (2016). Abca1 deficiency protects the heart against myocardial infarction-induced injury. Atherosclerosis. 251. 159–163. 8 indexed citations
4.
Kooijman, Sander, Illiana Meurs, Kim L. L. Habets, et al.. (2014). Hematopoietic α7 nicotinic acetylcholine receptor deficiency increases inflammation and platelet activation status, but does not aggravate atherosclerosis. Journal of Thrombosis and Haemostasis. 13(1). 126–135. 22 indexed citations
5.
Kampen, Erik-Jan Van, Reeni B. Hildebrand, Bart Lammers, et al.. (2014). Orp8 Deficiency in Bone Marrow-Derived Cells Reduces Atherosclerotic Lesion Progression in LDL Receptor Knockout Mice. PLoS ONE. 9(10). e109024–e109024. 8 indexed citations
6.
Tanck, Michael W.T., Ruud Out, Elise F. Villard, et al.. (2012). Human ATP–Binding Cassette G1 Controls Macrophage Lipoprotein Lipase Bioavailability and Promotes Foam Cell Formation. Arteriosclerosis Thrombosis and Vascular Biology. 32(9). 2223–2231. 32 indexed citations
7.
Lammers, Bart, Ying Zhao, Amanda C. Foks, et al.. (2012). Leukocyte ABCA1 Remains Atheroprotective in Splenectomized LDL Receptor Knockout Mice. PLoS ONE. 7(10). e48080–e48080. 5 indexed citations
8.
Meurs, Illiana, Bart Lammers, Ying Zhao, et al.. (2011). The effect of ABCG1 deficiency on atherosclerotic lesion development in LDL receptor knockout mice depends on the stage of atherogenesis. Atherosclerosis. 221(1). 41–47. 60 indexed citations
10.
Lammers, Bart, Ying Zhao, Menno Hoekstra, et al.. (2011). Augmented Atherogenesis in LDL Receptor Deficient Mice Lacking Both Macrophage ABCA1 and ApoE. PLoS ONE. 6(10). e26095–e26095. 15 indexed citations
11.
Hildebrand, Reeni B., Bart Lammers, Illiana Meurs, et al.. (2010). Restoration of High-Density Lipoprotein Levels by Cholesteryl Ester Transfer Protein Expression in Scavenger Receptor Class B Type I (SR-BI) Knockout Mice Does Not Normalize Pathologies Associated With SR-BI Deficiency. Arteriosclerosis Thrombosis and Vascular Biology. 30(7). 1439–1445. 45 indexed citations
12.
Lammers, Bart, Prakash G. Chandak, Elma Aflaki, et al.. (2010). Macrophage Adipose Triglyceride Lipase Deficiency Attenuates Atherosclerotic Lesion Development in Low-Density Lipoprotein Receptor Knockout Mice. Arteriosclerosis Thrombosis and Vascular Biology. 31(1). 67–73. 40 indexed citations
13.
Lammers, Bart, Ruud Out, Reeni B. Hildebrand, et al.. (2009). Independent protective roles for macrophage Abcg1 and Apoe in the atherosclerotic lesion development. Atherosclerosis. 205(2). 420–426. 21 indexed citations
14.
Hoekstra, Menno, Dan Ye, Reeni B. Hildebrand, et al.. (2009). Scavenger receptor class B type I-mediated uptake of serum cholesterol is essential for optimal adrenal glucocorticoid production. Journal of Lipid Research. 50(6). 1039–1046. 57 indexed citations
15.
Hoekstra, Menno, Bart Lammers, Ruud Out, et al.. (2008). Activation of the Nuclear Receptor PXR Decreases Plasma LDL-Cholesterol Levels and Induces Hepatic Steatosis in LDL Receptor Knockout Mice. Molecular Pharmaceutics. 6(1). 182–189. 38 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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