B.A. Oostra

1.5k total citations
21 papers, 1.1k citations indexed

About

B.A. Oostra is a scholar working on Genetics, Molecular Biology and Cognitive Neuroscience. According to data from OpenAlex, B.A. Oostra has authored 21 papers receiving a total of 1.1k indexed citations (citations by other indexed papers that have themselves been cited), including 13 papers in Genetics, 12 papers in Molecular Biology and 5 papers in Cognitive Neuroscience. Recurrent topics in B.A. Oostra's work include Genetics and Neurodevelopmental Disorders (11 papers), Autism Spectrum Disorder Research (5 papers) and Ubiquitin and proteasome pathways (3 papers). B.A. Oostra is often cited by papers focused on Genetics and Neurodevelopmental Disorders (11 papers), Autism Spectrum Disorder Research (5 papers) and Ubiquitin and proteasome pathways (3 papers). B.A. Oostra collaborates with scholars based in Netherlands, Australia and Belgium. B.A. Oostra's co-authors include Cornelia M. van Duijn, Fakhredin A. Sayed‐Tabatabaei, J. C. M. Witteman, Aaron Isaacs, C.E. Bakker, Edwin Reyniers, Pietro Chiurazzi, R. Willemsen, Giovanni Neri and Maria Grazia Pomponi and has published in prestigious journals such as Nucleic Acids Research, Circulation Research and Journal of Bacteriology.

In The Last Decade

B.A. Oostra

21 papers receiving 1.1k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
B.A. Oostra Netherlands 11 648 531 267 176 136 21 1.1k
Mark J. Daly United States 4 1.5k 2.4× 1.4k 2.6× 443 1.7× 77 0.4× 325 2.4× 4 2.5k
Haydeé Rosas‐Vargas Mexico 17 321 0.5× 432 0.8× 88 0.3× 72 0.4× 49 0.4× 44 946
Raija Miettinen Finland 16 160 0.2× 471 0.9× 65 0.2× 282 1.6× 100 0.7× 23 854
Rose Tobias Canada 15 523 0.8× 303 0.6× 77 0.3× 28 0.2× 168 1.2× 19 979
Douglas E. Crompton Australia 18 343 0.5× 457 0.9× 81 0.3× 152 0.9× 35 0.3× 28 1.4k
Satoru Takahashi Japan 17 281 0.4× 382 0.7× 128 0.5× 26 0.1× 48 0.4× 95 1.1k
Milen Velinov United States 19 476 0.7× 413 0.8× 121 0.5× 53 0.3× 57 0.4× 57 1.0k
Han Yan China 19 416 0.6× 532 1.0× 50 0.2× 39 0.2× 57 0.4× 45 1.1k
Judith Conroy Ireland 16 404 0.6× 473 0.9× 149 0.6× 42 0.2× 77 0.6× 31 1.0k
Hongsheng Gui United States 17 442 0.7× 438 0.8× 36 0.1× 87 0.5× 177 1.3× 56 996

Countries citing papers authored by B.A. Oostra

Since Specialization
Citations

This map shows the geographic impact of B.A. Oostra's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by B.A. Oostra with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites B.A. Oostra more than expected).

Fields of papers citing papers by B.A. Oostra

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by B.A. Oostra. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by B.A. Oostra. The network helps show where B.A. Oostra may publish in the future.

