Ariel Feiglin

882 total citations
20 papers, 369 citations indexed

About

Ariel Feiglin is a scholar working on Molecular Biology, Genetics and Pulmonary and Respiratory Medicine. According to data from OpenAlex, Ariel Feiglin has authored 20 papers receiving a total of 369 indexed citations (citations by other indexed papers that have themselves been cited), including 12 papers in Molecular Biology, 4 papers in Genetics and 3 papers in Pulmonary and Respiratory Medicine. Recurrent topics in Ariel Feiglin's work include RNA regulation and disease (4 papers), Bioinformatics and Genomic Networks (4 papers) and Genomics and Rare Diseases (2 papers). Ariel Feiglin is often cited by papers focused on RNA regulation and disease (4 papers), Bioinformatics and Genomic Networks (4 papers) and Genomics and Rare Diseases (2 papers). Ariel Feiglin collaborates with scholars based in Israel, United States and Germany. Ariel Feiglin's co-authors include Ouri Schwob, Tal Stern, Ofra Benny, Michael Berger, Erez Y. Levanon, Eli Kopel, Yanay Ofran, Eli Eisenberg, Ron Unger and Isaac S. Kohane and has published in prestigious journals such as Nature Communications, Bioinformatics and PLoS ONE.

In The Last Decade

Ariel Feiglin

19 papers receiving 365 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Ariel Feiglin Israel 13 211 55 51 50 41 20 369
Pablo Domizi United States 12 254 1.2× 57 1.0× 52 1.0× 25 0.5× 45 1.1× 21 413
David Ketelsen United States 8 285 1.4× 113 2.1× 51 1.0× 76 1.5× 43 1.0× 10 434
Gillian Fitzgerald Switzerland 5 169 0.8× 38 0.7× 30 0.6× 78 1.6× 23 0.6× 7 356
Eriko Ohnishi Japan 4 261 1.2× 54 1.0× 16 0.3× 26 0.5× 27 0.7× 6 409
Wendy L. Roberts United States 13 199 0.9× 51 0.9× 83 1.6× 56 1.1× 17 0.4× 15 473
Astrid Rohrbeck Germany 15 292 1.4× 79 1.4× 86 1.7× 76 1.5× 29 0.7× 30 482
Nicholas S. Davis United States 8 335 1.6× 103 1.9× 59 1.2× 132 2.6× 51 1.2× 12 503
Ping Xiang United States 15 366 1.7× 49 0.9× 43 0.8× 34 0.7× 15 0.4× 37 535

Countries citing papers authored by Ariel Feiglin

Since Specialization
Citations

This map shows the geographic impact of Ariel Feiglin's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ariel Feiglin with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ariel Feiglin more than expected).

Fields of papers citing papers by Ariel Feiglin

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Ariel Feiglin. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ariel Feiglin. The network helps show where Ariel Feiglin may publish in the future.

