Anas M. Alazami

3.9k total citations
55 papers, 1.5k citations indexed

About

Anas M. Alazami is a scholar working on Molecular Biology, Genetics and Immunology. According to data from OpenAlex, Anas M. Alazami has authored 55 papers receiving a total of 1.5k indexed citations (citations by other indexed papers that have themselves been cited), including 25 papers in Molecular Biology, 24 papers in Genetics and 15 papers in Immunology. Recurrent topics in Anas M. Alazami's work include Immunodeficiency and Autoimmune Disorders (11 papers), Genetics and Neurodevelopmental Disorders (7 papers) and Genomics and Rare Diseases (6 papers). Anas M. Alazami is often cited by papers focused on Immunodeficiency and Autoimmune Disorders (11 papers), Genetics and Neurodevelopmental Disorders (7 papers) and Genomics and Rare Diseases (6 papers). Anas M. Alazami collaborates with scholars based in Saudi Arabia, United States and United Kingdom. Anas M. Alazami's co-authors include Fowzan S. Alkuraya, Hadia Hijazi, Fatema Alzahrani, Ranad Shaheen, Amal Y. Kentab, Eissa Faqeih, Serdar Coşkun, Mustafa A. Salih, Saad S. M. Hassan and Firdous Abdulwahab and has published in prestigious journals such as SHILAP Revista de lepidopterología, Scientific Reports and Journal of Allergy and Clinical Immunology.

In The Last Decade

Anas M. Alazami

52 papers receiving 1.5k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Anas M. Alazami Saudi Arabia 22 857 584 156 147 129 55 1.5k
Ghada M. H. Abdel‐Salam Egypt 20 895 1.0× 437 0.7× 69 0.4× 119 0.8× 114 0.9× 89 1.4k
Weston P. Miller United States 25 814 0.9× 211 0.4× 126 0.8× 255 1.7× 143 1.1× 72 1.7k
Henning B. Boldt Denmark 23 833 1.0× 231 0.4× 154 1.0× 215 1.5× 75 0.6× 44 2.2k
Khushnooda Ramzan Saudi Arabia 18 644 0.8× 320 0.5× 236 1.5× 49 0.3× 49 0.4× 79 1.2k
Arif O. Khan Saudi Arabia 24 845 1.0× 610 1.0× 67 0.4× 38 0.3× 56 0.4× 131 1.8k
Mais Hashem Saudi Arabia 23 1.1k 1.2× 544 0.9× 35 0.2× 336 2.3× 166 1.3× 51 1.8k
Eissa Faqeih Saudi Arabia 28 1.5k 1.7× 920 1.6× 31 0.2× 143 1.0× 181 1.4× 80 2.2k
Paolo Simi Italy 22 633 0.7× 571 1.0× 36 0.2× 54 0.4× 64 0.5× 82 1.4k
Ariana Kariminejad Iran 21 756 0.9× 715 1.2× 40 0.3× 63 0.4× 127 1.0× 100 1.5k
Sibel Kantarci United States 15 821 1.0× 1.0k 1.7× 49 0.3× 71 0.5× 35 0.3× 23 1.8k

Countries citing papers authored by Anas M. Alazami

Since Specialization
Citations

This map shows the geographic impact of Anas M. Alazami's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Anas M. Alazami with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Anas M. Alazami more than expected).

Fields of papers citing papers by Anas M. Alazami

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Anas M. Alazami. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Anas M. Alazami. The network helps show where Anas M. Alazami may publish in the future.

