Anika Kasprick

678 total citations
18 papers, 379 citations indexed

About

Anika Kasprick is a scholar working on Pathology and Forensic Medicine, Genetics and Rheumatology. According to data from OpenAlex, Anika Kasprick has authored 18 papers receiving a total of 379 indexed citations (citations by other indexed papers that have themselves been cited), including 16 papers in Pathology and Forensic Medicine, 13 papers in Genetics and 12 papers in Rheumatology. Recurrent topics in Anika Kasprick's work include Autoimmune Bullous Skin Diseases (16 papers), Coagulation, Bradykinin, Polyphosphates, and Angioedema (13 papers) and Urticaria and Related Conditions (11 papers). Anika Kasprick is often cited by papers focused on Autoimmune Bullous Skin Diseases (16 papers), Coagulation, Bradykinin, Polyphosphates, and Angioedema (13 papers) and Urticaria and Related Conditions (11 papers). Anika Kasprick collaborates with scholars based in Germany, Netherlands and Japan. Anika Kasprick's co-authors include Ralf J. Ludwig, Katja Bieber, Detlef Zillikens, Frank Petersen, Xinhua Yu, Enno Schmidt, Kathrin Kalies, Hendri H. Pas, Jennifer E. Hundt and Franziska Schulze and has published in prestigious journals such as Nature Communications, Scientific Reports and British Journal of Pharmacology.

In The Last Decade

Anika Kasprick

17 papers receiving 379 citations

Peers

Anika Kasprick

PFM

GENET

RHEUM

IMMUN

DERMA

Yask Gupta Germany

Franziska Schulze Germany

Mong-Shang Lin United States

Hana Jedličková Czechia

Vera Teixeira Portugal

Hua Qian China

L. Gusdorf France

Renee H. Grau United States

Nathalie Gunera-Saad France

Atsuko Fukuyasu Japan

Yask Gupta Germany

Anika Kasprick

201 ×0.8

PFM

174 ×0.8

GENET

150 ×0.8

RHEUM

35 ×0.5

IMMUN

22 ×0.5

DERMA

Citations per year, relative to Anika Kasprick Anika Kasprick (= 1×) peers Yask Gupta

Countries citing papers authored by Anika Kasprick

Since Specialization

Citations

This map shows the geographic impact of Anika Kasprick's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Anika Kasprick with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Anika Kasprick more than expected).

Fields of papers citing papers by Anika Kasprick

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Anika Kasprick. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Anika Kasprick. The network helps show where Anika Kasprick may publish in the future.

Co-authorship network of co-authors of Anika Kasprick

This figure shows the co-authorship network connecting the top 25 collaborators of Anika Kasprick. A scholar is included among the top collaborators of Anika Kasprick based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Anika Kasprick. Anika Kasprick is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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18 of 18 papers shown

Kasprick, Anika, et al.. (2024). Dexamethasone’s Clinical Efficacy in Experimental Autoimmune Pancreatitis Correlates with a Unique Transcriptomic Signature, Whilst Kinase Inhibitors Are Not Effective. Biomedicines. 12(11). 2480–2480.

Emtenani, Shirin, Manuela Pigors, Anika Kasprick, et al.. (2022). Cutaneous kinase activity correlates with treatment outcomes following PI3K delta inhibition in mice with experimental pemphigoid diseases. Frontiers in Immunology. 13. 865241–865241. 6 indexed citations

Pipi, Elena, Anika Kasprick, Hiroaki Iwata, et al.. (2021). Multiple Modes of Action Mediate the Therapeutic Effect of Intravenous IgG in Experimental Epidermolysis Bullosa Acquisita. Journal of Investigative Dermatology. 142(6). 1552–1564.e8. 4 indexed citations

Kasprick, Anika, Nancy Ernst, Katharina Boch, et al.. (2021). Topical Application of the PI3Kβ-Selective Small Molecule Inhibitor TGX-221 Is an Effective Treatment Option for Experimental Epidermolysis Bullosa Acquisita. Frontiers in Medicine. 8. 713312–713312. 6 indexed citations

