Arati B. Kamath

1.4k total citations
15 papers, 1.2k citations indexed

About

Arati B. Kamath is a scholar working on Immunology, Infectious Diseases and Health, Toxicology and Mutagenesis. According to data from OpenAlex, Arati B. Kamath has authored 15 papers receiving a total of 1.2k indexed citations (citations by other indexed papers that have themselves been cited), including 12 papers in Immunology, 5 papers in Infectious Diseases and 3 papers in Health, Toxicology and Mutagenesis. Recurrent topics in Arati B. Kamath's work include T-cell and B-cell Immunology (6 papers), Tuberculosis Research and Epidemiology (5 papers) and Immune Cell Function and Interaction (5 papers). Arati B. Kamath is often cited by papers focused on T-cell and B-cell Immunology (6 papers), Tuberculosis Research and Epidemiology (5 papers) and Immune Cell Function and Interaction (5 papers). Arati B. Kamath collaborates with scholars based in United States. Arati B. Kamath's co-authors include Lisheng Wang, Hiranmoy Das, Jack F. Bukowski, Samuel M. Behar, Prakash Nagarkatti, Mitzi Nagarkatti, Joshua S. Woodworth, Lin Li, Hajer Debbabi and Jennifer Alt and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Clinical Investigation and The Journal of Experimental Medicine.

In The Last Decade

Arati B. Kamath

15 papers receiving 1.2k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Arati B. Kamath United States 14 752 276 236 228 186 15 1.2k
Emmanuel Stephen‐Victor France 21 647 0.9× 221 0.8× 386 1.6× 154 0.7× 125 0.7× 42 1.4k
Emilie Doz France 12 547 0.7× 327 1.2× 286 1.2× 276 1.2× 88 0.5× 16 1.0k
M L Lohmann-Matthes Germany 16 701 0.9× 120 0.4× 304 1.3× 208 0.9× 177 1.0× 25 1.4k
Yukiyoshi Yanagihara Japan 23 751 1.0× 90 0.3× 261 1.1× 76 0.3× 148 0.8× 117 1.9k
A. Gallagher United Kingdom 10 375 0.5× 170 0.6× 293 1.2× 144 0.6× 509 2.7× 12 1.2k
Julio Enrique Castañeda‐Delgado Mexico 23 305 0.4× 329 1.2× 480 2.0× 250 1.1× 77 0.4× 64 1.3k
Evan O. Gregg United Kingdom 16 174 0.2× 129 0.5× 133 0.6× 183 0.8× 44 0.2× 28 666
Pedro Moura‐Alves Germany 15 419 0.6× 355 1.3× 433 1.8× 266 1.2× 56 0.3× 20 981
Chantal Martin France 11 194 0.3× 215 0.8× 174 0.7× 179 0.8× 67 0.4× 20 778
Tova Meshulam United States 22 327 0.4× 323 1.2× 502 2.1× 229 1.0× 98 0.5× 46 1.3k

Countries citing papers authored by Arati B. Kamath

Since Specialization
Citations

This map shows the geographic impact of Arati B. Kamath's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Arati B. Kamath with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Arati B. Kamath more than expected).

Fields of papers citing papers by Arati B. Kamath

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Arati B. Kamath. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Arati B. Kamath. The network helps show where Arati B. Kamath may publish in the future.

Co-authorship network of co-authors of Arati B. Kamath

This figure shows the co-authorship network connecting the top 25 collaborators of Arati B. Kamath. A scholar is included among the top collaborators of Arati B. Kamath based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Arati B. Kamath. Arati B. Kamath is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