Co-authorship network of co-authors of Ariel Feiglin

This figure shows the co-authorship network connecting the top 25 collaborators of Ariel Feiglin. A scholar is included among the top collaborators of Ariel Feiglin based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Ariel Feiglin. Ariel Feiglin is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Feiglin, Ariel, et al.. (2024). DNA and RNA base editors can correct the majority of pathogenic single nucleotide variants. npj Genomic Medicine. 9(1). 16–16. 9 indexed citations
2.
Feiglin, Ariel, et al.. (2023). TREM2 has a significant, gender-specific, effect on human obesity. Scientific Reports. 13(1). 482–482. 9 indexed citations
3.
Guarducci, Cristina, Simona Cristea, Avery Feit, et al.. (2023). Abstract GS3-07: GS3-07 Clonal evolution and mechanisms of acquired resistance to CDK4/6 inhibitors in ER-wild type and ER-mutant breast cancer. Cancer Research. 83(5_Supplement). GS3–7. 3 indexed citations
4.
Qiu, Xintao, Avery Feit, Ariel Feiglin, et al.. (2021). CoBRA: Containerized Bioinformatics Workflow for Reproducible ChIP/ATAC-Seq Analysis. Genomics Proteomics & Bioinformatics. 19(4). 652–661. 16 indexed citations
5.
Buchumenski, Ilana, Shalom Hillel Roth, Eli Kopel, et al.. (2021). Global quantification exposes abundant low-level off-target activity by base editors. Genome Research. 31(12). 2354–2361. 14 indexed citations
6.
Ben‐Shmuel, Aviad, Noah Joseph, Omer Levy, et al.. (2021). Targeting the actin nucleation promoting factor WASp provides a therapeutic approach for hematopoietic malignancies. Nature Communications. 12(1). 5581–5581. 13 indexed citations
7.
Kopel, Eli, Ariel Feiglin, Tamar Sofer, et al.. (2020). Increased RNA editing in maternal immune activation model of neurodevelopmental disease. Nature Communications. 11(1). 5236–5236. 27 indexed citations
8.
Jagtap, Smita, Jessica M. Thanos, Ting Fu, et al.. (2019). Aberrant mitochondrial function in patient-derived neural cells from CDKL5 deficiency disorder and Rett syndrome. Human Molecular Genetics. 28(21). 3625–3636. 14 indexed citations
9.
Guarducci, Cristina, Agostina Nardone, Ariel Feiglin, et al.. (2019). Abstract PD7-12: Inhibition of CDK7 overcomes resistance to CDK4/6 inhibitors in hormone receptor positive breast cancer cells. Cancer Research. 79(4_Supplement). PD7–12. 15 indexed citations
10.
Kopel, Eli, Ariel Feiglin, Dror Avni, et al.. (2018). Decreased A-to-I RNA editing as a source of keratinocytes' dsRNA in psoriasis. RNA. 24(6). 828–840. 34 indexed citations
11.
Feiglin, Ariel, et al.. (2018). Tissue necrosis and its role in cancer progression. Oncogene. 38(11). 1920–1935. 101 indexed citations
12.
Feiglin, Ariel, Bryce K. Allen, Isaac S. Kohane, & Sek Won Kong. (2017). Comprehensive Analysis of Tissue-wide Gene Expression and Phenotype Data Reveals Tissues Affected in Rare Genetic Disorders. Cell Systems. 5(2). 140–148.e2. 9 indexed citations
13.
Nyska, Abraham, et al.. (2017). A novel Fer/FerT targeting compound selectively evokes metabolic stress and necrotic death in malignant cells. Nature Communications. 8(1). 940–940. 19 indexed citations
14.
Somekh, Judith, Mor Peleg, Alal Eran, et al.. (2016). A model-driven methodology for exploring complex disease comorbidities applied to autism spectrum disorder and inflammatory bowel disease. Journal of Biomedical Informatics. 63. 366–378. 12 indexed citations
15.
Heimer, Gali, Yair Sadaka, Ariel Feiglin, et al.. (2015). CAOS—Episodic Cerebellar Ataxia, Areflexia, Optic Atrophy, and Sensorineural Hearing Loss. Journal of Child Neurology. 30(13). 1749–1756. 42 indexed citations
16.
Feiglin, Ariel, et al.. (2014). Co-expression and co-localization of hub proteins and their partners are encoded in protein sequence. Molecular BioSystems. 10(4). 787–794. 1 indexed citations
17.
Feiglin, Ariel, et al.. (2012). Static network structure can be used to model the phenotypic effects of perturbations in regulatory networks. Bioinformatics. 28(21). 2811–2818. 16 indexed citations
18.
Feiglin, Ariel, John Moult, Byung Kook Lee, Yanay Ofran, & Ron Unger. (2012). Neighbor Overlap Is Enriched in the Yeast Interaction Network: Analysis and Implications. PLoS ONE. 7(6). e39662–e39662. 1 indexed citations
19.
20.
Oates, Jennifer, et al.. (1996). Vocal characteristics and response to levodopa in Parkinson’s disease. Movement Disorders. 11. 13 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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