Andreas Wyttenbach

7.6k total citations
49 papers, 2.8k citations indexed

About

Andreas Wyttenbach is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Cell Biology. According to data from OpenAlex, Andreas Wyttenbach has authored 49 papers receiving a total of 2.8k indexed citations (citations by other indexed papers that have themselves been cited), including 32 papers in Molecular Biology, 26 papers in Cellular and Molecular Neuroscience and 9 papers in Cell Biology. Recurrent topics in Andreas Wyttenbach's work include Genetic Neurodegenerative Diseases (24 papers), Mitochondrial Function and Pathology (23 papers) and Muscle Physiology and Disorders (7 papers). Andreas Wyttenbach is often cited by papers focused on Genetic Neurodegenerative Diseases (24 papers), Mitochondrial Function and Pathology (23 papers) and Muscle Physiology and Disorders (7 papers). Andreas Wyttenbach collaborates with scholars based in United Kingdom, Switzerland and United States. Andreas Wyttenbach's co-authors include Sarah Hands, David C. Rubinsztein, Julia Rankin, Y. Narain, Aviva M. Tolkovsky, Robert A. Furlong, Jenny Carmichael, Jina Swartz, V. Hugh Perry and Jessica L. Teeling and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Journal of Neuroscience.

In The Last Decade

Andreas Wyttenbach

48 papers receiving 2.8k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Andreas Wyttenbach United Kingdom 29 1.8k 1.4k 477 436 359 49 2.8k
Kyung‐Tai Min United States 25 1.6k 0.9× 864 0.6× 211 0.4× 306 0.7× 317 0.9× 44 2.9k
Pedro Fernández-Fúnez United States 20 1.5k 0.8× 865 0.6× 225 0.5× 447 1.0× 495 1.4× 40 2.1k
Huidy Shu United States 9 1.1k 0.6× 857 0.6× 724 1.5× 347 0.8× 402 1.1× 10 2.5k
Ko Miyoshi Japan 28 1.3k 0.7× 720 0.5× 267 0.6× 638 1.5× 345 1.0× 47 2.5k
R. Grace Zhai United States 31 2.3k 1.3× 1.5k 1.1× 174 0.4× 1.1k 2.6× 328 0.9× 66 3.8k
Udai Bhan Pandey United States 27 2.2k 1.2× 804 0.6× 1.6k 3.3× 312 0.7× 285 0.8× 55 3.6k
P. Rusmini Italy 33 1.6k 0.9× 702 0.5× 961 2.0× 695 1.6× 360 1.0× 59 2.9k
Dominik Paquet Germany 15 1.4k 0.8× 390 0.3× 369 0.8× 310 0.7× 482 1.3× 26 2.0k
Aaron Voigt Germany 24 1.0k 0.6× 526 0.4× 742 1.6× 329 0.8× 362 1.0× 44 1.9k
Alyson Peel United States 21 1.7k 0.9× 988 0.7× 176 0.4× 800 1.8× 702 2.0× 23 3.5k

Countries citing papers authored by Andreas Wyttenbach

Since Specialization
Citations

This map shows the geographic impact of Andreas Wyttenbach's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Andreas Wyttenbach with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Andreas Wyttenbach more than expected).

Fields of papers citing papers by Andreas Wyttenbach

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Andreas Wyttenbach. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Andreas Wyttenbach. The network helps show where Andreas Wyttenbach may publish in the future.

