Bai‐Yan Li

1.7k total citations
103 papers, 1.4k citations indexed

About

Bai‐Yan Li is a scholar working on Molecular Biology, Oncology and Cellular and Molecular Neuroscience. According to data from OpenAlex, Bai‐Yan Li has authored 103 papers receiving a total of 1.4k indexed citations (citations by other indexed papers that have themselves been cited), including 58 papers in Molecular Biology, 31 papers in Oncology and 14 papers in Cellular and Molecular Neuroscience. Recurrent topics in Bai‐Yan Li's work include Ion channel regulation and function (14 papers), Bone health and treatments (13 papers) and Heart Rate Variability and Autonomic Control (9 papers). Bai‐Yan Li is often cited by papers focused on Ion channel regulation and function (14 papers), Bone health and treatments (13 papers) and Heart Rate Variability and Autonomic Control (9 papers). Bai‐Yan Li collaborates with scholars based in China, United States and Japan. Bai‐Yan Li's co-authors include Hiroki Yokota, John H. Schild, Shengzhi Liu, Guo‐Fen Qiao, Andy Chen, Yao Fan, Sungsoo Na, Xun Sun, Harikrishna Nakshatri and Feng Yan and has published in prestigious journals such as Circulation, Cancer Research and Scientific Reports.

In The Last Decade

Bai‐Yan Li

101 papers receiving 1.4k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Bai‐Yan Li China 21 744 329 161 156 156 103 1.4k
Marie‐Gabrielle Ludwig Switzerland 20 1.1k 1.4× 196 0.6× 69 0.4× 216 1.4× 174 1.1× 26 1.7k
Namik Kaya Saudi Arabia 23 761 1.0× 138 0.4× 88 0.5× 202 1.3× 133 0.9× 83 1.6k
Luqing Zhang China 22 746 1.0× 252 0.8× 121 0.8× 120 0.8× 149 1.0× 47 1.3k
Jianliang Zhang China 27 751 1.0× 317 1.0× 101 0.6× 189 1.2× 177 1.1× 75 2.1k
Susanne Lutz Germany 26 1.4k 1.9× 164 0.5× 418 2.6× 174 1.1× 112 0.7× 54 2.1k
Zhongji Liao United States 14 656 0.9× 101 0.3× 86 0.5× 157 1.0× 110 0.7× 21 1.6k
Gijsbertus J. Pronk Netherlands 17 1.5k 2.1× 338 1.0× 47 0.3× 290 1.9× 157 1.0× 22 2.4k
Roberto Piñeiro Spain 20 616 0.8× 508 1.5× 238 1.5× 100 0.6× 230 1.5× 44 2.1k
Guilai Liu Germany 17 900 1.2× 116 0.4× 162 1.0× 311 2.0× 198 1.3× 30 1.6k
Pieter J. Peeters Belgium 26 838 1.1× 370 1.1× 32 0.2× 249 1.6× 96 0.6× 40 2.2k

Countries citing papers authored by Bai‐Yan Li

Since Specialization
Citations

This map shows the geographic impact of Bai‐Yan Li's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Bai‐Yan Li with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Bai‐Yan Li more than expected).

Fields of papers citing papers by Bai‐Yan Li

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Bai‐Yan Li. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Bai‐Yan Li. The network helps show where Bai‐Yan Li may publish in the future.

