Arthur J. Chu

2.3k total citations
59 papers, 1.9k citations indexed

About

Arthur J. Chu is a scholar working on Molecular Biology, Hematology and Immunology. According to data from OpenAlex, Arthur J. Chu has authored 59 papers receiving a total of 1.9k indexed citations (citations by other indexed papers that have themselves been cited), including 21 papers in Molecular Biology, 16 papers in Hematology and 12 papers in Immunology. Recurrent topics in Arthur J. Chu's work include Blood Coagulation and Thrombosis Mechanisms (16 papers), Immune Response and Inflammation (9 papers) and Hemophilia Treatment and Research (8 papers). Arthur J. Chu is often cited by papers focused on Blood Coagulation and Thrombosis Mechanisms (16 papers), Immune Response and Inflammation (9 papers) and Hemophilia Treatment and Research (8 papers). Arthur J. Chu collaborates with scholars based in United States, China and Canada. Arthur J. Chu's co-authors include Gary J. Blomquist, Lilly Bourguignon, S. A. Rooney, Alasdair M. Gilfillan, Douglas A. Smart, Seamus A. Rooney, Naoko Iida, Lisha Zhang, Hongbo Zhu and Mien‐Chie Hung and has published in prestigious journals such as Journal of Biological Chemistry, Journal of Lipid Research and British Journal of Pharmacology.

In The Last Decade

Arthur J. Chu

59 papers receiving 1.8k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Arthur J. Chu United States 23 670 298 266 218 213 59 1.9k
Hideo Kaneko Japan 31 1.3k 1.9× 210 0.7× 137 0.5× 722 3.3× 142 0.7× 242 3.5k
Akihiko Saito Japan 25 838 1.3× 164 0.6× 116 0.4× 138 0.6× 213 1.0× 65 1.9k
Yoshihito Iuchi Japan 26 822 1.2× 118 0.4× 60 0.2× 244 1.1× 260 1.2× 54 2.1k
Hans Hansen Denmark 29 1.4k 2.2× 316 1.1× 139 0.5× 257 1.2× 166 0.8× 47 2.5k
Glen L. Gunsalus United States 34 1.3k 1.9× 285 1.0× 128 0.5× 114 0.5× 206 1.0× 72 3.7k
Suresh Mishra Canada 29 1.3k 2.0× 171 0.6× 450 1.7× 316 1.4× 287 1.3× 84 2.8k
Ruth Freudinger Germany 31 1.1k 1.7× 433 1.5× 202 0.8× 79 0.4× 184 0.9× 48 2.2k
David T. Thwaites United Kingdom 33 1.1k 1.7× 248 0.8× 229 0.9× 47 0.2× 158 0.7× 63 2.8k
Hitomi Watanabe Japan 23 523 0.8× 218 0.7× 80 0.3× 334 1.5× 73 0.3× 70 1.7k
Janet S. Kerr United States 26 778 1.2× 149 0.5× 351 1.3× 201 0.9× 117 0.5× 66 2.1k

Countries citing papers authored by Arthur J. Chu

Since Specialization
Citations

This map shows the geographic impact of Arthur J. Chu's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Arthur J. Chu with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Arthur J. Chu more than expected).

Fields of papers citing papers by Arthur J. Chu

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Arthur J. Chu. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Arthur J. Chu. The network helps show where Arthur J. Chu may publish in the future.