Co-authorship network of co-authors of Anas M. Alazami

This figure shows the co-authorship network connecting the top 25 collaborators of Anas M. Alazami. A scholar is included among the top collaborators of Anas M. Alazami based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Anas M. Alazami. Anas M. Alazami is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
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Al‐Mazrou, Amer, Ali Almehaidib, Khalid A. Alsaleem, et al.. (2025). Homozygous Loss of Function PIK3CD Mutation in Multiple Siblings Leading To B Cell Dysregulation and Autoimmunity. Journal of Clinical Immunology. 45(1). 139–139.
3.
Al‐Saud, Bandar, et al.. (2024). A unique STK4 mutation truncating only the C-terminal SARAH domain results in a mild clinical phenotype despite severe T cell lymphopenia: Case report. Frontiers in Immunology. 15. 1329610–1329610. 2 indexed citations
4.
Raducanu, Vlad‐Stefan, Muhammad Tehseen, Amer Al‐Mazrou, et al.. (2024). Severe Combined Immunodeficiency from a Homozygous DNA Ligase 1 Mutant with Reduced Catalytic Activity but Increased Ligation Fidelity. Journal of Clinical Immunology. 44(7). 151–151. 4 indexed citations
5.
Sheikh, Farrukh, Hasan Al‐Dhekri, Bandar Al‐Saud, et al.. (2022). Phenotypic and Genotypic Characterization of Hereditary Angioedema in Saudi Arabia. Journal of Clinical Immunology. 43(2). 479–484.
6.
Janssen, Erin, Anas M. Alazami, Abdullah Alsuliman, et al.. (2022). A homozygous truncating mutation of FGL2 is associated with immune dysregulation. Journal of Allergy and Clinical Immunology. 151(2). 572–578.e1. 2 indexed citations
7.
Shaheen, Ranad, Sateesh Maddirevula, Ghada M. H. Abdel‐Salam, et al.. (2019). PUS7 mutations impair pseudouridylation in humans and cause intellectual disability and microcephaly. Human Genetics. 138(3). 231–239. 68 indexed citations
8.
Ahmed, Hala, Hassan Amer, Abdelbary Prince, et al.. (2018). Deletion of DDB1- and CUL4- associated factor-17 (Dcaf17) gene causes spermatogenesis defects and male infertility in mice. Scientific Reports. 8(1). 9202–9202. 20 indexed citations
9.
Holohan, Brody, Wanil Kim, Tsung-Po Lai, et al.. (2016). Impaired telomere maintenance in Alazami syndrome patients with LARP7 deficiency. BMC Genomics. 17(S9). 749–749. 31 indexed citations
10.
Barakeh, Duna, Eissa Faqeih, Shams Anazi, et al.. (2015). The many faces of KIF7. Human Genome Variation. 2(1). 15006–15006. 14 indexed citations
11.
Alazami, Anas M., Salma Awad, Serdar Coşkun, et al.. (2015). TLE6 mutation causes the earliest known human embryonic lethality. Genome biology. 16(1). 240–240. 147 indexed citations
12.
Seidahmed, Mohammed Zain, et al.. (2015). Report of a case of Raine syndrome and literature review. American Journal of Medical Genetics Part A. 167(10). 2394–2398. 18 indexed citations
13.
Alazami, Anas M., et al.. (2015). Novel compound heterozygous frameshift mutations of C2orf37 in a familial Indian case of Woodhouse–Sakati syndrome. Journal of Genetics. 94(3). 489–492. 8 indexed citations
14.
Wakil, Salma M., Shamsa Anazi, Anas M. Alazami, et al.. (2014). The clinical utility of molecular karyotyping for neurocognitive phenotypes in a consanguineous population. Genetics in Medicine. 17(9). 719–725. 11 indexed citations
15.
Shaheen, Ranad, Anas M. Alazami, Muneera Alshammari, et al.. (2012). Study of autosomal recessive osteogenesis imperfecta in Arabia reveals a novel locus defined by TMEM38B mutation. Journal of Medical Genetics. 49(10). 630–635. 108 indexed citations
16.
Alazami, Anas M., Mohammad Al-Owain, Fatema Alzahrani, et al.. (2012). Loss of function mutation in LARP7, chaperone of 7SK ncRNA, causes a syndrome of facial dysmorphism, intellectual disability, and primordial dwarfism. Human Mutation. 33(10). 1429–1434. 64 indexed citations
17.
Salih, Mustafa A., et al.. (2012). A novel syndrome of lethal familial hyperekplexia associated with brain malformation. BMC Neurology. 12(1). 125–125. 3 indexed citations
18.
Alazami, Anas M., Ranad Shaheen, Fatema Alzahrani, et al.. (2009). FREM1 Mutations Cause Bifid Nose, Renal Agenesis, and Anorectal Malformations Syndrome. The American Journal of Human Genetics. 85(3). 414–418. 64 indexed citations
19.
Alazami, Anas M., Amr Al‐Saif, Abdulaziz Alsemari, et al.. (2008). Mutations in C2orf37, Encoding a Nucleolar Protein, Cause Hypogonadism, Alopecia, Diabetes Mellitus, Mental Retardation, and Extrapyramidal Syndrome. The American Journal of Human Genetics. 83(6). 684–691. 94 indexed citations
20.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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