Co-authorship network of co-authors of B.A. Oostra

This figure shows the co-authorship network connecting the top 25 collaborators of B.A. Oostra. A scholar is included among the top collaborators of B.A. Oostra based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with B.A. Oostra. B.A. Oostra is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Coppus, Antonia, Heleen M. Evenhuis, Frank E. Visser, et al.. (2010). Survival in elderly persons with Down syndrome. Pure Amsterdam UMC. 7 indexed citations
2.
Hashem, Vera I., James N. Galloway, M. Mori, et al.. (2009). Ectopic expression of CGG containing mRNA is neurotoxic in mammals. Human Molecular Genetics. 18(13). 2443–2451. 83 indexed citations
3.
Sayed‐Tabatabaei, Fakhredin A., B.A. Oostra, Aaron Isaacs, Cornelia M. van Duijn, & J. C. M. Witteman. (2006). ACE Polymorphisms. Circulation Research. 98(9). 1123–1133. 300 indexed citations
4.
Oostra, B.A., et al.. (2004). Studying the genetics of Hirschsprung's disease: unraveling an oligogenic disorder. Clinical Genetics. 67(1). 6–14. 76 indexed citations
5.
Oostra, B.A. & R. Willemsen. (2003). A fragile balance: FMR1 expression levels. Human Molecular Genetics. 12(suppl 2). R249–R257. 95 indexed citations
6.
Oostra, B.A. & Pietro Chiurazzi. (2001). The fragile X gene and its function. Clinical Genetics. 60(6). 399–408. 39 indexed citations
7.
Diego‐Otero, Yolanda de, et al.. (2000). A fragile X case with an amplification/deletion mosaic pattern. Human Genetics. 106(3). 366–369. 5 indexed citations
8.
Peters, Harm, R. Bollmann, Gundula Leschik, et al.. (1999). Rapid FMR1-protein analysis of fetal blood: an enhancement of prenatal diagnostics. Human Genetics. 105(3). 258–260. 3 indexed citations
9.
Vries, Bert B.A. de, Serieta Mohkamsing, A.M.W. van den Ouweland, et al.. (1999). Screening for the fragile X syndrome among the mentally retarded: a clinical study. The Collaborative Fragile X Study Group.. PubMed. 36(6). 467–70. 32 indexed citations
10.
Chiurazzi, Pietro, et al.. (1998). In Vitro Reactivation of the FMR1 Gene Involved in Fragile X Syndrome. Human Molecular Genetics. 7(1). 109–113. 149 indexed citations
11.
Ouweland, A.M.W. van den, et al.. (1998). [A large-scale diagnostic program for the fragile X syndrome among the mentally handicapped. I. An epidemiologic survey].. PubMed. 142(29). 1666–71. 1 indexed citations
12.
Verhoye, Marleen, Jan Sijbers, R. Frank Kooy, et al.. (1998). <title>MRI as a tool to study brain structure from mouse models for mental retardation</title>. Proceedings of SPIE, the International Society for Optical Engineering/Proceedings of SPIE. 3337. 314–320. 1 indexed citations
13.
Kooy, R. Frank, B.A. Oostra, & P J Willems. (1997). Molecular detection of dynamic mutations. Data Archiving and Networked Services (DANS). 23–53. 6 indexed citations
14.
Fryns, Jean‐Pierre, et al.. (1996). Mental status and FMR1 gene mutation in females. American Journal of Medical Genetics Part A. 64(1). 20. 4 indexed citations
15.
Heutink, Peter, et al.. (1995). A Mutation in Codon-142 in Central Areolar Choroidal Dystrophy. Investigative Ophthalmology & Visual Science. 36. 5 indexed citations
16.
Vries, Bert B.A. de, Hazel M. Robinson, Irene Stolte‐Dijkstra, et al.. (1995). General overgrowth in the fragile X syndrome: variability in the phenotypic expression of the FMR1 gene mutation. Journal of Medical Genetics. 32(10). 764–769. 35 indexed citations
17.
Oostra, B.A., C.E. Bakker, & Edwin Reyniers. (1994). FMR1 Knockout mice: A model to study fragile X mental retardation. The American Journal of Human Genetics. 55. 156 indexed citations
18.
Fryns, Jean‐Pierre, et al.. (1994). Clinical and molecular studies in fragile X patients with a Prader-Willi-like phenotype. American Journal of Medical Genetics Part A. 51. 1 indexed citations
19.
Heutink, Peter, Evert M. van Schothorst, A. Bardoel, et al.. (1994). Further Localization of the Genefor Hereditary Paragangliomasand Evidence for Linkage inUnrelated Families. European Journal of Human Genetics. 2(3). 148–158. 56 indexed citations
20.
Saag, Paul T. van der, et al.. (1989). RFLP for the human retinoic acid receptor gene RAR-β. Nucleic Acids Research. 17(21). 8897–8897. 3 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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