Barbara Visentin
Impact in
- Cell Biology top 5%
- Endoplasmic Reticulum Stress and Disease
- Cellular transport and secretion
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- Sphingolipid Metabolism and Signaling
- Lipid Membrane Structure and Behavior
- Protein Kinase Regulation and GTPase Signaling
Papers in
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- Endoplasmic Reticulum Stress and Disease 2
- Cellular transport and secretion 2
- Co-authors
- Roger A. SabbadiniKelli MorenoJohn VekichRosalía MatteoAmy L. CavalliShuangxing YuHassan HallVikas Kundra
- Journals
- Journal of Lipid Research (2 papers)Clinical Cancer Research (1 paper)Molecular Therapy — Oncolytics (1 paper)Biochemical Pharmacology (1 paper)Biochemical and Biophysical Research Communications (1 paper)
- Partner nations
- United StatesUnited KingdomItaly
In The Last Decade
Barbara Visentin
9 papers receiving 631 citations
Peers
Comparison fields: 5 of 72
- Cell Biology 260
- Molecular Biology 550
- Cancer Research 71
- Immunology 81
- Physiology 65
Countries citing papers authored by Barbara Visentin
This map shows the geographic impact of Barbara Visentin's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Barbara Visentin with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Barbara Visentin more than expected).
Fields of papers citing papers by Barbara Visentin
This network shows the impact of papers produced by Barbara Visentin. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Barbara Visentin. The network helps show where Barbara Visentin may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Barbara Visentin, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2017 | 6 | |
| 2 | 2017 | 4 | |
| 3 | 2016 | 21 | |
| 4 | 2015 | 67 | |
| 5 | 2012 | 3 | |
| 6 | 2009 | 96 | |
| 7 | 2006 | 378 | |
| 8 | 2004 | 44 | |
| 9 | 2003 | 27 |
About Barbara Visentin
Barbara Visentin is a scholar working on Cell Biology, Gastroenterology, Molecular Biology, Biotechnology and Immunology, having authored 9 papers that have together received 646 indexed citations. Recurring topics across this work include Sphingolipid Metabolism and Signaling (4 papers), Protein Kinase Regulation and GTPase Signaling (2 papers), Cell death mechanisms and regulation (2 papers), Endoplasmic Reticulum Stress and Disease (2 papers), Angiogenesis and VEGF in Cancer (2 papers), Lipid Membrane Structure and Behavior (2 papers), Cellular transport and secretion (2 papers) and PI3K/AKT/mTOR signaling in cancer (2 papers). The work is most often cited by research in Cell Biology (260 citations), Molecular Biology (550 citations), Cancer Research (71 citations), Immunology (81 citations) and Physiology (65 citations). Barbara Visentin has collaborated with scholars based in United States, United Kingdom and Italy. Frequent co-authors include Roger A. Sabbadini, Kelli Moreno, John Vekich, Rosalía Matteo, Amy L. Cavalli, Shuangxing Yu, Hassan Hall, Vikas Kundra, Yiling Lu and Gordon B. Mills. Their work appears in journals such as Journal of Lipid Research, Clinical Cancer Research, Molecular Therapy — Oncolytics, Biochemical Pharmacology and Biochemical and Biophysical Research Communications.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.