Anton Arkhipov
- Molecular Biology top 5%
- Oncology top 5%
- Cell Biology top 2%
- Materials Chemistry top 10%
- Atomic and Molecular Physics, and Optics top 5%
- Co-authors
- Klaus SchultenPeter L. FreddolinoYibing ShanDavid E. ShawJohn KuriyanAmy Y. ShihEric T. KimDavid E. Wemmer
- Topics
- Neural dynamics and brain function (17 papers)Lipid Membrane Structure and Behavior (9 papers)Protein Structure and Dynamics (8 papers)
- Partner nations
- United StatesJapanSwitzerland
In The Last Decade
Anton Arkhipov
41 papers receiving 3.6k citations
Hit Papers
Peers
Comparison fields: 5 of 137
- Molecular Biology 2.4k
- Oncology 497
- Cell Biology 497
- Materials Chemistry 445
- Atomic and Molecular Physics, and Optics 435
Countries citing papers authored by Anton Arkhipov
This map shows the geographic impact of Anton Arkhipov's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Anton Arkhipov with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Anton Arkhipov more than expected).
Fields of papers citing papers by Anton Arkhipov
This network shows the impact of papers produced by Anton Arkhipov. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Anton Arkhipov. The network helps show where Anton Arkhipov may publish in the future.
Co-authorship network of co-authors of Anton Arkhipov
This figure shows the co-authorship network connecting the top 25 collaborators of Anton Arkhipov. A scholar is included among the top collaborators of Anton Arkhipov based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Anton Arkhipov. Anton Arkhipov is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 0 | |
| 2 | 2 | |
| 3 | 3 | |
| 4 | 6 | |
| 5 | 1 | |
| 6 | 23 | |
| 7 | 29 | |
| 8 | 31 | |
| 9 | 144 | |
| 10 | 34 | |
| 11 | 388 | |
| 12 | Oncogenic Mutations Counteract Intrinsic Disorder in the EGFR Kinase and Promote Receptor Dimerizationbreakdown → | 276 |
| 13 | 83 | |
| 14 | 109 | |
| 15 | 198 | |
| 16 | 11 | |
| 17 | 98 | |
| 18 | 289 | |
| 19 | 79 | |
| 20 | 58 |
About Anton Arkhipov
Anton Arkhipov is a scholar working on Biophysics, Cognitive Neuroscience and Cellular and Molecular Neuroscience, having authored 44 papers that have together received 3.7k indexed citations. Recurring topics across this work include Neural dynamics and brain function (17 papers), Lipid Membrane Structure and Behavior (9 papers) and Protein Structure and Dynamics (8 papers). The work is most often cited by research in Structural Biology (52 citations), Molecular Biology (2.4k citations) and Cell Biology (497 citations). Anton Arkhipov has collaborated with scholars based in United States, Japan and Switzerland. Frequent co-authors include Klaus Schulten, Peter L. Freddolino, Yibing Shan, David E. Shaw, John Kuriyan, Amy Y. Shih, Eric T. Kim, Klaus Schulten, David E. Wemmer and Nicholas Endres. Their work appears in journals such as Cell, Proceedings of the National Academy of Sciences and Nature Communications.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.