Co-authorship network of co-authors of Arthur J. Chu

This figure shows the co-authorship network connecting the top 25 collaborators of Arthur J. Chu. A scholar is included among the top collaborators of Arthur J. Chu based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Arthur J. Chu. Arthur J. Chu is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Chu, Arthur J.. (2004). Biochemical Strategies to Anticoagulation: A Comparative Overview. Current Vascular Pharmacology. 2(3). 199–228. 13 indexed citations
2.
Chu, Arthur J.. (2004). Tissue factor upregulation drives a thrombosis–inflammation circuit in relation to cardiovascular complications. Cell Biochemistry and Function. 24(2). 173–192. 34 indexed citations
3.
4.
Chu, Arthur J., et al.. (2003). Novel anticoagulant polyethylenimine: Inhibition of thrombin-catalyzed fibrin formation. Archives of Biochemistry and Biophysics. 415(1). 101–108. 14 indexed citations
5.
Chu, Arthur J. & Suresh T. Mathews. (2003). Anticoagulant potential of an antibody against factor VII. Journal of Surgical Research. 114(1). 37–41. 1 indexed citations
6.
Chu, Arthur J., et al.. (2002). Novel anticoagulant activity of polybrene: inhibition of monocytic tissue factor hypercoagulation following bacterial endotoxin induction. Blood Coagulation & Fibrinolysis. 13(2). 123–128. 5 indexed citations
7.
Chu, Arthur J., et al.. (2002). Possible role of Marcks in the cellular modulation of monocytic tissue factor‐initiated hypercoagulation. British Journal of Haematology. 118(2). 569–576. 5 indexed citations
8.
Chu, Arthur J., et al.. (2001). Antimicrobial Peptide Buforin I Inhibits Tissue Factor-Initiated Coagulation. Archives of Biochemistry and Biophysics. 392(1). 3–7. 9 indexed citations
9.
Chu, Arthur J., et al.. (2000). IV. Anticoagulant Activity of Compound 48/80: Inhibition of Factor VII Activation in Leukemia THP-1 Monocytes. Journal of Cardiovascular Pharmacology. 36(5). 649–655. 10 indexed citations
10.
Chu, Arthur J., et al.. (2000). III. Instantaneous Inhibition by Compound 48/80 of Tissue Factor-Initiated Extrinsic Coagulation Is Mediated by the Downregulation of Factor VII Activation. Archives of Biochemistry and Biophysics. 377(2). 357–365. 7 indexed citations
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Chu, Arthur J. & J.K. Prasad. (1998). Antagonism by IL-4 and IL-10 of Endotoxin-Induced Tissue Factor Activation in Monocytic THP-1 Cells: Activating Role of CD14 Ligation. Journal of Surgical Research. 80(1). 80–87. 22 indexed citations
14.
Chu, Arthur J., et al.. (1997). Antagonism by ethanol of endotoxin-induced tissue factor activation in relation to the depressed endotoxin binding to monocyte-like U937 cells. Cell Biochemistry and Function. 15(4). 271–281. 16 indexed citations
15.
Moffat, Frederick L., Zhiming Li, Michael D. Peck, et al.. (1996). Supplemental L-arginine HCI augments bacterial phagocytosis in human polymorphonuclear leukocytes. Journal of Cellular Physiology. 168(1). 26–33. 42 indexed citations
16.
Chu, Arthur J., et al.. (1994). Lidocaine inhibits choline uptake and phosphatidylcholine biosynthesis in human leukemic monocyte‐like U937 cells. Cell Biochemistry and Function. 12(2). 89–98. 6 indexed citations
17.
Chu, Arthur J.. (1994). Mechanism by which ethanol inhibits phosphatidylcholine biosynthesis in human leukemic monocyte‐like U937 cells. Cell Biochemistry and Function. 12(1). 45–55. 6 indexed citations
18.
Chu, Arthur J.. (1994). Differential regulations of phosphatidylcholine biosynthesis in U937 cells by inhibitors of protein and tyrosine kinases. International Journal of Biochemistry. 26(2). 189–193. 6 indexed citations
19.
Chu, Arthur J.. (1992). Bacterial lipopolysaccharide stimulates phospholipid synthesis and phosphatidylcholine breakdown in cultured human leukemia monocytic THP-1 cells. International Journal of Biochemistry. 24(2). 317–323. 16 indexed citations
20.
Chu, Arthur J.. (1991). Inhibition of endotoxin-induced monocytic procoagulant activity by n-alcohols and anesthetics. Comparative Biochemistry and Physiology Part C Comparative Pharmacology. 99(3). 451–456. 6 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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