Co-authorship network of co-authors of Andreas Wyttenbach

This figure shows the co-authorship network connecting the top 25 collaborators of Andreas Wyttenbach. A scholar is included among the top collaborators of Andreas Wyttenbach based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Andreas Wyttenbach. Andreas Wyttenbach is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Quraishe, Shmma, et al.. (2015). Selective and compartmentalized myelin expression of HspB5. Neuroscience. 316. 130–142. 3 indexed citations
2.
Green, Edward W., Leonor Miller‐Fleming, Sarah Hands, et al.. (2013). DJ-1 modulates aggregation and pathogenesis in models of Huntington's disease. Human Molecular Genetics. 23(3). 755–766. 39 indexed citations
3.
Hands, Sarah, et al.. (2011). In Vitro and in Vivo Aggregation of a Fragment of Huntingtin Protein Directly Causes Free Radical Production. Journal of Biological Chemistry. 286(52). 44512–44520. 93 indexed citations
4.
Sinadinos, Christopher, Catherine M. Cowan, Andreas Wyttenbach, & Amritpal Mudher. (2011). Increased throughput assays of locomotor dysfunction in Drosophila larvae. Journal of Neuroscience Methods. 203(2). 325–334. 19 indexed citations
5.
Sajjad, Muhammad, et al.. (2010). Heat Shock Proteins: Therapeutic Drug Targets for Chronic Neurodegeneration?. Current Pharmaceutical Biotechnology. 11(2). 198–215. 23 indexed citations
6.
Hands, Sarah & Andreas Wyttenbach. (2010). Neurotoxic protein oligomerisation associated with polyglutamine diseases. Acta Neuropathologica. 120(4). 419–437. 56 indexed citations
7.
Tolkovsky, Aviva M. & Andreas Wyttenbach. (2009). Differential Phosphoprotein Labelling (DIPPL) Using 32P and 33P. Methods in molecular biology. 527. 21–29. 1 indexed citations
8.
Sinadinos, Christopher, et al.. (2009). Live axonal transport disruption by mutant huntingtin fragments in Drosophila motor neuron axons. Neurobiology of Disease. 34(2). 389–395. 48 indexed citations
9.
King, Matthew, et al.. (2008). Cytoplasmic Inclusions of Htt Exon1 Containing an Expanded Polyglutamine Tract Suppress Execution of Apoptosis in Sympathetic Neurons. Journal of Neuroscience. 28(53). 14401–14415. 15 indexed citations
10.
Wyttenbach, Andreas, Sarah Hands, Matthew King, Karen Lipkow, & Aviva M. Tolkovsky. (2008). Amelioration of protein misfolding disease by rapamycin: Translation or autophagy?. Autophagy. 4(4). 542–545. 28 indexed citations
11.
King, Matthew, et al.. (2008). Rapamycin Inhibits Polyglutamine Aggregation Independently of Autophagy by Reducing Protein Synthesis. Molecular Pharmacology. 73(4). 1052–1063. 101 indexed citations
12.
Wyttenbach, Andreas, et al.. (2004). Study of Gene Flow Through a Hybrid Zone in the Common Shrew (Sorex Araneus) Using Microsatellites. Hereditas. 125(2-3). 159–168. 17 indexed citations
13.
Yung, Hong Wa, Andreas Wyttenbach, & Aviva M. Tolkovsky. (2004). Aggravation of necrotic death of glucose-deprived cells by the MEK1 inhibitors U0126 and PD184161 through depletion of ATP. Biochemical Pharmacology. 68(2). 351–360. 34 indexed citations
14.
Wyttenbach, Andreas. (2004). Role of Heat Shock Proteins During Polyglutamine Neurodegeneration: Mechanisms and Hypothesis. Journal of Molecular Neuroscience. 23(1-2). 69–96. 57 indexed citations
15.
Wyttenbach, Andreas. (2002). Heat shock protein 27 prevents cellular polyglutamine toxicity and suppresses the increase of reactive oxygen species caused by huntingtin. Human Molecular Genetics. 11(9). 1137–1151. 407 indexed citations
16.
Ho, Luk, Jenny Carmichael, Jina Swartz, et al.. (2001). The molecular biology of Huntington's disease. Psychological Medicine. 31(1). 3–14. 79 indexed citations
18.
Wyttenbach, Andreas, et al.. (2000). 12-O-Tetradecanoyl-phorbol-13-acetate down-regulates the Huntingtin promoter at Spl sites. Neuroreport. 11(14). 3157–3161. 3 indexed citations
19.
Wyttenbach, Andreas, Pavel M. Borodin, & Jean Hausser. (1998). Meiotic drive favors Robertsonian metacentric chromosomes in the common shrew (<i>Sorex araneus,</i> Insectivora, Mammalia). Cytogenetic and Genome Research. 83(3-4). 199–206. 29 indexed citations
20.
Wyttenbach, Andreas, et al.. (1997). Isolation and characterization of simple sequence repeats in the genome of the common shrew. Molecular Ecology. 6(8). 797–800. 36 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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