15 of 15 papers shown
1.
Kamath, Arati B., Joshua S. Woodworth, & Samuel M. Behar. (2006). Antigen-Specific CD8+ T Cells and the Development of Central Memory during Mycobacterium tuberculosis Infection. The Journal of Immunology. 177(9). 6361–6369. 82 indexed citations
2.
Debbabi, Hajer, Shamik Ghosh, Arati B. Kamath, et al.. (2005). Primary type II alveolar epithelial cells present microbial antigens to antigen-specific CD4+T cells. American Journal of Physiology-Lung Cellular and Molecular Physiology. 289(2). L274–L279. 85 indexed citations
3.
Kamath, Arati B. & Samuel M. Behar. (2005). Anamnestic Responses of Mice followingMycobacterium tuberculosisInfection. Infection and Immunity. 73(9). 6110–6118. 20 indexed citations
4.
Kamath, Arati B., et al.. (2004). Cytolytic CD8+ T Cells Recognizing CFP10 Are Recruited to the Lung after Mycobacterium tuberculosis Infection. The Journal of Experimental Medicine. 200(11). 1479–1489. 101 indexed citations
6.
Kamath, Arati B., Lisheng Wang, Hiranmoy Das, et al.. (2003). Antigens in tea-beverage prime human Vγ2Vδ2 T cellsin vitroandin vivofor memory and nonmemory antibacterial cytokine responses. Proceedings of the National Academy of Sciences. 100(10). 6009–6014. 90 indexed citations
7.
Kamath, Arati B., Jennifer Alt, Hajer Debbabi, & Samuel M. Behar. (2003). Toll-Like Receptor 4-Defective C3H/HeJ Mice Are Not More Susceptible than Other C3H Substrains to Infection withMycobacterium tuberculosis. Infection and Immunity. 71(7). 4112–4118. 70 indexed citations
8.
Wang, Lisheng, Hiranmoy Das, Arati B. Kamath, Li Lin, & Jack F. Bukowski. (2002). Human Vγ2Vδ2 T Cells Augment Migration-Inhibitory Factor Secretion and Counteract the Inhibitory Effect of Glucocorticoids on IL-1β and TNF-α Production. The Journal of Immunology. 168(10). 4889–4896. 9 indexed citations
9.
Wang, Lisheng, Hiranmoy Das, Arati B. Kamath, & Jack F. Bukowski. (2001). Human Vγ2Vδ2 T Cells Produce IFN-γ and TNF-α with an On/Off/On Cycling Pattern in Response to Live Bacterial Products. The Journal of Immunology. 167(11). 6195–6201. 70 indexed citations
10.
Wang, Lisheng, Arati B. Kamath, Hiranmoy Das, Li Lin, & Jack F. Bukowski. (2001). Antibacterial effect of human Vγ2Vδ2 T cells in vivo. Journal of Clinical Investigation. 108(9). 1349–1357. 130 indexed citations
11.
Wang, Lisheng, Arati B. Kamath, Hiranmoy Das, Lin Li, & Jack F. Bukowski. (2001). Antibacterial effect of human Vγ2Vδ2 T cells in vivo. Journal of Clinical Investigation. 108(9). 1349–1357. 122 indexed citations
12.
Das, Hiranmoy, Lisheng Wang, Arati B. Kamath, & Jack F. Bukowski. (2001). Vγ2Vδ2 T-cell receptor–mediated recognition of aminobisphosphonates. Blood. 98(5). 1616–1618. 156 indexed citations
13.
Kamath, Arati B., Iris Camacho, Prakash Nagarkatti, & Mitzi Nagarkatti. (1999). Role of Fas–Fas Ligand Interactions in 2,3,7,8-Tetrachlorodibenzo- p-dioxin (TCDD)-Induced Immunotoxicity: Increased Resistance of Thymocytes from Fas-Deficient (lpr) and Fas Ligand-Defective (gld) Mice to TCDD-Induced Toxicity. Toxicology and Applied Pharmacology. 160(2). 141–155. 59 indexed citations
14.
Kamath, Arati B. & Prakash Nagarkatti. (1998). Characterization of Phenotypic Alterations Induced by 2,3,7,8-Tetrachlorodibenzo-p-dioxin on Thymocytesin Vivoand Its Effect on Apoptosis. Toxicology and Applied Pharmacology. 150(1). 117–124. 61 indexed citations
15.
Kamath, Arati B., Huimin Xu, Prakash Nagarkatti, & Mitzi Nagarkatti. (1997). Evidence for the Induction of Apoptosis in Thymocytes by 2,3,7,8-Tetrachlorodibenzo-p-dioxinin Vivo. Toxicology and Applied Pharmacology. 142(2). 367–377. 126 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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