Hiroyasu, Sho, Matthew R. Zeglinski, Hongyan Zhao, et al.. (2021). Granzyme B inhibition reduces disease severity in autoimmune blistering diseases. Nature Communications. 12(1). 302–302. 70 indexed citations

Kasprick, Anika, Shirin Emtenani, Khalaf Kridin, et al.. (2020). Identification of novel therapeutic targets for blocking acantholysis in pemphigus. British Journal of Pharmacology. 177(22). 5114–5130. 23 indexed citations

Kasprick, Anika, Bryan Smith, Penelope Ward, et al.. (2020). Treatment with anti‐neonatal Fc receptor (FcRn) antibody ameliorates experimental epidermolysis bullosa acquisita in mice. British Journal of Pharmacology. 177(10). 2381–2392. 18 indexed citations

Kasprick, Anika, Katja Bieber, & Ralf J. Ludwig. (2019). Drug Discovery for Pemphigoid Diseases. Current Protocols in Pharmacology. 84(1). e55–e55. 19 indexed citations

Samavedam, Unni, Anika Kasprick, Katja Bieber, et al.. (2018). Whole-Genome Expression Profiling in Skin Reveals SYK As a Key Regulator of Inflammation in Experimental Epidermolysis Bullosa Acquisita. Frontiers in Immunology. 9. 249–249. 26 indexed citations

10.

Yu, Xinhua, Anika Kasprick, Karin Hartmann, & Frank Petersen. (2018). The Role of Mast Cells in Autoimmune Bullous Dermatoses. Frontiers in Immunology. 9. 386–386. 7 indexed citations

11.

Koga, Hiroshi, Anika Kasprick, Rosa López-Gigosos, et al.. (2018). Therapeutic Effect of a Novel Phosphatidylinositol-3-Kinase δ Inhibitor in Experimental Epidermolysis Bullosa Acquisita. Frontiers in Immunology. 9. 1558–1558. 21 indexed citations

12.

Bieber, Katja, Shijie Sun, Mareike Witte, et al.. (2017). Regulatory T Cells Suppress Inflammation and Blistering in Pemphigoid Diseases. Frontiers in Immunology. 8. 1628–1628. 37 indexed citations

13.

Koga, Hiroshi, Andreas Recke, Gestur Vidarsson, et al.. (2016). PDE4 Inhibition as Potential Treatment of Epidermolysis Bullosa Acquisita. Journal of Investigative Dermatology. 136(11). 2211–2220. 18 indexed citations

14.

Hirose, Misa, Anika Kasprick, Franziska Schulze, et al.. (2016). Reduced Skin Blistering in Experimental Epidermolysis Bullosa Acquisita After Anti-TNF Treatment. Molecular Medicine. 22(1). 918–926. 26 indexed citations

15.

Bieber, Katja, Mareike Witte, Shijie Sun, et al.. (2016). T cells mediate autoantibody-induced cutaneous inflammation and blistering in epidermolysis bullosa acquisita. Scientific Reports. 6(1). 38357–38357. 33 indexed citations

16.

Yu, Xinhua, Anika Kasprick, & Frank Petersen. (2015). Revisiting the role of mast cells in autoimmunity. Autoimmunity Reviews. 14(9). 751–759. 23 indexed citations

17.

Kasprick, Anika, Xinhua Yu, Julia Scholten, et al.. (2015). Conditional depletion of mast cells has no impact on the severity of experimental epidermolysis bullosa acquisita. European Journal of Immunology. 45(5). 1462–1470. 24 indexed citations

18.

Gupta, Yask, Steffen Möller, Unni Samavedam, et al.. (2015). The retinoid‐related orphan receptor alpha is essential for the end‐stage effector phase of experimental epidermolysis bullosa acquisita. The Journal of Pathology. 237(1). 111–122. 18 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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