Co-authorship network of co-authors of Bai‐Yan Li

This figure shows the co-authorship network connecting the top 25 collaborators of Bai‐Yan Li. A scholar is included among the top collaborators of Bai‐Yan Li based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Bai‐Yan Li. Bai‐Yan Li is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Zhao, Jiaxin, et al.. (2025). SERCA2 regulates Piezo1 channel activation and contributes to the cardiac function and baroreflex in mice. Acta Pharmacologica Sinica. 46(12). 3386–3395. 1 indexed citations
2.
Aryal, Uma K., Hee-Chang Lim, Jing Liu, et al.. (2024). Therapeutic Insights into Low-intensity Vibration for Generating Induced Tumor-Suppressive Cells and Modulating the Bone Microenvironment. Engineering. 43. 201–215. 2 indexed citations
3.
Li, Kexin, et al.. (2023). The Double-Edged Proteins in Cancer Proteomes and the Generation of Induced Tumor-Suppressing Cells (iTSCs). Proteomes. 11(1). 5–5. 4 indexed citations
4.
Li, Kexin, et al.. (2023). Three unconventional maxims in the natural selection of cancer cells: Generation of induced tumor-suppressing cells (iTSCs). International Journal of Biological Sciences. 19(5). 1403–1412. 1 indexed citations
5.
Li, Kexin, Xun Sun, Kazumasa Minami, et al.. (2023). Proteomes from AMPK-inhibited peripheral blood mononuclear cells suppress the progression of breast cancer and bone metastasis. Theranostics. 13(4). 1247–1263. 10 indexed citations
6.
Siegel, Amanda P., Shengzhi Liu, Sunil S. Tholpady, et al.. (2023). Canine-Inspired Chemometric Analysis of Volatile Organic Compounds in Urine Headspace to Distinguish Prostate Cancer in Mice and Men. Cancers. 15(4). 1352–1352. 10 indexed citations
7.
Li, Kexin, Xun Sun, Kazumasa Minami, et al.. (2023). The inhibition of pancreatic cancer progression by K-Ras-overexpressing mesenchymal stem cell-derived secretomes. Scientific Reports. 13(1). 15036–15036. 3 indexed citations
8.
9.
Wang, Luqi, Shengzhi Liu, Maitri Kalra, et al.. (2022). Chemometric Analysis of Urinary Volatile Organic Compounds to Monitor the Efficacy of Pitavastatin Treatments on Mammary Tumor Progression over Time. Molecules. 27(13). 4277–4277. 5 indexed citations
10.
11.
Li, Ying, Yan Feng, Li Liu, et al.. (2020). The baroreflex afferent pathway plays a critical role in H2S-mediated autonomic control of blood pressure regulation under physiological and hypertensive conditions. Acta Pharmacologica Sinica. 42(6). 898–908. 18 indexed citations
12.
Chen, Andy, Yue Wang, Yao Fan, et al.. (2019). Vinculin Force Sensor Detects Tumor-Osteocyte Interactions. Scientific Reports. 9(1). 5615–5615. 26 indexed citations
13.
Xu, Bing, Hongyu Gao, Bai‐Yan Li, et al.. (2019). Lipopolysaccharides Improve Mesenchymal Stem Cell-Mediated Cardioprotection by MyD88 and stat3 Signaling in a Mouse Model of Cardiac Ischemia/Reperfusion Injury. Stem Cells and Development. 28(9). 620–631. 18 indexed citations
14.
Wang, Luqi, Yue Wang, Andy Chen, et al.. (2018). Effects of a checkpoint kinase inhibitor, AZD7762, on tumor suppression and bone remodeling. PMC. 4 indexed citations
15.
Yan, Feng, Xinyu Li, Ying Li, et al.. (2018). Spontaneous activities in baroreflex afferent pathway contribute dominant role in parasympathetic neurocontrol of blood pressure regulation. CNS Neuroscience & Therapeutics. 24(12). 1219–1230. 8 indexed citations
16.
Guan, Jian, Miao Zhao, Chao He, et al.. (2018). Anti-Hypertensive Action of Fenofibrate via UCP2 Upregulation Mediated by PPAR Activation in Baroreflex Afferent Pathway. Neuroscience Bulletin. 35(1). 15–24. 10 indexed citations
17.
Minami, Kazumasa, Shengzhi Liu, Liu Yang, et al.. (2017). Inhibitory Effects of Dopamine Receptor D1 Agonist on Mammary Tumor and Bone Metastasis. Scientific Reports. 7(1). 45686–45686. 40 indexed citations
18.
Chen, Andy, Luqi Wang, Bai‐Yan Li, et al.. (2017). Reduction in Migratory Phenotype in a Metastasized Breast Cancer Cell Line via Downregulation of S100A4 and GRM3. PMC. 1 indexed citations
19.
20.
Li, Bai‐Yan, et al.. (2007). Unmyelinated visceral afferents exhibit frequency dependent action potential broadening while myelinated visceral afferents do not. Neuroscience Letters. 421(1). 62–66. 26 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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